http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
한국인에서 D-Dimer의 위양성과 관련된 인자와 검사의 정확도 향상을 위한 Cut-Off Value
홍만용 ( Man Yong Hong ),이창근 ( Chang Kun Lee ),유상용 ( Sang Yong Yoo ),신대희 ( Dae Hee Shin ),정상식 ( Sang Sig Cheong ),권장훈 ( Jang Hoon Kwon ),장우성 ( Woo Sung Jang ),유승진 ( Seung Jin Yoo ),오광훈 ( Kwang Hoon Oh ) 대한내과학회 2013 대한내과학회지 Vol.84 No.3
Background/Aims: The D-dimer value is a simple blood test used to evaluate venous thromboembolism (VTE). However, due to its low specificity, another test is needed for a definite diagnosis, such as a radiographic test. We evaluate the factors associated with a false positive D-dimer test and propose a new cut-off value for detecting VTE more effectively in Koreans. Methods: This was a retrospective, observational study. From January 2009 to December 2009, 2,047 patients (988 men, 63 ± 15 years) had the D-dimer value checked to evaluate VTE. The main outcome of interest was a positive D-dimer test. Odds ratio and 95% confidence intervals were determined using logistic regression analysis. The new D-dimer cut-off was evaluated using receiver operating characteristics (ROC) curves. Results: The result was positive in 1,093 patients (53%), for a false positive percentage for VTE of 95% and a false negative percentage for VTE of 1%. Significant false positive predictors for a positive D-dimer were increasing age, trauma, postoperative, acute infection, tuberculosis, stroke, malignancy, chronic renal failure, acute coronary syndrome, heart failure, and lung disease. The discriminative value of the D-dimer test was assessed using ROC curve analysis. A D-dimer value of 0.68 mg/L on admission was the best cut-off value for predicting the development of VTE with a sensitivity of 95% and specificity of 57%. Conclusions: Many factors affect the D-dimer value and we must consider these factors before using the D-dimer value to evaluate VTE. A D-dimer value of 0.68 mg/L appears to be a good cut-off value for evaluating VTE more effectively in Koreans. (Korean J Med 2013;84:372-378)
증례 / 재발성 구강내 진균감염증이 동반된 IgG3 아형 결핍증 1례
최정희(Jeong Hee Choi),유상용(Sang Yong Yoo),송정엽(Jeong Yeob Song),김선신(Sun Sin Kim),이수걸(Soo Keol Lee),남동호(Dong Ho Nahm),박해심(Hae Sim Park) 대한천식알레르기학회 2000 천식 및 알레르기 Vol.20 No.4
Common clinical manifestations in patients with IgG subclass deficiency include recurrent respiratory tract infection, recurrent otitis media and sinopulmonary infection by virus or bacteria. The administration of intravenous immunoglobulin(IVIG) has been regarded as the most effective therapy in these patients. We experienced a 22-year-old patient with IgG3 subclass deficiency and recurrent fungal infection of oral cavity and lips. IVIG was given at 0.2g/kg/dose twice a month for 6 months. After treatment with IVIG, the patient improved clinically. (J Asthma Allergy Clin Immunol 20: 650-654, 2000)
IgG1 및 IgG3 아형 결핍이 동반된 중증 아스피린 - 과민성 천식 환자에서 intravenous immunoglobulin 치료 1 례
박해심(Hae Sim Park),김희연(Hee Yeon Kim),남동호(Dong Ho Nahm),유상용(Sang Yong Yoo),안성균(Sung Gyun Ahn),한동석(Dong Suk Han) 대한천식알레르기학회 1999 천식 및 알레르기 Vol.19 No.4
Common complications in a patient with IgG subclass deficiency include recurrent respiratory infections and concomitant inflammatory lung disease. The most effective therapy in these pati- ents is the administration of intravenous immunoglobulin. The authors report a case of severe aspirin-sensitive asthma and recurrent pneumonia with combined IgG1, and IgG3 subclass deficie- ncy in a 19-year-old man. The patient was treated with 0.4g/kg at monthly intervals for 6 months, and is still receiving 0.2g/kg every 2 weeks at our clinic. After the replacement of in- travenous immunoglobulin, the patient has clinically improved.
아급성형 Gaucher 씨 병에서의 동종골수이식과 유세포 분석기를 이용한 Glucocerebrosidase 활성화 추적 1 예
김덕기(Deog Ki Kim),김현수(Hyun Soo Kim),유상용(Sang Yong Yoo),정철권(Cheol Kweon Jeong),박준성(Joon Seong Park),하만준(Mahn Joon Ha),김현주(Hyon Ju Kim),김효철(Hugh Chul Kim) 대한내과학회 2001 대한내과학회지 Vol.61 No.2
Gaucher's disease (GD) is the most common inherited lysosomal storage disease, manifested by generalized accumulation of glucocerebroside in macrophages of the reticuloendothelial system due to a deficient lysosomal β-glucocerebrosidase (GC). It is inherited by an autosomal recessive pattern in which three clinical phenotypes have been described based on the presence and severity of neurologic involvement. GD is treated possible by GC enzyme replacement therapy, allogeneic bone marrow transplantation (BMT), and gene therapy. We here report the experience of successful allogeneic BMT in a 16-year-old female patient with GD type III which was demostrated markedly increased Gaucher cells in bone marrow and absence of GC activity in peripheral blood monocytes by FACS using 5'- pentafluorobenzoylaminofluorescein-di-β-D-glucoside (PFBFDGlu) as substrate. Donor marrow engraftment was confirmed by chromosome analysis using microsatellite and by bone marrow examination. Assay of GC activity using FACS revealed normal level of enzyme activity. She remains alive and well after 12 months of BMT.(Korean J Med 61:195-200, 2001)