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金東柱,金漢宇 대한전자공학회 2002 電子工學會論文誌-CI (Computer and Information) Vol.39 No.11
Computational models up to now for Korean morphology have been linear in that it deal with only segmentation of morphemes rather than formation of the internal structure of a word. When integrating a linear computational model with syntax analysis, it requires an additional interface component between this model and the syntax to bind morphemes into sentence constituents. Furthermore the linear model is not semantically intuitive. In this paper, based on word-syntactical viewpoint, we propose an integrated computational model that deals with morpheme segmentation, formation of syntactic element (sentence constituent), and even internal structure of word. Formalism of two-level morphology is employed to cope with morpheme segmentation and alternation problems, and functional diacritics are proposed to incorporate categorial context into the two-level formalism. A modified GLR-based algorithm is also proposed to check syntactical constraint of morphemes. 한국어 형태론에 대한 기존의 전산모형은 선형적인 것들로 단어 내부구조 분석보다 형태소 분리 문제에만 관심을 두고 있다. 이러한 선형적 전산모형을 구문 분석 과정과 통합적으로 고려할 경우, 구문 단위 요소의 형성을 위해 형태소 분석 결과를 묶어야만 하는 추가적인 과정이 필요할 뿐만 아니라 의미적 직관성을 얻기도 어려웠다. 본 논문에서는 형태소 분리와 구문 요소 형성뿐만 아니라 단어의 구조 분석까지도 통합적으로 다룰 수 있는 단어통사론적 시각에 따른 전산 모형을 제안한다. 먼저 형태소 분리와 변형 문제를 다루기 위해 2단계형태론의 형식화를 도입하고, 품사 문맥을 반영하기 위해 기능성 구분문자를 제안한다. 그리고 형태소의 통사적 결합 검사를 위해 GLR에 기반한 변형 알고리즘을 제안한다.
금동주 ( Dong Joo Keum ),양두경 ( Doo Kyoung Yang ),김광진 ( Kwang Jin Kim ),지삼룡 ( Sam Ryong Jee ),윤진혁 ( Chin Hyuk Yun ),이성원 ( Sung Won Lee ),정원태 ( Won Tae Chung ) 대한류마티스학회 2000 대한류마티스학회지 Vol.7 No.3
Behcet`s disease is a recurring illness characterized by the triple symptom complex of aphthous stomatitis, genital ulceration, and uveitis. The disease is multisystemic disorder with involvement of skin, gastrointestinal tract, blood vessels, central nervous system, joints, and epididymis. Vascular involvement of Behcet`s disease affects both arteries and veins and blood vessels of all sizes. Periosteal new bone formation is one feature of hypertrophic osteoarthropathy. Other features are clubbing and arthritis. Periosteal new bone formation on the bones of the lower extremities without other features of hypertrophic osteoarthropathy has been reported in patients with varicose veins, vitamin A intoxication, infantile cortical hyperostosis, tuberous sclerosis, congenital syphilis and chronic renal failure with hyperparathyroidism, and chronic venous insufficiency. However, periosteal new bone formation has not, to our knowledge, been described in patients with Behcet`s disease yet although similar findings are occasionally noted in polyarteritis nodosa. We report a case of Behcet`s disease complicated by leg swelling with periosteal new bone formation of right lower leg.
십이지장 궤양에서 Helicobacter pylori 독성인자 유전자 아형과 IS605의 역할
금동주 ( Dong Joo Keum ),한상영 ( Sang Young Han ),김광진 ( Kwang Jin Kim ),지삼룡 ( Sam Ryong Jee ),이종훈 ( Jong Hun Lee ),최석렬 ( Seok Ryeol Choi ),신우원 ( Woo Won Shin ),김정만 ( Jeong Man Kim ),홍숙희 ( Sook Hee Hong ),김 대한소화기학회 2003 대한소화기학회지 Vol.41 No.2
Background/Aims: The Helicobacter pylori (H. pylori) cagA, vacA, and ieeA are considered to be important virulence factors that have been implicated in the development of duodenal ulcers. It was reported that the presence of IS605 elements might be respon
범복막염으로 술후 E.coli에 의해 발생한 괴사성 근막염 1예
김동현,금동주,김명준,최슬기,최정석,이현성,이상민,강미선 인제대학교 백병원 2003 仁濟醫學 Vol.24 No.1
Necrotizing fasciitis is an uncommon severe soft tissue infection characterized by extensive necrosis of superficial fascia with widespread involvement of the surrounding tissues and concurrent systemic toxicity. It is accompanied by local pain, fever and systemic toxicity and is rapidly progressive and destructive infection and is often fetal if not promptly recognized and aggressively treated. It is usually caused by mixed aerobic-anaerobic bacteria. It can affect any part of the body but is most common on the extremities, perineum and abdominal wall, especially the leg. Predisposing factors include diabetes mellitus, alcoholism, intravenous drug abusers, abdominal surgery, perineal infection. Tissue damage and systemic toxicity are believed to result from the release of endogenous cytokines and bacterial toxins. Early recognition, broad-spectrum antibiotics coverage and adequate debridement has been associated with improved survival. We report a case of necrotizing fasciitis caused by E. coli after operation.
위선암에서 Helicobacter pylori 독성인자와 유전자 아형의 관련성
신종민,한상영,금동주,김광진,지삼룡,홍기봉,이종훈,최석렬,신우원,Shin Jong Min,Han Sang Young,Keum Dong Joo,Kim Kwang Jin,Jee Sam Ryong,Hong Gi Bong,Lee Jong Hun,Choi Seok Ryeol,Shin Woo Won 대한위암학회 2002 대한위암학회지 Vol.2 No.1
Purpose: The H. pylori cagA gene, vacA gene and iceA gene are considered to be important virurence factors that have been implicated in the development of gastric adenocarcinoma. It was reported that the presence of IS605 elements may be responsible for rearrangements and lead to partial or total deletions of the cag pathogenicity island (PAI) and the virulence of cag PAI may be changed. However, different results regarding the association between these virulence factors and clinical disease have been reported from different geographic regions. This study evaluated the relationship between H. pylori virulence factors such as cagA, vacA, iceA, IS605 and gastric adenocarcinoma. Materials and Methods: H. pylori isolates were obtained from 54 infected patients (24 cases of gastric adenocarcinoma, 30 cases of control). H. pylori isolates were identified by PCR with ureC gene and 16S rRNA. PCR was performed to examine cagA, vacA, iceA and IS605 genotypes. Results: Significant difference was found in the negative rates of cagA between gastric adenocarcinoma group and control ($62.5\%\;vs.\;33.3\%$ P=0.033). No significant difference was found in the prevalence of iceA, vacA between gastric adenocar cinoma and control. The genotype of cagA+ vacA s1-m1 iceA1 was predominant in H. pylori isolates irrespective of the clinical outcome. IS605 in PAI was not found in gastric adenocarcinoma gruop and control. The positive rates of IS605 in genome were $33.3\%$ in gastric adenocarcinoma group and $36.7\%$ in control (P>0.05). In gastric carcinoma, the positive rate of $cagA^{+}/IS605$ was lower than in control ($12.5\%\;vs\;40.0\%$, P=0.025) and the positive rate of cagA-/IS605 was higher than in control ($54.2\%\;vs\;23.3\%$, P=0.02). Conclusion: H. pylori virulence factors had not related significantly with gastric adenocarcinoma. Further study is needed to examine the specificity of H. pylori strains.