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정종갑,이상아,최성이,강엽,김태호,전자영,배명애,안희진,정하나,황은숙,이관우 한국분자세포생물학회 2015 Molecules and cells Vol.38 No.12
The TAZ activator 2-butyl-5-methyl-6-(pyridine-3-yl)-3- [2'-(1H-tetrazole-5-yl)-biphenyl-4-ylmethyl]-3H-imidazo[4,5- b]pyridine] (TM-25659) inhibits adipocyte differentiation by interacting with peroxisome proliferator-activated receptor gamma. 1 TM-25659 was previously shown to decrease weight gain in a high fat (HF) diet-induced obesity (DIO) mouse model. However, the fundamental mechanisms underlying the effects of TM-25659 remain unknown. Therefore, we investigated the effects of TM-25659 on skeletal muscle functions in C2 myotubes and C57BL/6J mice. We studied the molecular mechanisms underlying the contribution of TM-25659 to palmitate (PA)-induced insulin resistance in C2 myotubes. TM-25659 improved PAinduced insulin resistance and inflammation in C2 myotubes. In addition, TM-25659 increased FGF21 mRNA expression, protein levels, and FGF21 secretion in C2 myotubes via activation of GCN2 pathways (GCN2-phosphoeIF2α- ATF4 and FGF21). This beneficial effect of TM-25659 was diminished by FGF21 siRNA. C57BL/6J mice were fed a HF diet for 30 weeks. The HF-diet group was randomly divided into two groups for the next 14 days: the HF-diet and HF-diet + TM-25659 groups. The HF diet + TM-25659- treated mice showed improvements in their fasting blood glucose levels, insulin sensitivity, insulin-stimulated Akt phosphorylation, and inflammation, but neither body weight nor food intake was affected. The HF diet + TM- 25659-treated mice also exhibited increased expression of both FGF21 mRNA and protein. These data indicate thatTM-25659 may be beneficial for treating insulin resistance by inducing FGF21 in models of PA-induced insulin resistance and HF diet-induced insulin resistance.
食餌脂肪量이 鉛中毒 白鼠의 血中 Transaminase 活性에 미치는 影響
丁鐘甲 圓光大學校大學院 1979 學位論叢 Vol.2 No.-
This study is about effect of lipid contents in diet on Albino rat poisoned by lead acetate. After feeding, for four weeks the diet of different contents of lipid and lead acetate of 16mg/day, the activity of Transaminase, the weight of body and the weight of liver was examined. The results are as follows; 1. Generally the Rate of growth decreased according to the increase of lipid content in diet. 2. The weight of liver increased according to the increase of lipid content in diet. (Table-6). 3. The activity of transaminase increased according to the lipid contents in diet. (Table-7). In conclusion one may be guessed that the high lipid diet enforced the toxicity of lead acetate.
사용자 인증을 통한 클라이언트 기반 DDoS 공격 대응 매커니즘
정종갑 ( Jongkap Jeong ),이희조 ( Heejo Lee ) 한국정보처리학회 2011 한국정보처리학회 학술대회논문집 Vol.18 No.2
최근의 분산 서비스 거부(DDoS) 공격 방법은 점점 더 자동화, 다양화되고 있으며, 단순한 개별 기업에 대한 공격에서부터 국가간의 사이버전쟁으로까지 그 영역이 확대되고 있다. DDoS 공격은 이미 2000 년도 이전부터 발생해온 오래된 공격이고, 이를 방어하기 위한 다양한 DDoS 공격 방어 시스템을 구축하고 있음에도 불구하고 아직까지 이를 효과적으로 차단하지 못하고 있다. 이는 그간 DDoS 공격 방어 시스템이 공격이 시작되었을 때 개개의 공격 패턴을 탐지하고 차단하는 방법으로 발전해 왔기 때문이다. 따라서 좀 더 효과적인 DDoS 공격 대응을 위해 공격 트래픽이 발송되기 전에 클라이언트에서부터 공격에 대응하는 방안이 필요하다. 이에 본 논문에서는 사용자 인증을 통하여 등록된 사용자임을 검증하고, 클라이언트와 인증장비간의 인증을 통해 인증된 사용자의 트래픽만 허용하는 매커니즘을 제안한다.
안영근,정종갑,김주영,김정훈,김관수 한국환경독성학회 1987 환경독성보건학회지 Vol.2 No.1
The effects of ginseng ethanol extract on the toxicity of lead acetate in mice were examined. Mice were given intraperitoneally daily doses of lead acetate 50mg/kg with ginseng ethanol extract 50mg/kg, 100mg and 200mg/kg for 3 weeks. The exposure of lead acetate showed the toxicity at all experimental assay such as the gain of body weight, the ratio of some organs weight to body weight, serum transaminase activity and creatinine value, hematocrit and WBC counts. These toxicities were inhibited significantly by the ginseng ethanol extract administration. The 50mg/kg and 100mg/kg administration of ginseng ethanol extract inhibited histopathological changes on kidney by lead acetate, whereas the 200mg/kg administration of the fraction enhanced histopathological changes.
안영근,정종갑,김주영,김정훈,김관수 원광대학교 식품약품안전성연구소 1987 食品藥品安全性硏究 Vol.1 No.-
The effects of ginseng ethanol extract of the toxicity of lead acetate in mice were examined. Mice were given intraperitoneally daily doses of lead acetate 50㎎/㎏ with giseng ethanol extract 50㎎/㎏, 100㎎ and 200㎎/㎏ for 3 weeks. The exposure of leas acetate showed the toxicity at all experimental assay such as the gain of body weight, the ratio of some organs weight to body weight, serum transaminase activity and creatinine value, hematocrit and WBC counts. These toxicities were inhibited significantly by the ginseng ethanol extract administration. The 50㎎/㎏ and 100㎎/㎏ administration of ginseng ethanol extract inhibited histopathological changes on kidney by lead acetate, whereas the 200㎎/㎏ administration of the fraction enhanced histopathological changes.