http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
비선형 최적화 기반 디지털 능동 위상 배열 안테나 부엽 차단 빔 패턴 합성 기법
이원렬(Won-Ryeol Lee),배창식(Chang-Sik Bae),송종화(Jong-Wha Song),김성태(Sung-Tai Kim),홍순기(Sun K. Hong) 한국전자파학회 2024 한국전자파학회논문지 Vol.35 No.5
최근 안테나 빔 패턴의 부엽으로 들어오는 클러터 및 재밍 신호와 같은 간섭 신호로 인해 레이다의 탐지 성능 저하를 방지하기 위하여 디지털 능동 위상 배열 안테나에서 소수의 소자들을 활용하여 추가적인 안테나 사용 없이 부엽 차단 빔 패턴을 형성하는 연구가 제안되어 왔다. 기존의 원하는 부엽 차단 빔 패턴을 정확히 얻기 위해서는 시뮬레이션을 활용하여 수동적으로 소자의 변수(크기, 위상, 위치 등)들을 변경하며 최적의 해를 도출해야 한다. 본 논문에서는 수동적인 방식 대신 패턴 합성법과 소자 신호 크기 및 위상 가중치, 소자 위치를 변수로 갖는 nature inspired 기반의 비선형 최적화 알고리즘(i.e. genetic algorithm, particle swarm optimization)들을 활용하여 신뢰성 있는 최적의 부엽 차단 빔 패턴 합성 및 알고리즘별 결과 비교를 진행하였다. 최적화 결과 두 알고리즘 모두 성공적으로 부엽을 덮을 수 있는 패턴을 생성하였으며, 입자 군집 최적화 알고리즘이 높은 적합도에서 수렴하는 것을 확인하였다. Recently, research has been proposed using a few components in digital active phased array antennas to achieve sidelobe blanking beam patterns without the need for additional antennas, aiming to mitigate the degradation of radar detection performance caused by interference signals such as clutter and jamming entering through the sidelobes. To precisely obtain the desired sidelobe blanking beam pattern, simulations are required to manually vary the parameters of the components (such as magnitude, phase, and position) and derive an optimal solution. In this study, instead of a manual approach, a reliable synthesis of optimal sidelobe blanking beam patterns is proposed based on pattern synthesis methods and nature-inspired nonlinear optimization algorithms (that is, genetic algorithm and particle swarm optimization) with the magnitude, phase and position of array elements as variables. A comparison of the algorithmic results for the synthesis of the optimal sidelobe blanking beam patterns was performed. The optimization results showed that both algorithms successfully generated patterns capable of blanking sidelobes, with the particle swarm optimization demonstrating convergence at higher fitness levels.
Xu, Fei,Mi, Dongbo,Bae, Hong Ryeol,Suh, Min Chul,Yoon, Ung Chan,Hwang, Do-Hoon Korean Chemical Society 2013 Bulletin of the Korean Chemical Society Vol.34 No.9
A series of fluorene-carbazole copolymers containing the pendant phosphor chromophore $Ir(absn)_2(acac)$ (absn: 2-(1-naphthyl)benzothiazole; acac: acetylacetone) were designed and synthesized via Yamamoto coupling. In the film state, these copolymers exhibited absorption and emission peaks at approximately 389 and 426 nm, respectively, which originated from the fluorene backbone. However, in electroluminescent (EL) devices, a significantly red-shifted emission at approximately 611 nm was observed, which was attributed to the pendant iridium(III) complex. Using these copolymers as a single emission layer, polymer light-emitting devices with ITO/PEDOT:PSS/polymer:DNTPD/TmPyPb/LiF/Al configurations exhibited a saturated red emission at 611 nm. The attached iridium(III) complex had a significant effect on the EL performance. A maximum luminous efficiency of 0.85 cd/A, maximum external quantum efficiency of 0.77, maximum power efficiency of 0.48 lm/W, and maximum luminance of 883 $cd/m^2$ were achieved from a device fabricated with the copolymer containing the iridium(III) complex in a 2% molar ratio.
Fei Xu,Dongbo Mi,Hong Ryeol Bae,서민철,윤웅찬,황도훈 대한화학회 2013 Bulletin of the Korean Chemical Society Vol.34 No.9
A series of fluorene-carbazole copolymers containing the pendant phosphor chromophore Ir(absn)2(acac) (absn: 2-(1-naphthyl)benzothiazole; acac: acetylacetone) were designed and synthesized via Yamamoto coupling. In the film state, these copolymers exhibited absorption and emission peaks at approximately 389 and 426 nm, respectively, which originated from the fluorene backbone. However, in electroluminescent (EL) devices, a significantly red-shifted emission at approximately 611 nm was observed, which was attributed to the pendant iridium(III) complex. Using these copolymers as a single emission layer, polymer light-emitting devices with ITO/PEDOT:PSS/polymer:DNTPD/TmPyPb/LiF/Al configurations exhibited a saturated red emission at 611 nm. The attached iridium(III) complex had a significant effect on the EL performance. A maximum luminous efficiency of 0.85 cd/A, maximum external quantum efficiency of 0.77, maximum power efficiency of 0.48 lm/W, and maximum luminance of 883 cd/m2 were achieved from a device fabricated with the copolymer containing the iridium(III) complex in a 2% molar ratio.
Li, Hongliang,An, Jin Ryeol,Seo, Mi Seon,Ha, Kwon-Soo,Han, Eun-Taek,Hong, Seok-Ho,Jung, Won-Kyo,Lee, Dae-Sung,Yim, Mi-Jin,Choi, Grace,Lee, Jeong Min,Bae, Young Min,Choi, Il-Whan,Park, Won Sun Elsevier 2019 european journal of pharmacology Vol.849 No.-
<P><B>Abstract</B></P> <P>In the present study, we investigated the inhibitory effect of tacrolimus, a macrolide immunosuppressive drug that acts through calcineurin inhibition, on voltage-gated K<SUP>+</SUP> (Kv) channels expressed in native smooth muscle cells isolated from the coronary arteries of rabbits. Tacrolimus reduced the amplitude of Kv currents in a dose-dependent manner with an IC<SUB>50</SUB> value and Hill coefficient of 7.80 ± 3.01 μM and 1.07 ± 0.25, respectively. Tacrolimus caused a shift in the activation curve toward a more positive potential and in the inactivation curve toward a more negative potential. Tacrolimus-induced inhibition of Kv current was increased by the application of train pulses (1 or 2 Hz). Furthermore, the recovery time constant of inactivation was extended in the presence of tacrolimus, suggesting that tacrolimus inhibited Kv channels in a use-dependent manner. Two kinds of Kv subtype inhibitors, DPO-1 and guangxitoxin did not affect the degree of tacrolimus-induced inhibition of Kv current. Furthermore, pretreatment with another calcineurin inhibitor, cyclosporine A, did not affect the Kv current, and did not alter the inhibitory effect of tacrolimus. Using perforated-patch clamp experiments, inhibition of Kv channels by tacrolimus caused membrane depolarization. From these results, we concluded that tacrolimus inhibited the vascular Kv currents in a dose, state (open and closed)-dependent manner.</P> <P><B>Graphical abstract</B></P> <P>[DISPLAY OMISSION]</P>
一側 黑質體破壤 白鼠에서 Apomorphine의 回轉運動效果에 미치는 中樞 Opiate 系의 影響
양원모,허광렬,곽용근,김기원,조규박,은홍배 의과학연구소 1991 全北醫大論文集 Vol.15 No.1
To investigate the role of dopamine receptors and possible involvement of brain opiate system in the apomorphine-induced rotational behavior of the substantia nigra-lesioned rats with 6-hydroxydopamine, affects of morphine and naloxone on the action of docpamine agonist or antagonist were examined. Rats were primed with apomorphine after denervation. 1. Apomorphine elicited dose-dependent contralateral cilcling behavior. Maximum development of supersensitivity to apomorphine was observed 4 weeks after denervation in apomorphine-primed rat. The ratatory effect of apomorphine was enhanced by subcutaneous morphine, but diminished by naloxone. 2. A selective D1 agonist, SKF 38393 or a selective D2 agonist LY 171555 induced rotatory behavior of substantia nigra-lesioned rats. Rotatory effect of subcutaneous SKF 38393 was not affected by morphine and naloxone, but the effect of subcutaneous SKF 38393 was potentiated by morphine and dinimished by naloxone. 3. Subcutaneous SCH 23390 or sulpiride decreased the rotatory effect of apomorphine. Naloxone potentiated this effect of SCH 23390, but did not affect the effect of sulpiride. 4. Administration of morphine into ipsilateral caudate nucleus potentiated the rotatory effect of SKF 38393 and LY 171555, while ipsilateral treatment of naloxone did not affect the effect of SKF 38393, but diminished the action of LY 171555. Otherwise, contralateral injection of naloxone did now affect the effect of SKF 38393, but potentiated the action of LY 171555. 5. Chronic administration of naloxone increased the rotatory effects of apormorphine, SKF 38393 and LY 171555. Abrupt withdrawal of the drug potentiated the effect of apomorphine. From the above results, it is suggested that the rotatory effect of apomorphine is due to both the activation of dopamine D1 and D2 receptor, which are party moduated by brain opiate system.