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에스트로겐과 다이옥신 수용체 효모를 이용한 내분비계장애 영향 평가
이병천 ( Byoung Cheun Lee ),김수진 ( Sue Jin Kim ),윤준헌 ( Jun Heon Yoon ),김은주 ( Eun Ju Kim ),( Duong NC ),엄익춘 ( Ig Chun Eom ),( Shiraishi F. ),최경희 ( Kyung Hee Choi ) 한국환경분석학회 2010 환경분석과 독성보건 Vol.13 No.2
Estrogenic and dioxin activities in river water and effluents of sewage treatment plants were evaluated by different in vitro assays. The expression of β-galatosidase in yeast cells (Yeast two-hybrid assay) was used as an indicator of pollutants effects. Estrogenic activities were estimated using medaka estrogen receptor (mER) and human estrogen receptor (hER) yeast while the dioxin activities were measured by Aryl hydrocarbon receptor (AhR) yeast. Five samples were collected including two river water (RW) samples and three effluents (MW) samples. Target chemicals were extracted using a series of solvents which are hexane/dichloromethane, acetone/dichloromethane and methanol. Standard dose-response curves were established with 3 estrogenic chemicals (estradiol, nonylphenol and bisphenol A) and 3 dioxin-like chemicals (naphthoflavone, comestrol and equol). The results showed that both estrogen receptors (i.e., mER and hER) were suitable to be applied into the assessment of micropollutants` estrogenicity, in which, the sensitivity of mER to estrogenic activity was higher than that of hER. AhR also showed a suitable sensitivity to target dioxins. Estrogenic activity of MW-1 was highest among the samples in both mER and hER yeast assays. The estrogenic activities of two other effluents samples, however, were lower than that of MW-1 and other RW samples. This indicates that river water may receive estrogenic-polluted water other than the effluents of STPs. Similarly, dioxin activities in RW samples were higher than that of effluents. It is suggested that the untreated agriculture wastewater could be the reason. This study contributes to the establishment and application of risk assessment of micropollutant in the environment.
사람의 정상 피부세포 및 폐세포의 발암에 미치는 2,3,7,8-tetrachlorodibenzo-ρ -dioxin의 영향
강미경(Mi Kyung Kang),염태경(Tai Kyung Ryeom),김강련(Kang Ryune Kim),김옥희(Ok Hee Kim),강호일(Hoil Kang) 한국환경성돌연변이발암원학회 2006 한국환경성돌연변이·발암원학회지 Vol.26 No.3
2,3,7,8-Tetrachlorodibenzo-ρ-dioxin (TCDD) displays high toxicity in animals and has been implicated in human carcinogenesis. Although TCDD is recognized as potent carcinogens, relatively little is known about their role in the tumor promotion and carcinogenesis. It is known that TCDD can increase of cancer risk from various types of tissue by a mechanism possibly involving the aryl hydrocarbon receptor (AhR) activation. In this study, effects of TCDD on cellular proliferation of normal human skin and lung fibroblasts, Detroit551 and WI38 cells were investigated. In addition, to enhance our understanding of TCDD-mediated carcinogenesis, we have investigated process in which expression of Erk1/2, cyclinD1, oncogene such as Ha-ras and c-myc, and their cognate signaling pathway. TCDD that are potent activators of AhR-mediated activity was found to induce significant increase of cytochrome P4501A1 mRNA expression, suggesting a presence of functional AhR. These results support that CYP1A1 enzyme may be involved in the generation of TCDD-induced toxicity. Moreover mitogen-activated protein kinases (MAPKs) phosphorylation and cyclin D1 overexpression are induced by TCDD, which corresponded with the progression of cellular proliferation. However, TCDD did not affected Haras and c-myc mRNA expression. Taken together, it seems that TCDD are could be a part of cellular proliferation in non-tumorigenic normal human cells such as Detroit551 and WI38 cells through the upregulation of MAPKs signaling pathway regulating growth of cell population. Therefore, AhR-activating TCDD could potentially contribute to tumor promotion and Detroit551 and WI38 cells have been used as a detection system of tumorigenic effects of TCDD.
다이옥신 배출 저감 기술 현황: 활성탄 주입 기술을 중심으로
채호진(Ho Jin Chae),조성빈(Seong Bin Jo),김태영(Tae Young Kim),이수출(Soo Chool Lee),김재창(Jae Chang Kim) 한국에너지기후변화학회 2020 에너지기후변화학회지 Vol.15 No.2
The incineration ratio of municipal waste is gradually increasing for the treatment of the rapidly increasing waste. Waste incineration has the risk of releasing various pollutants, but among them, the emission of dioxin is the most strongly regulated. The formation mechanism of the dioxins are classified by gas phase synthesis at high temperatures (homogeneous pathway) and de Novo synthesis at relatively low temperatures (heterogeneous pathway). High temperature synthesis is closely related to furnace, and low temperature generation is closely related to energy recovery units and/or air pollution control devices (APCDs). In order to minimize the generation of dioxins in the furnace, it was considered that homogenization of the waste, optimization of operating conditions, using inhibitors, etc. And the APCDs was considered including activated carbon injection method, decomposition method by catalytic reaction, and decomposition method using other electrical technique. In this paper, we will look around the recent technological trends in these emission reduction technologies, and discuss the latest research trends focused on the use of activated carbon injection + bag filter system, which are currently the most widely used emission reduction technologies.