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周鶴群 ( Zhou He-qun ) 성균관대학교 대동문화연구원 2023 大東文化硏究 Vol.124 No.-
이 논문은 유일한 女神仙으로서 何仙姑가 八仙에 진입하게 된 과정을 살피고, 나아가 하선고가 팔선에 진입한 후의 이미지 변화를 고찰하는 데 그 목적이 있다. 하선고는 여러 다른 지역에서 명성을 떨쳤던 지방 여선이었다. 그러나 全眞教 교도들이 祖師 呂洞賓의 신통을 돋보이게 하기 위해 하선고 고사를 각색하였고 그로 인해 하선고는 道教 계보에 편입하게 되었다. 이후 하선고는 여동빈의 제자로서 서서히 팔선에 진입하였다. 하선고가 팔선에 합류된 것은 다른 7명의 신선보다 비교적 늦은 시기인 嘉靖 年間(1522~1566)이었다. 명대 이후 팔선 서사는 점차 世俗化하여 하선고의 이미지에서 女性性이 강조되기 시작하였다. 이는 특히 연극・소설 등 通俗文學에서 두드러졌다. 따라서 하선고 개인의 고사는 더 이상 주목받지 못하게 되었으며, 다만 팔선 멤버들과 함께 ‘親民的 신선’의 정체성을 공유하게 된다. 더불어 하선고의 여성성이 활용되어 극적인 스토리에 광채를 돋우는 역할을 담당했다. This paper aims to explore the process of He Xiangu’s entry into the Eight Immortals as the only female immortal and to further examine the changes in her image after she joined the group. He Xiangu was initially a local immortal who was well-known in several regions. However, The Quanzhen School altered her story to showcase the exceptional ability of their master, Lü Dongbin and included He Xiangu in the Taoist canon. Later, as a disciple of Lü Dongbin, she gradually became a member of the Eight Immortals. He Xiangu joined the Eight Immortals during the Jiajing period(1522~1566) of the Ming Dynasty, which was a relatively late date compared to the other seven members. After the Ming Dynasty, the narratives of the Eight Immortals began to be secularized, and He Xiangu’s image became more focused on femininity. This is especially evident in popular literature such as operas and novels. As a result, He Xiangu’s personal story no longer receives much attention, but she shares with the other members of the Eight Immortals the identity of ‘the god with the common touch’. In addition, the femininity of the female immortal He Xiangu adds flavor to the dramatic story.
( Yue Qun Wang ),( Xiang Li Ye ),( Jun Mei Zhou ),( Yong Qi Wan ),( Hua Ping Xie ),( Yun Deng ),( Yan Yan ),( Yong Qing Li ),( Xiong Wei Fan ),( Wu Zhou Yuan ),( Xiao Yang Mo ),( Xiu Shan Wu ) 생화학분자생물학회 2011 BMB Reports Vol.44 No.1
Zinc finger (ZNF) proteins play a critical role in cell growth, proliferation, apoptosis, and intracellular signal transduction. In this paper, we cloned and characterized a novel human KRAB-related zinc finger gene, ZNF425, which encodes a protein of 752 amino acids. ZNF425 is strongly expressed in the three month old human embryos and then is almost undetectable in six month old embryos and in adult tissues. An EGFP-ZNF425 fusion protein can be found in both the nucleus and the cytoplasm. ZNF425 appears to act as a transcription repressor. Over-expression of ZNF425 inhibits the transcriptional activities of SRE, AP-1, and SRF. Deletion analysis indicates that the C2H2 domain is the main region responsible for the repression. Our results suggest that the ZNF425 gene is a new transcriptional inhibitor that functions in the MAPK signaling pathway. [BMB reports 2011; 44(1): 58-63]
ZNF424, a novel human KRAB/C2H2 zinc finger protein, suppresses NFAT and p21 pathway
( Yue Qun Wang ),( Jun Mei Zhou ),( Xiang Li Ye ),( Yong Qi Wan ),( Yong Qing Li ),( Xiao Yan Mo ),( Wu Zhou Yuan ),( Yan Yan ),( Na Luo ),( Ze Qun Wang ),( Xiong Wei Fan ),( Yun Deng ),( Xiu Shan Wu 생화학분자생물학회 2010 BMB Reports Vol.43 No.3
Zinc finger-containing transcription factors are the largest single family of transcriptional regulators in mammals, which play an essential role in cell differentiation, cell proliferation, apoptosis, and neoplastic transformation. Here we have cloned a novel KRAB-related zinc finger gene, ZNF424, encoding a protein of 555aa. ZNF424 gene consisted of 4 exons and 3 introns, and mapped to chromosome 19p13.3. ZNF424 gene was ubiquitously expressed in human embryo tissues by Northern blot analysis. ZNF424 is conserved across species in evolution. Using a GFP-labeled ZNF424 protein, we demonstrate that ZNF424 localizes mostly in the nucleus. Transcriptional activity assays shows ZNF424 suppresses transcriptional activity of L8G5-luciferase. Overexpression of ZNF424 in HEK- 293 cells inhibited the transcriptional activity of NFAT and p21, which may be silenced by siRNA. The results suggest that ZNF424 protein may act as a transcriptional repressor that suppresses NFAT and p21 pathway to mediate cellular functions. [BMB reports 2010; 43(3): 212-218]
First report of Bradysia difformis (Diptera: Sciaridae) Damage to Phalaenopsis orchid in China
Qun Xin Han,Dong Mei Cheng,Juan Luo,Cui Zuan Zhou,Qing Sheng Lin,Mei Mei Xiang 한국응용곤충학회 2015 Journal of Asia-Pacific Entomology Vol.18 No.1
Phalaenopsis orchid is among the most valuable ornamental flowering plants in the world. Since visible damage substantially decreases its amenity, limited damage is allowed in its production. An unknown insect species (Diptera: Sciaridae)was found to cause serious damage to the seedling of the Phalaenopsis orchid in greenhouses in Guangdong, China. The insect occurred in high populations in almost all greenhouses that grow Phalaenopsis orchid and the number of sciarid adults trapped on a yellow sticky card could reach as many as 303 in 24 h. An effectivemanagement strategy on any pest requires an accurate identification. Therefore, it is urgent to identify this pest correctly to mitigate its damage to the industry. Damage to Phalaenopsis orchid and morphological characteristics of the pest was described in this study. Molecular analyses based on the 488-bp portion of the mitochondrialDNA fromthe cytochrome oxidase I(mt COI) regionwere conducted to supplementmorphological characteristics in identifying this pest. Bothmorphological characteristics and phylogenetic tree constructedwith mt COI genes identified this sciarid as Bradysia difformis Frey, 1948 (= Bradysia paupera Tuomikoski, 1960) (Diptera: Sciaridae). A literature search indicated that B. difformis has been a common pest of greenhouse and forestry nurseries in Europe and South Africa. Our study is the first record of B. difformis damaging Phalaenopsis orchid in China.
Cinobufacin Suppresses Cell Proliferation via miR-494 in BGC-823 Gastric Cancer Cells
Zhou, Rong-Ping,Chen, Gang,Shen, Zhi-Li,Pan, Li-Qun Asian Pacific Journal of Cancer Prevention 2014 Asian Pacific journal of cancer prevention Vol.15 No.3
Cinobufacin is used clinically to treat patients with many solid malignant tumors. However, the mechanisms underlying action remain to be detailed. Our study focused on miRNAs involved in cinobufacin inhibition of GC cell proliferation. miRNA microarray analysis and real time PCR identified miR-494 as a significant cinobufacin-associated miRNA. In vivo, ectopic expression of miR-494 inhibited the proliferation and induced apoptosis of BGC-823 cells on CCK-8 and flow cytometry analysis. Further study verified BAG-1 (anti-apoptosis gene) to bea target of miR-494 by luciferase reporter assay and Western blotting. In summary, our study demonstrated that cinobufacin may inhibit the proliferation and promote the apoptosis of BGC-823 cells. Cinobufacin-associated miR-494 may indirectly be involved in cell proliferation and apoptosis by targeting BAG-1, pointing to use as a potential molecular target of cinobufacin in gastric cancer therapy.
Ziqiang, Zhou,Feng, Feng,Jinglong, Zhang,Yihui, Xiong,Xinyi, Ping,Qun, Huang 한국재료학회 1995 Fabrication and Characterization of Advanced Mater Vol.1 No.1
The pressure-composition-temperature(PCT) curves of $Mm_{0.6}Ml_{0.4}Ni_{5_x}Co_{x}(x{\leqslant}2.5)$hydrogne stroage alloys(HSA) are determined and the mathermatical model of PCT curve the author proposed is applied to analyse the data of PCT curve of the alloy system. It been shown from the results that the PCT curve of $Mm_{0.6}Ml_{0.4}Ni_{5-x}Co_{x}$can also be modeled by the mathematical model and the calculated PCT curves are in good agreement with the experimental curves. A lot of physical parameters of these alloys which are very useful are estimated from the modeling.
Expression and Functional Role of ALDH1 in Cervical Carcinoma Cells
Rao, Qun-Xian,Yao, Ting-Ting,Zhang, Bing-Zhong,Lin, Rong-Chun,Chen, Zhi-Liao,Zhou, Hui,Wang, Li-Juan,Lu, Huai-Wu,Chen, Qin,Di, Na,Lin, Zhong-Qiu Asian Pacific Journal of Cancer Prevention 2012 Asian Pacific journal of cancer prevention Vol.13 No.4
Tumor formation and growth is dictated by a very small number of tumor cells, called cancer stem cells, which are capable of self-renewal. The genesis of cancer stem cells and their resistance to conventional chemotherapy and radiotherapy via mechanisms such as multidrug resistance, quiescence, enhanced DNA repair abilities and anti-apoptotic mechanisms, make it imperative to develop methods to identify and use these cells as diagnostic or therapeutic targets. Aldehyde dehydrogenase 1 (ALDH1) is used as a cancer stem cell marker. In this study, we evaluated ALDH1 expression in CaSki, HeLa and SiHa cervical cancer cells using the Aldefluor method to isolate ALDH1-positive cells. We showed that higher ALDH1 expression correlated with significantly higher rates of cell proliferation, microsphere formation and migration. We also could demonstrate that SiHa-ALDH1-positive cells were significantly more tumorigenic compared to SiHa-ALDH1-negative cells. Similarly, SiHa cells overexpressing ALDH1 were significantly more tumorigenic and showed higher rates of cell proliferation and migration compared to SiHa cells where ALDH1 expression was knocked down using a lentivirus vector. Our data suggested that ALDH1 is a marker of cervical cancer stem cells and expand our understanding of its functional role.