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Functional group effects on a metal-organic framework catalyst for CO2 cycloaddition
노진미,김영익,박효진,이지현,윤민영,박명환,김영조,김민 한국공업화학회 2018 Journal of Industrial and Engineering Chemistry Vol.64 No.-
A variety of metal-organic frameworks (MOFs) have been reported as efficient catalysts for CO2 fixation reactions, such as cycloaddition to cyclic carbonates. The permanent porosity of the frameworks and the Lewis acidity of the MOF metal sites have been considered as the major contributors to the catalytic activity in the cycloaddition of CO2. In this study, we have, instead, focused on the effects of the organic functional groups for effective catalytic ability. A total of eight different functionalized Zr-based MOFs were tested. It was revealed that the non-functionalized pristine UiO-66 MOF (UiO = University of Oslo) showed the best conversion at low temperature (77% at 50 °C), whereas the hydroxy-functionalized UiO-66-OH MOF displayed the best conversion at high temperature (91% at 140 °C). The Zr-MOF could be recycled up to four times without a significant decrease in the reactivity.
황기은(GE Hwang),조율희(YH Cho),심성한(SH Shim),최부영(BY Choi),차병헌(BH Cha),황윤영(YY Hwang),정성노(SR Chung),최수경(SK Choi),김영조(YJ Kim) 대한산부인과학회 1998 Obstetrics & Gynecology Science Vol.41 No.10
Dystrophin associated muscular dystrophies [dystrophinopathies] range from the severe Duchenne to the milder Becker muscular dystrophy [DMD and BMD]. Mapping and molecular genetic studies indicate that both are the result of mutations in the huge gene that encodes dystrophin. Approximately two-thirds of the mutations in both forms are deletions of one or many exons in the dystrophin gene. This study was performed in order to establish an diagnostic strategy for dystrophinopathy. In this study muscle biopsies were taken from the clinically suspected DMD and BMD patients, and immunohistochemical staining and Western blotting with anti-dystrophin antibodies were applied to the biopsy specimens. Also, the deletion analyses with multiplex PCRs for the 18 deletion prone exons, and linkage analyses for the diagnosis of carrier status and prenatal diagnosis were performed. We suggest that the following diagnostic strategy would be useful for the genetic counseling on dystrophinpathy families. 1. First of all clinically suspected dystrophinopathy patient should be subjected to deletion analysis with multiplex PCRs. If any deletions were found, the confirmatory diagnosis and prenatal diagnosis is possible. But the carrier diagnosis should be made with linkage analysis. 2. In the cases with no deletions, a muscle biopsy should be taken from the patient and protein study with immunohistochemistry or Western blotting should be performed. With normal dystrophin pattern, consider other neuromuscular diseases other than dystrophinopathy. If dystrophin is of reduced or increased size, with or without reduction in the amount of dystrophin, BMD should be suspected. If dystrophin is absent, DMD should be suspected. In this case prenatal and carrier diagnosis should be performed with linkage analysis.