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김한수,육현철 建陽大學校 1994 建陽論叢 Vol.- No.3
After measuring, comparing, and analyzing the backstroke kick start and the backstroke dolphin kick start 25m of the Korean excellent backstroke 10 swimers from November 4th 1989 to November 15th 1989, the conclusions of measuring and comparing the differences between dolphin kick 3-5-7-9-and-11 times and records are as follows: 1. According to the records and observations of start by the dolphin kick frequency of 10 backstroke swimers, the best records were produced from 5 swimers when doing start by 7 times of dolphin kick, 3 swiners were from 5 tims of dolphin kick, and 2 swimers were from 9 frequencies of dolphin kick. Therefore this suggests that 7 times in dolphin kick of the Korea backstroke men' swimers be the most profitable. 2. By comparing the dolphin kick start and backsroke kick start, dolphin kick as in figure 3 was mean 14"15, and backstroke kick start was 14"52. The dolphin kick start was faster .57 seconds than the backstroke kick start. 3. According to the conclusions of observing the swimers who do well dolphin kick start, swimers should have good back flexibility if they want to do well dolphin kick start, whereas the swimers with insufficient dolphin kick start did not have back flexibility.
세균감염에 의한 초생타조(Struthio camelus camelus)의 쇠약 증후군의 발생 증례
육현수 ( Hyun Su Yuk ),김영진 ( Young Jin Kim ),도홍기 ( Hong Ki Do ),노수일 ( Soo Il Roh ),김범석 ( Bum Seok Kim ),임채웅 ( Chae Woong Lim ) 한국가축위생학회 1999 韓國家畜衛生學會誌 Vol.22 No.2
The most common cause of death is ostrich chick fading syndrome(OCFS), which is due to bacterial infection during artificial incubation and hatching. Six farmed ostrich chicks aged 3 and 10 days in Chonbuk province, were submitted to Chonbuk Livestock Development and Research Institute for necropsy. Clinically, birds showed hair loss, ocular exudate, lethargy, diarrhea, and subsequently died 3-5 days after onset of clinical signs. Grossly, umbilicus was enlarged. White-yellowish purulent nodules were scattered on the lung and the membrane of air-sac was thickened and had inflamed exudate on the surface in two chicks that died 3 days after hatching. In 10 days-old chick, intestine was shown redding segmentally. Yolk sac was still retarded and its surface was partially hemorrahgic. The synovial fluid of the leg was yellowish. Microscopically, multifocal purulent exudates were scattered on the lung. Capillary microthrombi in the glomerulus were prominent and tubular epithelia were necrotic. Necrotic hepatocytes were scattered and intestine were congested. Microbiologically, Pseudomonas sp and/or E coli were isolated from air-sac, lung and/or liver. This case suggests that poor hygiene during artificial incubation, hatching or in the first week after hatching may cause high mortality of the ostrich chicks.
육현수 ( Hyun Su Yuk ),이오형 ( Oh Hyung Lee ),임병무 ( Byung Moo Rim ) 한국가축위생학회 2005 韓國家畜衛生學會誌 Vol.28 No.1
To examine the prevalence and type of lesions in urinary bladder of Korean cattle, a random sampling survey was performed at a Jeonju abattoir in September 2000 and January 2001. Collected urinary bladder were examined grossly and histopathologically and the patterns of disease were investigated with season and sex. Of 735 cattle(99 bulls and 246 cows in September and 141 bulls, 3 steers, and 246 cows in January) surveyed, 26.3% cattle with evident lesions were found. The most common gross finding was urolithiasis of urinary bladder(23.8%). Other lesions included hemorrhage(5.9%), congestion(4.1%), hematuria(2.7%), pyuria(0.7%), hyperplasia(0.5%). Urinary calculi collected in this study contained 80 to 90% of calcium oxalate and 10 to 20% of struvite, with or without 20% of cystine and 10% of uric acid. This study on urolithiasis and pathology of urinary bladder of slaughtered Korean cattle revealed subclinical aspects and management problem in Korean beef product.
Su-Jin Kim,Hyun-Gyun Yuk,Woo-Suk Bang 한국식품영양과학회 2021 한국식품영양과학회 학술대회발표집 Vol.2021 No.10
This study was to evaluate the bactericidal effects and antibacterial mechanism on Escherichia coli O157:H7, Salmonella Typhimurium, Listeria monocytogenes serovar 1/2a, and Staphylococcus aureus by combining blue light LED (405, 460 nm) and chitosan at low temperature. The D-values of E. coli O157:H7, S. Typhimurium, L. monocytogenes, and S. aureus irradiated with 405 nm LED at 4℃ were 5.7, 3.2, 1.0, and 2.1 h, respectively. Also, the D-values of combination treatment by 405 nm LED and 1% chitosan was significantly reduced to 2.1, 2.3, 0.4, and 1.4 h, respectively. Loss of efflux pump activity, damage to the glucose uptake system, and changes in membrane depolarization and permeability were observed by flow cytometry in all strains irradiated. Additionally, cell morphology of E. coli O157:H7 and L. monocytogenes were observed by transmission electron microscope (TEM). In the treated cells, the normal cellular structure was destroyed and the intracellular components were released. These results showed that the combination of 405 nm LED with chitosan (1%) caused severe damage to the cell membrane and cytoplasmic regions of bacterial cells, leading to cell death.
Chul-Su Yang,Jae-Min Yuk,Sung-Ryong Ko,Byung-Goo Cho,Hyun-Joo Sohn,Young-Sook Kim,Jae-Joon Wee,Jae-Ho Do,Eun-Kyeong Jo 고려인삼학회 2008 Journal of Ginseng Research Vol.32 No.4
In the previous studies, we isolated the compound K rich fractions (CKRF) and showed that CKRF inhibited Toll-like receptor (TLR) 4- or TLR9-induced inflammatory signaling. To extend our previous studies, we investigated the molecular mechanisms of CKRF in the TLR4-associated signaling via nuclear factor. (NF)-κB, and ill vivo role of CKRF for induction of tolerance in lipopolysaccharide (LPS)-induced septic shock. In murine bone marrow-dervied macrophages, CKRF significantly inhibited the induction of mRNA expression of proinflammatory mediators such as tumor necrosis factor-a, interleukin-6, cyclooxygenase-2, and inducible nitric oxide synthase. In addition, CKRF significantly attenuated the transcriptional activities of TLR4/LPS-induced NF-κB. Nuclear translocation of NF-κB in response to LPS stimulation was significantly abrogated by pre-treatment with CKRF. Furthermore, CKRF inhibited the recruitment of p65 to the interferon-sensitive response element tlanking region in response to LPS. Finally, oral administration of CKRF significantly protected mice from Gram-negative bacterial LPS-induced lethal shock and inhibited systemic inflammatory cytokine levels. Together, these results demonstrate that CKRF modulates the TLR4-dependent NF-κB activation, and suggest a therapeutic role for Gram-negative septic shock.