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서동수,김효중,조준형,서태수 ( Dong Soo Suh,Hyo Joong Kim,Jun Hyung Cho,Tae Soo Suh ) 한국화학공학회 1995 Korean Chemical Engineering Research(HWAHAK KONGHA Vol.33 No.3
This study was undertaken to establish an industrial process for the preparation of low-soda alumina useful in the manufacture of electronic ceramics. First, starting material, Al(OH)₃, was admixed with the chlorine-containing compounds, NH₄Cl or HCl, employed as the treating agent for reducing the soda in starting material, and then admixture was calcined at a temperature sufficient to eliminate the soda as a volatilizable compound which was produced by reaction of the soda and the above mentioned treating agents. This treatment is so-called the volatilization method. The results obtained in the study show that addition amount of treating agents has an enormous effect on the elimination of soda, and that optimum addition amount of NH₄Cl and HCl is 2.43 and 8.38(mole of treating agent/mole of Na₂O), respectively.
상피성 난소암 세포에서 MyD88 발현과 paclitaxel의 항세포고사 신호
서동수 ( Dong Soo Suh ),조무성 ( Moo Sung Jo ),유신애 ( Shin Ae Yu ),김기형 ( Ki Hyung Kim ),윤만수 ( Man Soo Yoon ) 대한산부인과학회 2010 Obstetrics & Gynecology Science Vol.53 No.4
Objective: The objectives of this study was to evaluate the correlation between myeloid differentiation protein 88 (MyD88) expression and paclitaxel effects on epithelial ovarian cancer cells and to evaluate whether paclitaxel had anti-apoptotic signals. Methods: Epithelial ovarian cancer cells isolated from ascites and established cell lines were treated with increasing concentrations of paclitaxel (0.2 to 20 μM) for 24 and 48 hours and cell viability was determined using the CellTiter 96 AQueous One Solution Cell Proliferation Assay. Cytokine profiling was performed from culture supernatants using the Luminex 200 system. Nuclear factor-kappaB (NF-κB) activity was determined using a Luciferase reporter system. Levels of phospho-extracellular signal-regulated kinase (p-ERK) were measured by Western blot analysis. Results: A strong signal for MyD88 expression was observed in R182, 01-19b and SKOV3 cells (MyD88-positive). A2780, R454 and 01-28 cells showed low levels of MyD88 (MyD88-negative). Paclitaxel effectively decreased cell viability in MyD88-negative A2780, R454, 01-28 cells after 24 and 48 hours (57%, 49%, 42% and 35%, 28%, 29%, respectively). MyD88-positive cells were resistant to paclitaxel. There was a significant increase in caspase-3/7 activity following paclitaxel treatment in MyD88-negative cells. No significant change in caspase-3/7 activity was detected in MyD88-positive cells. Paclitaxel induced NF-κB activation and enhanced the secretion of interleukin-6 (IL-6) and IL-8 in a dose dependent manner and induced ERK phosphorylation on MyD88-positive cells. Conclusion: Paclitaxel treatment for MyD88-positive ovarian cancer could have detrimental effects due to the paclitaxel-induced enhancement of NF-κB, ERK activities and pro-inflammatory cytokine production, which promote chemoresistance and tumor progression.
상피성 난소암세포에서 이소플라본 유도체 NV-196의 세포고사 유도 효과
서동수 ( Dong Soo Suh ),주종길 ( Jong Kil Joo ),김승철 ( Seung Chul Kim ),이동형 ( Dong Hyung Lee ),이우희 ( Woo Hee Yi ),윤만수 ( Man Soo Yoon ),김기형 ( Ki Hyung Kim ) 대한산부인과학회 2011 Obstetrics & Gynecology Science Vol.54 No.2
Objective: The objectives of this study were to determine the efficacy of NV-196, a synthetic isoflavone derivative, as a chemosensitizer in chemoresistant CP70 and R182 epithelial ovarian cancer (EOC) cells and to characterize the mechanism behind its sensitizing effect. Methods: EOC cells were treated with tenfold dilutions of NV-196 (0.1 to 10 μg/mL) for 24 and 48 hours. Cell viability was determined by the CellTiter 96 AQueous One Solution Cell Proliferation Assay. Apoptosis was assessed by Caspase-Glo assays and apoptotic cascade X-linked inhibitor of apoptosis protein (XIAP), caspase-2 and Bid were characterized by Western blot analyses. Results: As a monotherapy, NV-196 showed decreased cell viability in a time- and dose-dependent manner in both CP70 and R182 cells. A significant increase in caspase-3 activity was observed in both cells. Caspase-8 and -9 activation were also observed. Western blots demonstrated Bid and caspase-2 activation and cleavage of XIAP. NV-196 enhances the cytotoxic effects of carboplatin and paclitaxel. Conclusion: NV-196 induces cell death through the induction of apoptosis. Pretreatment with NV-196 may sensitize the ovarian cancer cells to carboplatin or paclitaxel. NV-196 may act as a chemosensitizing agent in epithelial ovarian cancer cells.
관광호텔 Front Desk Agent 의 직무만족에 관한 실증연구 - 서울시내 강남지역 특 1 급 호텔을 중심으로 -
서동수(Dong Soo Suh) 관광경영학회 2001 관광경영연구 Vol.12 No.-
The purpose of this study is to present an influencing factor which effects on front desk agent job satisfaction and to help improve their job satisfaction. In order to achieve the above purpose, I have run parallel analysis with both the literature and empirical study. I adopted a questionnaire for the empirical study, which is based on a literature study basically. The result of this study is First, Female is lower than man on the personnel evaluation of merits. second, career within 1 year has a good relationship with manager. on the other side, Career with over 7years has a low recognition on the personnel evaluation. third, Working Environment and duty right is lower than other factors. So the suggestion of this study is as follows. 1. Offer the opportunity of promotion equally between male and female. 2. Necessity of manager`s retraining and reeducation for taking the qualities of professional knowledge and staff control. 3. Magnification of duty right in working and Improvement of working environment.