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Printed Origami Structures (Adv. Mater. 20/2010)
Ahn, Bok Yeop,Shoji, Daisuke,Hansen, Christopher J.,Hong, Eunji,Dunand, David C.,Lewis, Jennifer A. WILEY-VCH Verlag 2010 Advanced Materials Vol.22 No.20
<B>Graphic Abstract</B> <P>Bok Y. Ahn, Jennifer Lewis, and co-workers report on p. 2251 a new method for creating complex 3D structures that combines direct-write assembly with a wet-folding origami technique. Planar lattices composed of a titanium hydride ink are printed, and then folded, rolled, or molded into the desired shape. These 3D objects are then transformed into metallic or ceramic structures by thermal annealing. <img src='wiley_img_2010/09359648-2010-22-20-ADMA201090069-content.gif' alt='wiley_img_2010/09359648-2010-22-20-ADMA201090069-content'> </P>
AHN, JEONG K.,PITLUK, ZACHARY W.,WARD, DAVID C. 충남대학교 생물공학연구소 1993 생물공학연구지 Vol.3 No.-
To further define transcriptional regrlation of the P38 promoter in the minute virus of mice (MVM) genome, we constructed a series of internal deletion and linker scanning mutations. The mutant P38 constructs were assayed for transcriptional activity in vitro by primer extension analysis with nuclear extracts from murine A92L fibroblasts. Mutations which disrupted the GC box and TATA box severely reduced transcription in vitro. DNase I footprinting analysis confirmed that the murine transcription factor SpI bound to the GC box; however, no factors were observed interacting with a putative transcriptional activation regulatory element, termed the TAR element. The linker scanning mutations were analyzed in vivo by using a chloramphenicol acetyltransferase expression assay system, in both the presence and absence of constructs expressing the viral nonstructural protein, NSI. The ability of NSI to transactivate the P38 promoter (up to 1,000-fold) depended entirely on the presence of intact GC and TATA box sequences. Disruption of the TAR element by either linker insertion mutations or an internal deletion did not inbibit transactivation of the P38 promoter. These results suggest that NSI transactivates the P38 promoter indiracty by interacting with one or more components of the P38 core-transcription complex.
Ahn, Bok Yeop,Shoji, Daisuke,Hansen, Christopher J.,Hong, Eunji,Dunand, David C.,Lewis, Jennifer A. WILEY-VCH Verlag 2010 Advanced Materials Vol.22 No.20
<B>Graphic Abstract</B> <P>Printed origami structures are fabricated by combining direct-write assembly of planar lattices with a wet-folding origami technique. This novel approach provides a low-cost, versatile route to create three-dimensional structures from diverse materials, whose shapes range from simple polyhedra to intricate origami forms. <img src='wiley_img_2010/09359648-2010-22-20-ADMA200904232-content.gif' alt='wiley_img_2010/09359648-2010-22-20-ADMA200904232-content'> </P>
Direct-write assembly of microperiodic planar and spanning ITO microelectrodes
Ahn, Bok Yeop,Lorang, David J.,Duoss, Eric B.,Lewis, Jennifer A. Royal Society of Chemistry 2010 Chemical communications Vol.46 No.38
<P>Printed Sn-doped In<SUB>2</SUB>O<SUB>3</SUB> (ITO) microelectrodes are fabricated by direct-write assembly of sol–gel inks with varying concentration. This maskless, non-lithographic approach provides a facile route to patterning transparent conductive features in planar arrays and spanning architectures.</P> <P>Graphic Abstract</P><P>Printed planar and spanning structures of Sn-doped In<SUB>2</SUB>O<SUB>3</SUB> (ITO) microelectrodes are fabricated by direct-write assembly of a concentrated sol–gel ink. <IMG SRC='http://pubs.rsc.org/services/images/RSCpubs.ePlatform.Service.FreeContent.ImageService.svc/ImageService/image/GA?id=c0cc01691h'> </P>
( David S. Kim ),( Beom Kyung Kim ),( Jun Yong Park ),( Do Young Kim ),( Sang Hoon Ahn ),( Kwang Hyub Han ),( Seung Up Kim ) 대한간학회 2018 춘·추계 학술대회 (KASL) Vol.2018 No.1
Aims: Trans-arterial chemoembolization (TACE) improves survival of patients with hepatocellular carcinoma (HCC). However, the treatment outcomes of TACE in patients with treatment-naive HCC versus recurrent HCC after curative resection has not been compared. Methods: A total of 448 patients with treatment-naive HCC and 275 patients with recurrent HCC after curative resection treated with TACE as the first-line anti-cancer treatment were recruited. Cox regression analysis was used to identify independent factors affecting overall mortality. Results: Patients with treatment-naive HCC at the time of TACE showed a significantly higher proportion of liver cirrhosis (61.9% vs. 49.3%), higher aspartate aminotransferase level (median 48 vs. 31 IU/L), higher alanine aminotransferase level (median 38 vs. 26 IU/L), higher alpha-fetoprotein level (median 96.6 vs. 7.7 ng/mL), higher total bilirubin level (mean 0.97 vs. 0.84 mg/ dL), longer prothrombin time (1.05 vs. 1.01 INR), higher tumor number (mean 2.1 vs. 1.7), larger tumor size (3.1 vs. 1.6 cm), and lower proportion of BCLC stage 0-B (vs stage C) (55.6% vs. 71.9%) (all P<0.05). Multivariate analysis showed that TACE for treatment-naive HCC (vs. recurrent HCC after curative resection) was one of independent risk factors of mortality (hazard ratio, 1.328; 95% confidence interval, 1.038-1.700; P=0.024), together with higher alpha-fetoprotein level and higher tumor number (all P<0.05). Conclusions: Patients with treatment-naive HCC showed poorer clinical characteristics than those with recurrent HCC after curative resection at the time of TACE and TACE for treatment-naive HCC (vs. TACE for recurrent HCC after curative resection) was independently associated with the increased risk of mortality.
( David Sooik Kim ),( Soo Young Park ),( Beom Kyung Kim ),( Jun Yong Park ),( Do Young Kim ),( Yu Rim Lee ),( Won Young Tak ),( Young Oh Kweon ),( Sang Hoon Ahn ),( Seung Up Kim ) 대한간학회 2020 춘·추계 학술대회 (KASL) Vol.2020 No.1
Aims: On September 2015, the Korean Association for the Study of Liver (KASL) guideline on initiating antiviral therapy (AVT) for patients with chronic hepatitis B (CHB)-related cirrhosis changed from HBV DNA level ≥2,000 IU/L and aminotransferase (AST) or alanine aminotransferase (ALT) levels over the upper normal limit to HBV DNA level ≥2,000 IU/L, regardless of AST or ALT levels. This study investigated whether the KASL guideline change reduced the risk of CHB-related hepatocellular carcinoma (HCC) in patients with cirrhosis in South Korea. Methods: A total of 429 patients with CHB-related cirrhosis who initiated AVT between 2014 and 2016 were recruited. The risk of HCC development was compared between patients who initiated AVT before and after September 2015 (previous [n=196, 45.7%] vs. current guideline [n=233, 54.3%]). Results: Univariate analysis showed that AVT initiation according to previous guideline, older age, and male gender significantly predicted increased risk of HCC development (all P<0.05). Subsequent multivariate analysis showed that AVT initiation according to previous guideline (HR=1.833), older age (HR=1.041), and male gender (HR=2.719) independently predicted increased risk of HCC development (all P<0.05). Additionally, multivariate analysis showed that AVT initiation according to previous guideline (HR=2.400) and male gender (HR=3.058) independently predicted mortality (P<0.05). The cumulative incidences of HCC and mortality were significantly higher in patients who initiated AVT before guideline change than in those who initiated AVT after guideline change (all P<0.05, log-rank test). Conclusions: The prognosis of patients with CHB-related cirrhosis who initiated AVT improved after KASL guideline modifications.
A divergent external loop confers antagonistic activity on floral regulators FT and TFL1
Ahn, Ji Hoon,Miller, David,Winter, Victoria J,Banfield, Mark J,Lee, Jeong Hwan,Yoo, So Yeon,Henz, Stefan R,Brady, Robert Leo,Weigel, Detlef Wiley (John WileySons) 2006 The EMBO journal Vol.25 No.3
<P>The Arabidopsis genes FT and TERMINAL FLOWER1 (TFL1) encode related proteins with similarity to human Raf kinase inhibitor protein. FT, and likely also TFL1, is recruited to the promoters of floral genes through interaction with FD, a bZIP transcription factor. FT, however, induces flowering, while TFL1 represses flowering. Residues responsible for the opposite activities of FT and TFL1 were mapped by examining plants that overexpress chimeric proteins. A region important in vivo localizes to a 14-amino-acid segment that evolves very rapidly in TFL1 orthologs, but is almost invariant in FT orthologs. Crystal structures show that this segment forms an external loop of variable conformation. The only residue unambiguously distinguishing the FT and TFL1 loops makes a hydrogen bond with a residue near the entrance of a potential ligand-binding pocket in TFL1, but not in FT. This pocket is contacted by a C-terminal peptide, which also contributes to the opposite FT and TFL1 activities. In combination, these results identify a molecular surface likely to be recognized by FT- and/or TFL1-specific interactors.</P>
EULER-BERNOULLI BEAM WITH DYNAMIC FRICTIONLESS CONTACT
Jeongho AHN,David E. STEWART 한국산업응용수학회 2005 한국산업응용수학회 학술대회 논문집 Vol.- No.-
In this work, we formulate the frictionless Euler-Bernoulli equation with dynamic contact condition along the length of a thin beam, and then set up a numerical formulation, employing the midpoint rule for the elastic part of the equation and the implicit Euler method for contact conditions. Convergence for our numerical formulation is investigated. The energy functional is defined, and our numerical scheme leads to energy dissipation. Using time discretization and the FEM with B-spline basis functions, we compute numerical solutions. In order to solve the linear complementarity problem that arises in the numerical method, we use a smoothed guarded Newton method. Those numerical schemes are implemented and some interesting numerical results are obtained. We also investigate numerically the question of whether the numerical solutions converge strongly to their limit, and if energy is conserved for the limit. Our numerical results give some evidence that this is so.