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과량의 에피네프린 피하투여로 유발된 가역적 심근증 1 예
이종구,김재중,송재관,박성욱,박승정,김도하,조재근,추윤호,최성준 대한내과학회 1996 대한내과학회지 Vol.50 No.1
Catecholamine is known to produce a cardiotoxic metabolic byproduct which has been shown to cause reversible cardiomyopathy in excessive amounts. Previous studies have found that reversible cardiomyopathy can be induced by catecholamine in both a time-and dose-dependent manner. In this article, we report one patient in whom 10mg of epinephrine was accidentally injected subcutaneously during the routine operation for chronic otitis media. The patient subsequently suffered from sudden ventricular fibrillation and reversible cardiomyopathy for the next five days, after which she made complete recovery.
하시모토갑상선염에 동반된 골수를 침범한 ( 병기 4E 기 ) 원발성 갑상선 림프종 1 예
이기업,송영기,박중열,김기수,김상희,공경엽,조재근,추윤호,엄완식,지현숙 대한내분비학회 1994 Endocrinology and metabolism Vol.9 No.4
Primary lymphoma of the thyroid is a relatively rare malignant tumor of the thyroid. It is known to be frequently associated with Hashimoto's thyroiditis. In Korea, a few cases of primary lymphoma of the thyroid have been reported and most of these cases were in the stage I E or II E. Recently, we experienced a case of the primary thyroid lymphoma, stage IV E associated with Hashimoto's thyroiditis in a 70-year-old woman who presented with dysphagia and dyspnea due to rapidly enlarging neck mass. She was treated with combination chemotherapy(cyclophosphamide, adriamycin, vincristine and methylprednisolone) and local radiotherapy and achieved partial response with resolution of dysphagia and dyspnea(J Kor Soc Endocrinol 9: 390-394, 1994).
간세포암의 전이 표지자로서 말초 혈액내 알부민 mRNA 검출의 의의
유병철,김정아,서동진,박능화,정영화,이영상,고문수,송일한,이근찬,추윤호,안계순 대한소화기학회 1999 대한소화기학회지 Vol.33 No.2
Background/Aims: Albumin mRNA, one of hepatocyte-specific genes, in peripheral blood has been reported as a marker of circulating hepatocytes in hepatocellular carcinoma (HCC). We evaluated the correlation between albumin mRNA in peripheral blood and evident vascular invasion or extrahepatic metastasis of HCC, and characterized the tumor and clinical features of patients with HCC in association with albumin mRNA in circulation. Methods: We analyzed albumin m-RNA in peripheral blood of 62 patients with hepatocellular carcinoma, 18 patients with chronic viral hepatitis and 8 patients with metastatic liver cancer. Also, we compared the radiological, clinical and tumor characteristics o patients with hepatocellular carcinoma. Results: In 36 of 62 patients with HCC (58%), 5 of 18 patients with chronic viral hepatitis (28%) and none of patients with metastatic liver cancer (0%) albumin mRNA was detected in peripheral blood. We analysed 61 patients with HCC (35 positive and 26 negative for albumin mRNA in peripheral circulation). We did not examine the remaining one patient whose serum ALT level was above 200 IU/L to reduce the possibility of false positive resul by severe hepatic necrosis. Conclusions: The HCC patients with albumin mRNA in peripheral blood were younger than those without it (55 ±9 vs 50 ±9; p=0.03). The rates of serum HBsAg positivity were higher in the patients with albumin mRNA in pheripheral blood than in those without it. Mos of HCC patients with extrahepatic metastasis (17/18; 84%), evident portal vein involvement (26/31 84%), and biliary invasion (9/9; 100%) had albumin mRNA in peripheral circulation. Albumin mRNA was detected more commonly in patients with tumor over 5 cm in diameter than in patients with tumor less 5 cm (76% vs 20%; p$lt;0.001). Moreover, albumin mRNA was more commonly detected in infiltrative type of tumor than in nodular type (90% vs 43%; p$lt;0.001). (Kor J Gastroentero 1999;33:240 - 251)
원발성 항인지질 항체 중후군을 동반한 Budd - Chiari 중후군의 임상상 및 혈관조영상 특성
김경아(Kyoung Ah Kim),정영화(Young Hwa Chung),서동완(Dong Wan Seo),김선희(Seon Hee Kim),추윤호(Yun Ho Chu),고정민(Jeong Min Kho),유빈(Bin Yoo),박철민(Cheol Min Park),성규보(Kyu Bo Sung),이영상(Yung Sang Lee),서동진(Dong Jin Suh) 대한내과학회 1996 대한내과학회지 Vol.51 No.4
N/A Objectives: Many patients with Budd-Chiari syndrome have no evident etiological factor especially in Asian countries. And various obstructive patterns of inferior vena cava and hepatic veins have been reported suggesting several different causes may be involved. Recently primary antiphospholipid antibody syndrome has been described as a characteristic clinical entity with multiple thromboembolic episodes and typical laboratory features such as serum antiphospholipid antibody, not being associated with any collagen vascular disease. To evaluate the etiological role of primary antiphospholipid antibody syndrome in Budd-Chiari syndrome and clarify the clinical features of Budd-Chiari syndrome patients with primary antiphospholipid antibody syndrome, we analyzed clinical and angiographic. data of 27 consecutive patients with Budd-Chiari syndrome (Age: 47±12 years, M: F=13:14). Methods: We analyzed clinical manifestations and angiographic characteristics of 4 Budd-Chiari syndrome patients with primary antiphospholipid antibody syndrome, comparing to those of 23 without it. Results: Underlying etiological factors were identified only in 6(22%); 4(15%) were associated with primary antiphospholipid antibody syndrome. Most of patients with Budd-Chiari syndrome showed superficial abdominal collaterals, ascites, symmetrical lower leg edema and hepatosplenomegaly with laboratory features of liver cirrhosis, regardless the association of primary antiphospholipid antibody syndrome. However, only out of 4 with primary antiphospholipid antibody syndrome, 2 had asymmetrical lower leg edema with ulcer; 2 complained of unexplained long-standing dry cough, 1 of intermittent fever. In both with lower leg ulcer, thrombotic obstructions of deep veins were identified. Another one with primary antiphospholipid antibody syndrome was proved to have pulmonary hypertension without definite vascular obstruction. All of 4 patients(100%) with primary antiphospholipid antibody syndrome in contrast to only 8 out of 23(35%) without it showed broad obstruction of inferior vena cava and all three hepatic veins(Sugiura type II; p<0.05). Conclusion: These data suggested that primary antiphospholipid antibody syndrome is one of common etiological factors in patients with Budd-Chiari syndrome, and that especially in Hudd-Chiari syndrome patients who present asymmetrical lower leg edema with ulcer, long-standing dry cough, unexplained fever, pulmonary hypertension of unknown cause or broad obstruction of inferior vena cava, the possibility of association with primary antiphospholipid antibody syndrome should be considered.