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김찬형,전양환,김재진,권준수 大韓神經精神醫學會 2005 신경정신의학 Vol.44 No.4
Recently, the Korean Neuropsychiatric Association (KCNP) has been expanded greatly in the number of members as well as the qualities in the area of practice and research. There are also 23 affiliated academic societies : Korean Academy of Psychotherapists, Korean Society for Analytical Psychology, the Korean Psychoanalytic Society, the Korean Association for Clinical Art, Korean Academy of Child & Adolescent Psychiatry, the Korean Association of Social Psychiatry, The Korean Society of Biological Therapies in Psychiatry, Korean College of Neuropsychopharmacology, the Korean Society of Biological Psychiatry, Society of Korean Women Psychiatrists, the Korean Society of Clinical Hypnosis, the Society for Human Sexuality, Korean Society of Sleep Research, the Korean Society for Psychopathology and Psychiatric Classification, Korean Academy of Psychiatry and the Law, Korean Psychosomatic Society, Korean Academy of Sleep Medicine, Korean Association for Geriatic Psychiatry, Korean Academy of Psychiatrists in Alcoholism & Addiction, Korean Academy of Adolescent Psychiatry, Korean Academy Schizophrenia, Korean Society for Depressive and Bipolar Disorders, and Korean Academy of Anxiety Disorders. To further encourage the progress of the KCNP affiliated academic societies, the KCNP planning committee has developed the proposed criteria for assessing the societies.
殷載淳,鄭甲基,梁在憲,權鎭,吳贊鎬,全焄 又石大學校 1995 論文集 Vol.17 No.-
The purpose of this research was to investigate effect of water-soluble protein of Trichosanthes kirillowii(TKP) on the proliferation of human tumor cells. TKP inhibited the proliferation of HeLa cells and KHOS-NP cells. The inhibitory activity of doxorubicin on HeLa cells and its of mitomycin C on KHOS-NP cells were increased by the combination of TKP. TKP did not affect on the proliferation of Balb/c 3T3 cells, but inhibited the proliferation of mouse spleen cells and human lymphocyte at 1 ㎍/ml. These results suggest that TKP bas the cytotoxicity on HeLa and KHOS-NP cells, and increase the cytotoxicity of doxorubicin or mitomycin C.
Paraquat 투여 백서에서 Cyclophosphamide와 Dexamethasone의 치료 효과
신정철,신양수,유병전,임현성,정태흥,박찬국,오금탁,이병래 朝鮮大學校 附設 醫學硏究所 1991 The Medical Journal of Chosun University Vol.16 No.1
Paraquat (N,N'-dimethy 1-4,4'bipyrydinium : Gramoxone^(R), a widely used herbicide, is extremely toxic to all green plants and many eukaryotic organisms. In human, paraquat intoxication leads to multiple organ failure, it preferentially damages the lung, kidney and liver and may result in death. The participation of superoxide in initiating tissue damage by administration of paraquat is well known in which one electron reduction of paraquat leads to the formation of radical species which react with molecular oxygen to give superoxide. This process initiates immunological changes with the activation of neutrophilic leukocytes, which is related to further production of superoxide. In this experiment, therapeutic effects of cyclophosphamide and dexamethasone on blood cell count, malondialdehyde as a indicator of lipid peroxidation, and antioxidant enzymes levels in the lung, liver and kidney of paraquat intoxicated rats were inbestigated. The WBC count were significantly decreased by combined treatment of cyclophosphamide and dexamethasone in paraquat intoxicated rats at 3 and 5 days but RBC count were not changed. The contents of malondialdehyde were significantly decreased in the lung and Kidney by combined treatment at 10 days after paraquat intoxication. The catalase acitvities were markedly increased in the lung and liver of rats at 10 days after paraquat intoxication, and superoxide dismutase activities showed minimal changes in the lung, liver and kidney of rats by combined treatment. These results suggest that paraquat induced tissue damage in rats can be redeced by combined administration of cyclophosphamide and dexamethasone.
항만물동량 증가세 둔화에 따른 중소형 항만 개발계획의 타당성 평가
전찬용(Chan-Yong Jeon),남기찬(Ki-Chan Nam),곽규석(Kyu-Seok Kwak),양원(Weon Yang) 한국항해항만학회 2007 한국항해항만학회 학술대회논문집 Vol.1 No.춘계
2000년대 초반까지는 과거의 높은 물동량 증가추세를 계속 이어가는 듯하였으나, 최근 들어 증가세가 둔화되는 양상을 보이고 있다. 이러한 현상은 수출입 물동량뿐만 아니라 환적 물동량에서도 공통적으로 발생하고 있으며, 지속될 경우 우리나라의 동북아 물류 중심지화라는 국가 경제적 목표를 달성하려는 정부의 항만개발계획에 차질을 초래할 우려가 있다. 이에 대응하고자 정부는 2006년 12월 '제2차 전국항만(무역항) 기본계획 수정계획(2006~2011)'을 고시하였다. 본 연구에서는 수정계획에 고시된 중소형 항만의 수출입 및 환적물동량 예측치 및 하역능력을 실제 항만의 실적치와 비교 분석한 후, 현실적인 크레인 대수, 선석점유율 적용을 통해 하역능력을 재산정하여 항만개발계획의 타당성을 평가하고자 한다. The quantity of goods transported seemed to increase until early 2000, however, the rate of increase has been slowing down recently. These situations can be shown both import-export and transit shipment commonly. East-North Asian Hub Port which is the goal of port development of government to achieve macroeconomic targets can be failed if these situations keep going. In order to cope with the matter, government informed "The 2nd revised general planning of the whole country ports(2006~2011)". In this paper, we'll compare and analysis the estimation of the quantity of import-export transported for mid-small ports informed by revised government planning with actual results and then evaluate the propriety of port development and present the way to develop the mid-small ports through the application of realistic number of G/C and berth share.
Implementation of 100CMM Thermal Storage Combustion Oxidizer for VOCs Reduction
Dal-Hwan Yoon,Dal-Hwan Yoon,Beom-Soon Han,Tae-Yeung Lim,Eun-Chan Yang,Yeong-Woong Keon,Hyeon-Woo An,Chan-Ho Kim,Sung-Chan Kang,Min-Ki Jung,Jong-Geun Kim,Hwi-Chan Oh,Chan-Hyouk Jeon,Min-Su Jeon,A-Yeon 제어로봇시스템학회 2022 제어로봇시스템학회 국제학술대회 논문집 Vol.2022 No.11
The heat storage type combustion oxidation device is a system capable of treating VOCs (Volatile Organic Compounds) discharged to the atmosphere at 95% or more. Aromatic hydrocarbons such as benzene are strong carcinogenic substances that cause leukemia, central nervous disorder, and chromosomal abnormalities, and hydrochloride causes destruction of the ozone layer and global warming. This VOCs reduction technology is expected to reduce the number of harmful substances generated for many operating and working conditions. This paper implements a 100CMM Regenerative Thermal Oxidation (RTO) device to reduce air pollution in VOCs generating sites. The carbon-free system is evaluated by measuring the concentration of the VOCs emission gas according to the operating conditions of the production process, analyzing and measuring the THC (Total Hydro-carbons) concentration change. As a result, through the 100CMM RTO experiment, stable RTO driving, renewable energy measurement, and hydrocarbons and nitrogen oxides can be reduced.
Jeon, Se Jin,Seo, Jung Eun,Yang, Sung-Il,Choi, Ji Woong,Wells, David,Shin, Chan Young,Ko, Kwang Ho BioMed Central 2011 JOURNAL OF BIOMEDICAL SCIENCE -BASEL- Vol.18 No.1
<P><B>Background</B></P><P>Fragile X syndrome (FXS), the most commonly inherited mental retardation and single gene cause of autistic spectrum disorder, occurs when the Fmr1 gene is mutated. The product of Fmr1, fragile X linked mental retardation protein (FMRP) is widely expressed in HeLa cells, however the roles of FMRP within HeLa cells were not elucidated, yet. Interacting with a diverse range of mRNAs related to cellular survival regulatory signals, understanding the functions of FMRP in cellular context would provide better insights into the role of this interesting protein in FXS. Using HeLa cells treated with etoposide as a model, we tried to determine whether FMRP could play a role in cell survival.</P><P><B>Methods</B></P><P>Apoptotic cell death was induced by etoposide treatment on Hela cells. After we transiently modulated FMRP expression (silencing or enhancing) by using molecular biotechnological methods such as small hairpin RNA virus-induced knock down and overexpression using transfection with FMRP expression vectors, cellular viability was measured using propidium iodide staining, TUNEL staining, and FACS analysis along with the level of activation of PI3K-Akt pathway by Western blot. Expression level of FMRP and apoptotic regulator BcL-xL was analyzed by Western blot, RT-PCR and immunocytochemistry.</P><P><B>Results</B></P><P>An increased FMRP expression was measured in etoposide-treated HeLa cells, which was induced by PI3K-Akt activation. Without FMRP expression, cellular defence mechanism via PI3K-Akt-Bcl-xL was weakened and resulted in an augmented cell death by etoposide. In addition, FMRP over-expression lead to the activation of PI3K-Akt signalling pathway as well as increased FMRP and BcL-xL expression, which culminates with the increased cell survival in etoposide-treated HeLa cells.</P><P><B>Conclusions</B></P><P>Taken together, these results suggest that FMRP expression is an essential part of cellular survival mechanisms through the modulation of PI3K, Akt, and Bcl-xL signal pathways.</P>
Design and Implementation of Internal Multi-band Folded Monopole Antenna for Mobile Station
Jeon, Jun-Ho,Yang, Woon-Geun,Hong, Yeon-Chan The Korea Institute of Information and Commucation 2011 Journal of information and communication convergen Vol.9 No.1
In this paper, we designed and implemented an internal multi-band folded monopole antenna for mobile handset. The proposed antenna covers Global System for Mobile Communications (GSM900: 880~960 MHz), Digital Communications System (DCS: 1710~1880 MHz), US-Personal Communications Service (US-PCS: 1850~1990 MHz), Bluetooth(2400~2484 MHz), WiMAX(3400~3600 MHz), and Wireless Local Area Network (WLAN: 5150~5350 MHz, 5725~5875 MHz) band for Voltage Standing Wave Ratio $(VSWR)\;{\le}\; 3$. The measured peak gains of the implemented antenna are -1.78dBi at 920MHz, 2.72dBi at 1795MHz, 2.25dBi at 1920MHz, 2.34dBi at 2442MHz, 2.11 dBi at 3550MHz, and 2.04 dBi at 5250MHz.
Iodide management in formamidinium-lead-halide–based perovskite layers for efficient solar cells
Yang, Woon Seok,Park, Byung-Wook,Jung, Eui Hyuk,Jeon, Nam Joong,Kim, Young Chan,Lee, Dong Uk,Shin, Seong Sik,Seo, Jangwon,Kim, Eun Kyu,Noh, Jun Hong,Seok, Sang Il American Association for the Advancement of Scienc 2017 Science Vol.356 No.6345
<P>The formation of a dense and uniform thin layer on the substrates is crucial for the fabrication of high-performance perovskite solar cells (PSCs) containing formamidinium with multiple cations and mixed halide anions. The concentration of defect states, which reduce a cell's performance by decreasing the open-circuit voltage and short-circuit current density, needs to be as low as possible. We show that the introduction of additional iodide ions into the organic cation solution, which are used to form the perovskite layers through an intramolecular exchanging process, decreases the concentration of deep-level defects. The defect-engineered thin perovskite layers enable the fabrication of PSCs with a certified power conversion efficiency of 22.1% in small cells and 19.7% in 1-square-centimeter cells.</P>