http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
오늘 본 자료
각종 간질환에서 B 형 간염 바이러스 DNA polymerase 활성도와 HBeAg의 관계
최상욱 ( Sang Wook Choi ),김부성 ( Boo Sung Kim ),박두호 ( Doo Ho Park ),이창돈 ( Chang Don Lee ),정진우 ( Jin Wu Jeong ),강혜정 ( Hea Jung Kang ),정욱성 ( Wook Sung Jeong ) 대한소화기학회 1988 대한소화기학회지 Vol.20 No.1
N/A DNA-Polymerase and HBeAg appear to be indicaters of relative infectivity of HBsAg-positive serum. The acute disease in the HBeAg-positive patients with acute hepatitis differed significantly from that of HBeAg-negative (HBsAg-positivie) patients. However it did not correlate with the type or severity of liver disease after HBV infection, since HBeAg was present in both chronic benign and chronic active hepatitis B infections. Presence of HBeAg was associated with increase in DNA polymerase activity and in number of circulating Dane particles in previous reports, but it does not correlate with DNA polymerase activity, Dane particles in our studies. To evaluate the activity of HBV DNA in various liver disease, we studed it in 20 patients with CAH, HCC, and 10 patients, who is carrier state with HBeAg, without HBeAg, AVH and CPH respectively. The mean value of HBV DNA polymerase activity is 4092.60+5429.36 cpm in HBe-positive heathy carriers, 203.80+52.80 cpm in HBe-negative healthy carriers, 386.30+471.58 cpm in CPH, 742.05+942. 77 cprn in VAH, 211.95+110.27 cpm in HCC, and 413.50+77.60 cpm in AVH. These results suggest that onging hepatitis B viral replication is more active in HBeAg positive healthy carriers than in carriers without HBeAg, a finding that may help explain the high prevalence of individuals. Thus, DNA polymerase appears to identify the period of peak hepatitis B virus replication, the result of antiviral agents therapy, and test will be useful in evaluating the safety and efficacy of future hepatitis B virus infection.
항 Ro 항체 양성인 특발성 혈소판감소성 자반증에서 전신성 홍반성 낭창으로 이행한 2 예
이경식,박동준,서정민,민우성,김호연,김동집,정욱성,이현재 대한내과학회 1987 대한내과학회지 Vol.33 No.4
ITP is an immunologically-mediated destructive thrombocytopenia. SLE is a chronic inflammatory disease which may affect many different organs and discloses various laboratory findings. Although the relationship of ITP and SLE is not well recognized at present. We report two cases of Anti-Ro antibody positive ITP subsequently developing SLE. We thick that presence of Anti-Ro antibody in ITP may show a predictable pattern in patients who later develop SLE.