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박주현,박주현 한국공업화학회 2020 한국공업화학회 연구논문 초록집 Vol.2020 No.-
최근 hydroxystyrene을 기반으로 한 레진은 3D Nand 공정에 사용되는 후막용 포토레지스트와 페키징용 포토레지스트에 핵심 레진으로 사용됨에 따라 다시 주목을 받고 있는 소재이다. 본 레진의 제조 공정은 프리라디칼 중합방법, 음이온 중합방법 그리고 리빙 라디칼 중합방법으로 나눌 수 있다. 중합방법의 선정은 공중합 모노머 또는 사용 용도의해 결정된다고 할 수 있다. 본 발표에서는 레진의 중합 방법과 그에 따른 물성에 대해 다룰 예정이다.
박주현,고영진,김종현,이종인,신지남,임성훈,임선 대한재활의학회 2004 Annals of Rehabilitation Medicine Vol.28 No.2
Objective: The purpose of this study was to investigate the effect of ischemic stroke on bone metabolism. Method: Female Sprague-Dawley rats (12 weeks old, n=48) were randomly divided into 4 separate groups; sham operation group (group A), stroke group (group B), ovariectomy group (group C), and stroke-ovariectomy group (group D). Two weeks after performing ovariectomy, cerebral ischemia was induced. The bone mineral density (BMD) and osteocalcin and carboxy-terminal telopeptide (CTX) were measured on three periods: the day before ischemia and 1 and 3 weeks post-stroke. All data were statistically analyzed. Results: One week after cerebral ischemia, lumbar spine BMD of group B and D although statistically insignificant showed a lower BMD score in comparison to group A and C, respectively. Three weeks after ischemia, compared to group C, the BMD score of lumbar spine in group D was reduced significantly (p<0.05). At one week post-stroke, compared with group A and C, the value of osteocalcin in group B and D were reduced significantly, respectively (p<0.05). One and three weeks after ischemia, the CTX value in all groups showed no statistical difference. Conclusion: The authors concluded that ischemic stroke affected bone metabolism by decreasing osteoblastic activity in the early phase of stroke rat.
Ginsenoside Rb1이 C6 Glioma 세포주의 Nitric Oxide 생성과 세포자멸사에 미치는 효과
박주현,이윤희,강구성,이수경,김선주,박지영,곽은경,손윤경 대한병리학회 2004 Journal of Pathology and Translational Medicine Vol.38 No.1
Background : Ginsenosides, the extract of Panax ginseng, exert various pharmacological effects such as anticancer activity by the mechanism that is not yet defined. In this study, we proposed that the anticancer effect of ginsenoside Rb1 is related to tumor cell apoptosis and ginsenoside Rb1 induces the tumor cell apoptosis via the nitric oxide (NO) production. Methods : Rat C6 glioma cells were activated by treating with lipopolysaccharide (LPS), interferon (IFN)- , and tumor necrosis factor (TNF)- on the culture medium to investigate the effects of ginsenoside Rb1. Results : Compared with C6 glioma cells treated with LPS/IFN- /TNF- , C6 glioma cells treated with LPS/IFN- /TNF- /ginsenoside Rb1 showed marked increase in the NO production and apoptosis. Ginsenoside Rb1 induces the NO production in C6 glioma cells in dose-dependent manner. When C6 glioma cells treated with LPS/IFN- /TNF- /ginsenoside Rb1 were incubated with the specific inhibitor of iNOS, S-Methyl-2-thiopseudoureasulfate (SMT), both NO production and apoptosis in C6 glioma cells was significantly decreased. Ginsenoside Rb1 induced the expression of iNOS mRNA and iNOS protein in C6 glioma cells. Conclusions : These results suggest that the induction of iNOS expression and subsequent