http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Factors Associated With Screen Time Among School-Age Children in Korea
Ham, Ok Kyung,Sung, Kyung Mi,Kim, Hee Kyung SAGE Publications 2013 The Journal of school nursing Vol.29 No.6
<P>The purpose of this study was to investigate the association of sociodemographic, psychosocial, and behavioral characteristics with screen time among school-age children in Korea. This study employed a nonexperimental, cross-sectional study design. A total of 370 children attending four elementary schools participated in the study. Self-report method was used, and instruments included screen time (time spent on TV/video/computer/video games), sleep duration, eating behavior, pros and cons of exercise, and exercise self-efficacy. According to the results, 45.7% of the children had screen time of 1–2.9 hr/day and 8.9% had 3 or more hr/day. Increased screen time showed an association with gender (boy), higher body mass index, fast food consumption, higher cons of exercise, having a working mother, and attendance at a school in an inner city area (<I>p</I> < .05). Understanding the factors associated with screen time may provide useful information in the development of health promotion programs aimed at decreasing sedentary behaviors.</P>
Anti-obesity and anti-hepatosteatosis effects of dietary scopoletin in high-fat diet fed mice
Ham, Ju Ri,Lee, Hae-In,Choi, Ra-Yeong,Sim, Mi-Ok,Choi, Myung-Sook,Kwon, Eun-Young,Yun, Kyeong Won,Kim, Myung-Joo,Lee, Mi-Kyung Elsevier 2016 Journal of Functional Foods Vol.25 No.-
<P><B>Abstract</B></P> <P>The effects of scopoletin on non-alcoholic fatty liver in obese mice were investigated. Mice were fed high-fat diet (HF) with or without two doses of scopoletin (0.01 and 0.05%, w/w) for 16 weeks. Both doses of scopoletin led to similar reductions in body weight, visceral fat, serum levels of leptin, lipid, TNFα, IL-6, IFNγ and MCP-1, insulin resistance and hepatic lipid accumulation, whereas they increased serum adiponectin and faecal lipid levels. Ingenuity pathway analysis revealed that hepatic gene networks related to lipid concentrations, inflammation of organs, quantity of adipose tissue, proliferation of cell and necrosis were down-regulated in the scopoletin group. The top up- or down-regulated genes were <I>Cidea</I>, <I>Apoa4</I>, <I>Cyp7a1</I>, <I>Errfi1</I>, <I>Col1a1</I>, <I>Mmp13</I>, <I>Cdkn1a</I>, <I>Gdf15</I> and <I>Saa1</I>, which emerged as associated genes related to hepatic steatosis and inflammation. These results indicate that scopoletin may ameliorate HF-induced hepatic dysfunction via regulation of lipid metabolic and inflammatory genes.</P> <P><B>Highlights</B></P> <P> <UL> <LI> Scopoletin attenuates non-alcoholic fatty liver disease in high-fat diet fed mice. </LI> <LI> Scopoletin identified 3 gene networks related to lipid metabolism and inflammation. </LI> <LI> Target genes of lipid metabolism are <I>Cidea</I>, <I>Apoa4</I>, <I>Cyp7a1</I> and <I>Errfi1</I>. </LI> <LI> Target genes of inflammation are <I>Col1a1</I>, <I>Mmp13</I>, <I>Cdkn1a</I> and <I>Saa1.</I> </LI> </UL> </P>
Anti-inflammatory and antioxidant effects of umbelliferone in chronic alcohol-fed rats
Mi-Ok Sim,Hae-In Lee,Ju Ri Ham,Kwon-Il Seo,Myung-Joo Kim,Mi-Kyung Lee 한국영양학회 2015 Nutrition Research and Practice Vol.9 No.4
BACKGROUND/OBJECTIVES: Inflammation is associated with various types of acute and chronic alcohol liver diseases. In this study, we examined whether umbelliferone (7-hydroxycoumarin, UF) ameliorates chronic alcohol-induced liver damage by modulating inflammatory response and the antioxidant system. METHODS: Rats were fed a Liber-Decarli liquid diet containing 5% alcohol with or without UF (0.05 g/L) for 8 weeks, while normal rats received an isocaloric carbohydrate liquid diet. RESULTS: Chronic alcohol intake significantly increased serum tumor necrosis factor-α (TNF-α) and interleukin 6 levels and decreased interleukin 10 level; however, UF supplementation reversed the cytokines related to liver damage. UF significantly suppressed hepatic lipopolysaccharide binding protein, toll-like receptor 4 (TLR4), nuclear factor kappa B, and TNF-α gene expression increases in response to chronic alcohol intake. Masson"s trichrome staining revealed that UF improved mild hepatic fibrosis caused by alcohol, and UF also significantly increased the mRNA expressions and activities of superoxide dismutase and catalase in liver, and thus, decreased lipid peroxide and mitochondrial hydrogen peroxide levels. CONCLUSIONS: The findings of this study indicate that UF protects against alcohol-induced liver damage by inhibiting the TLR4 signaling pathway and activating the antioxidant system.
Anti-inflammatory and antioxidant effects of umbelliferone in chronic alcohol-fed rats
Mi-Ok Sim,Hae-In Lee,Ju Ri Ham,Kwon-Il Seo,Myung-Joo Kim,Mi-Kyung Lee 대한지역사회영양학회 2015 Nutrition Research and Practice Vol.5 No.6
BACKGROUND/OBJECTIVES: Inflammation is associated with various types of acute and chronic alcohol liver diseases. In this study, we examined whether umbelliferone (7-hydroxycoumarin, UF) ameliorates chronic alcohol-induced liver damage by modulating inflammatory response and the antioxidant system. METHODS: Rats were fed a Liber-Decarli liquid diet containing 5% alcohol with or without UF (0.05 g/L) for 8 weeks, while normal rats received an isocaloric carbohydrate liquid diet. RESULTS: Chronic alcohol intake significantly increased serum tumor necrosis factor-α (TNF-α) and interleukin 6 levels and decreased interleukin 10 level; however, UF supplementation reversed the cytokines related to liver damage. UF significantly suppressed hepatic lipopolysaccharide binding protein, toll-like receptor 4 (TLR4), nuclear factor kappa B, and TNF-α gene expression increases in response to chronic alcohol intake. Masson"s trichrome staining revealed that UF improved mild hepatic fibrosis caused by alcohol, and UF also significantly increased the mRNA expressions and activities of superoxide dismutase and catalase in liver, and thus, decreased lipid peroxide and mitochondrial hydrogen peroxide levels. CONCLUSIONS: The findings of this study indicate that UF protects against alcohol-induced liver damage by inhibiting the TLR4 signaling pathway and activating the antioxidant system.