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- 저자 : Yuanzhong Xu (검색결과 2 건)
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Yuanzhong Xu,Bohan Hu,Tiezhou Wu,Tingyi Xiao 전력전자학회 2022 JOURNAL OF POWER ELECTRONICS Vol.22 No.2
This paper proposes a joint estimation scheme for the state of charge (SoC) and state of health (SoH) for lithium-ion batteries in electric vehicles. The estimation accuracy is improved from four aspects. First, to overcome the shortcomings of the electrochemical model and equivalent circuit model, the battery model is established by a fractional order (FO) model. Second, a genetic algorithm is used to identify the model parameters, realizing optimal parameter identification. Third, the FO adaptive extended Kalman filter-based SoC estimator is developed, and the innovation accuracy of the algorithm is improved by multi- innovation theory. Fourth, the joint estimation of SoC and SoH is formulated through a multi-timescale framework. The proposed model and method are verified through dynamic operating condition experiments, and the main results are as follows. (1) In the entire SoC range, the accuracy of the FO model is better than that of the integer order (IO) model. (2) The effectiveness of the optimized SoC estimation method is verified, and the estimation error can be controlled within 3%. (3) The effectiveness of the proposed joint estimation method in dynamic conditions is verified, and it shows high accuracy.
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Li Xiaohua,Gong Yuanzhong,Lin Xin,Lin Qiong,Luo Jianxiong,Yu Tianxing,Xu Junping,Chen Lifang,Xu Liyu,Hu Ying 한국미생물학회 2022 The journal of microbiology Vol.60 No.4
Acute lung injury caused by Candida albicans could result in high mortality and morbidity. MicroRNA-155 (miR-155) and suppressor of cytokine signaling 1 (SOCS1) have been believed to play a key in the regulation of inflammatory response. Whether miR-155/SOCS1 axis could regulate the acute lung injury caused by C. albicans has not been reported. The acute lung injury animal model was established with acute infection of C. albicans. miR-155 inhibitor, miR-155 mimic, and sh-SOCS1 were constructed. The binding site between miR- 155 and SOCS1 was identified with dual luciferase reporter assay. Knockdown of miR-155 markedly inhibited the germ tube formation of C. albicans. Knockdown of miR-155 significantly up-regulated the expression of SOCS1, and the binding site between miR-155 and SOCS1 was identified. Knockdown of miR-155 improved the acute lung injury, suppressed inflammatory factors and fungus loading through SOCS1. Knockdown of SOCS1 greatly reversed the influence of miR- 155 inhibitor on the cell apoptosis in vitro. The improvement of acute lung injury caused by C. albicans, suppression of inflammatory response and C. albicans infection, and inhibitor of cell apoptosis were achieved by knocking down miR-155 through SOCS1. This research might provide a new thought for the prevention and treatment of acute lung injury caused by C. albicans through targeting miR-155/SOCS1 axis.
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