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Ju Hyoung Lee,Kyeong Min Jo,Tae Oh Kim,Jong Ha Park,Seung hyun Park,Jae Won Jung,So Chong Hur,양성연 대한소화기내시경학회 2016 Clinical Endoscopy Vol.49 No.6
Brunner’s gland hamartomas are small benign lesions that are most commonly found in the bulb of the duodenum. They are veryuncommon, and most are found incidentally during upper gastrointestinal series or esophagogastroduodenoscopy. The lesions tendto be asymptomatic, but patients may present with symptoms of duodenal obstruction or hemorrhage secondary to ulceration. Histologically, a Brunner's gland hamartoma consists of the components of Brunner's gland cells, as well as glandular, adipose andmuscle cells. In this study, we report the case of a 30-year-old man who presented with upper gastrointestinal bleeding and obstructivesymptoms due to a giant Brunner's gland hamartoma in the duodenal bulb. The hamartoma was successfully removed by endoscopicresection. No significant complications were observed. Microscopically, the lesion was found to be entirely composed of variableBrunner's glands and adipocytes.
삼배체 염색체 이상을 보인 급성 림프구성 백혈병 1 예
전종구(Chong Ku Jun),차주영(Ju Young Cha),오형모(Hyung Mo Oh),신요식(Yo Shik Shin),김윤권(Yun Kwon Kim),김소연(So Yon Kim),김영중(Young Jung Kim),박병익(Byung Yik Park),조민구(Min Koo Cho),이권전(Gwon JUn Lee),이경인(Kyung In Lee),이은 대한내과학회 2001 대한내과학회지 Vol.61 No.2
In addition to age, white cell count and immunophenotype, karyotype has been reported to be one of the important prognostic factors in acute lymphocytic leukemias. Furthermore 70 percent of patients with acute B lymphocytic leukemia presented chromosomal abnormalities, which is known to have a close relationship with the prognosis. Among the abnormalities, triploid is rare and known to have the worse prognosis. Structural chromosomal abnormality of the 11q23 band is more common in childhood acute lymphocytic leukemia and has been rarely reported in adult lymphocytic leukemia. We present a case of a 29 year - old male patient with acute lymphocytic leukemia, who had triploid and chromosomal translocation including 11q23 band along with the review of related literature.(Korean J Med 61:190-194, 2001)
Seung-Ju Ahn,Jae-Kyoung Choi,Young Mi Joo,Min-A Lee,Pyung-Rak Choi,Yeong-Mi Lee,Myong-Shin Kim,So-Young Kim,Eun-Hee Jeon,Byung-In Min,Chong-Rak Kim 대한의생명과학회 2004 Biomedical Science Letters Vol.10 No.3
Lung cancer is a leading cause of cancer death worldwide; however, despite major advances in cancer treatment during the past two decades, the prognostic outcome of lung cancer patients has improved only minimally. This is largely due to the inadequacy of the traditional screening approach of diagnosis in lung cancer, which detects only well-established overt cancers and fails to identify precursor lesions in premalignant conditions of the bronchial tree. In recent years this situation has fundamentally changed with the identification of molecular abnormalities characteristic of premalignant changes; these concern tumour suppressor genes, loss of heterozygosity at crucial sites and activation of oncogenes. Basic knowledge at the molecular level has extremely important clinical implications with regard to early diagnosis, risk assessment and prevention, and therapeutic targets. In this study we used a "cap-finder" subtractive hybridization method, "long distance" polymerase chain reaction (PCR), streptavidin magnetic beads mediated subtraction, and spin column chromatography to detect differential expression genes of human small cell lung carcinoma. We have now isolated ninety two genes that expressed differentially in the human small cell lung carcinoma cells and analyzed of 12 clones with sequencing, nine cDNAs include tapasin (NGS-17) mRNA, BC200 alpha scRNA, chromosome 12q24 PAC RPCI3-462E2, protein phosphatase 1 (PPPICA), translocation protein 1 (TLOC1), ribosomal protein S24 (RPS24) mRNA, protein phosphatase (PPEF2), cathepsin Z, MDM2 gene and three novel genes. They may be oncogenesis-related proteins.
Jang, Moon Ju,Jeon, Young Joo,Choi, Won-il,Choi, Yi Seul,Kim, Su Yeoun,Chong, So Young,Oh, Doyeun,Kim, Nam Keun SAGE Publications 2013 Clinical and applied thrombosis/hemostasis Vol.19 No.3
<P>The frequency of methylenetetrahydrofolate reductase (MTHFR) mutations varies between racial and ethnic groups, and there are also conflicting data regarding <I>MTHFR</I> gene mutations in Asian patients with venous thromboembolism (VTE). The aim of this study was to examine the association between common <I>MTHFR</I> gene mutations (677C>T and 1298A>C) and risk of VTE in Koreans. This study was a retrospective case–control study. We enrolled 203 patients with VTE and 403 controls. For the 677C>T polymorphism, there was no difference in the frequency of the CT genotype and TT genotype between the patients with VTE and the controls. However, in the recessive analysis (CC + CT vs TT), the frequency of the TT genotype was significantly higher in VTE than in controls (odds ratio = 1.700; 95% confidence interval = 1.108-2.607, <I>P</I> = .015). In conclusion, the TT genotype of <I>MTHFR</I> 677C>T increases the risk of VTE in Koreans. This finding was supported by meta-analysis of previous Asian studies.</P>
Ahn, Seung-Ju,Choi, Jae-Kyoung,Joo, Young Mi,Lee, Min-A,Choi, Pyung-Rak,Lee, Yeong-Mi,Kim, Myong-Shin,KIm, So-Young,Jeon, Eun-Hee,Min, Byung-In,Kim, Chong-Rak THE KOREAN SOCIETY FOR BIOMEDICAL LABORATORY SCIEN 2004 Journal of biomedical laboratory sciences Vol.10 No.3
Lung cancer is a leading cause of cancer death worldwide; however, despite major advances in cancer treatment during the past two decades, the prognostic outcome of lung cancer patients has improved only minimally. This is largely due to the inadequacy of the traditional screening approach of diagnosis in lung cancer, which detects only well-established overt cancers and fails to identify precursor lesions in premalignant conditions of the bronchal tree. In recent years this situation has fundamentally changed with the identification of molecular abnormalities characteristic of premalignant changes; these concern tumour suppressor genes, loss of heterozygosity at crucial sites and activation of oncogenes. Basic knowledge at the molecular level has extremely important chical implications with regard to early diagnosis, risk assessment and prevention, and therapeutic targets. In this study we used a "cap-finder" subtractive hybndization method, "long distance" polymerase chain reaction (PCR), streptavidin magnetic beads mediated subtraction, and spin column chromatography to detect differential expression genes of human small cell lung carcinoma. We have now isolated ninety two genes that expressed differentially in the human small cell lung carcinoma cells and analyzed of 12 clones with sequencing, nine cDNAs include tapasin (NGS-17) mRNA, BC200 alpha scRNA, chromosome 12q24 PAC RPC13-462E2, protein phosphatase 1 (PPPICA), translocation protein 1 (TLOCl), ribosomal protein S24 (WS24) mRNA, protein phosphatase (PPEF2), cathepsin Z, MDM2 gene and three novel genes. They may be oncogenesis-related proteins.