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후복막강 부신경절종 적출시 Nicardipine과 Esmolol을 사용한 마취관리 : 증례보고 A case report
강규식,김덕,김천숙 순천향의학연구소 2004 Journal of Soonchunhyang Medical Science Vol.10 No.2
Paragangliomas are tumors arising from the neuroendocrine elements (Chief cells) of the paraganglia. Paragangliomas are uncommon tumors and are infrequently found in the extraadrenal retroperitoneum. We investigated and treated a patient with retroperitoneal paraganglioma, which was found incidentally at abdominal computer tomograhy. It was confirmed by pathological study. We report a case of retroperitoneal paraganglioma successfully managed intraoperatively with an infusion of nicardipine and an intermittent esmolol injection.
강규식,박종무,윤병우 대한신경과학회 2006 대한신경과학회지 Vol.24 No.6
Idiopathic thrombocytopenic purpura (ITP) is an immunologic disorder that destroys platelets and megakaryocytes. Hemorrhagic complications are common in patients with ITP. However, thrombotic complications such as coronary artery disease and cerebral infarctions are infrequent. We report a patient with ITP who developed a cerebral infarction despite thrombocytopenia after long-term danazol therapy and was negative for the antiphospholipid antibody.
액체-액체층을 보이는 결핵성 뇌농양의 확산강조영상과 수소자기공명분광분석
강규식,하재혁,이수호,이광우 대한신경과학회 2007 대한신경과학회지 Vol.25 No.2
We encountered a patient with tuberculous brain abscesses, which is very rare. Diffusion-weighted imaging showed high signal in the lower part of the large abscess cavity and low signal in the upper part of the cavity. Proton MRS showed resonances representing lactate. Combined studies with diffusion-weighted imaging and MRS are useful in the differential diagnosis of tuberculous abscess from other structures that feature ring-enhanced lesions.
일시적 국소 뇌허혈을 일으킨 수컷 쥐의 뇌에서는 프로락틴이 새로운 세포 생성을 촉진하지 못할 가능성이 있다: 예비 연구
강규식,이순태,권형민,김민정,김영주,이경미,박종무,주건,김만호,윤병우 대한신경과학회 2006 대한신경과학회지 Vol.24 No.2
Background: Production of neuronal progenitors is usually stimulated in the forebrain subventricular zone of mice after the intracerebroventricular infusion of prolactin. As a preliminary study, we infused prolactin to the male rat brain to test the hypothesis that prolactin promotes new cell proliferation in the brain and functional recovery after focal ischemia. Methods: Male rats were subjected to intraluminal middle cerebral artery occlusion. Prolactin was administered to the surface of the brain for 5 or 14 days starting 24 or 48 hours after stroke onset at doses of 6.4 µg per day. We administered the same volume of saline to the other ischemic rats used as a control group. Some rats were killed 6 or 17 days after stroke for analysis of infarct volume and newly generated cells within the subventricular zone and the striatum. The other rats were tested for neurological recovery 24 days after stroke. Results: There was no significant difference of infarct volume among the experiment groups. Treatment with prolactin did not increase the numbers of bromodeoxyuridine-immunoreactive cells in the subventricular zone and the striatum. Treatment with prolactin did not enhance neurological recovery in all tests performed. Conclusions: In this preliminary study, prolactin did not enhance new cell generation in the male rat brain nor reduce the neurological deficits after ischemic stroke.
한국인 환자의 아토르바스타틴에 의한 근육통 발병률에 대한 단일 병원 전향연구: 스타틴근육통 임상지수의 적용
강규식,김병건 대한신경과학회 2022 대한신경과학회지 Vol.40 No.1
Background: Statin-induced myalgia is a common cause of discontinuation and a barrier that interferes with long-term treatment. The incidence of myalgia reported by previous atorvastatin trials in Korea ranged from 0.8% to 3.4%. This study assessed the incidence of myalgia after atorvastatin was administered to Korean patients with dyslipidemia. In addition, the Statin Myalgia Clinical Index (SMCI) was used to assess the likelihood that a patient’s myalgia was caused by atorvastatin. Methods: Dyslipidemic patients were eligible to participate if they were statin-naïve or treated with statins other than atorvastatin. Muscle complaints were assessed at the baseline, the 10-weeks visit and the final follow-up visit (16 weeks or later). The SMCI score was calculated if a patient developed myalgia, which was rated as probable, possible or unlikely related to the statin. Results: A total of 89 patients were analyzed. The atorvastatin doses ranged from 10 to 80 mg. Six (7%) patients reported new and unexplained muscle pain. Information on the timing of myalgia relative to stopping was unavailable in two patients. After excluding these two patients, three (3.4%) out of 87 patients were classified by the SMCI as having possible or probable atorvastatin-associated myalgia. Conclusions: In this study, the incidence of myalgia was higher than the incidences reported by the previous trials in Korea. However, the incidence of statin-associated myalgia assessed using SMCI was comparable to those of the trials. The SMCI may help diagnosis of statin-associated myalgia in clinical practice and optimize treatment for patients with myalgia.