http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Kim, Hyung Joon,Cho, Sun Young,Cho, Woo Hee,Lee, Do Hyun,Lim, Do Hyoung,Seo, Pil Won,Park, Mi-Hyun,Lee, Wonae,Lee, Jai Hyuen,Kim, Doh Hyung The Korean Academy of Tuberculosis and Respiratory 2013 Tuberculosis and Respiratory Diseases Vol.75 No.5
Superior vena cava syndrome (SVCS) is usually caused by extrinsic compression or invasion of the superior vena cava (SVC) by malignant tumors involving mediastinal structures. Although thymomas are well-known causes of SVCS, cases of SVCS caused by malignant thymomas protruding into adjacent vessels draining the SVC with thrombosis have been very rarely reported worldwide. We experienced a 39-year-old female patient with SVCS that developed after the direct invasion of the left brachiocephalic vein (LBCV) and SVC by an anterior mediastinal mass with a high maximum standardized uptake value on the chest computed tomography (CT) and positron emission tomography-CT. Based on these results, she underwent en bloc resection of the tumor, including removal of the involved vessels, and was eventually diagnosed as having a type B2 thymoma permeating into the LBCV and SVC. We present this case as a very rare form of SVCS caused by an invasive thymoma.
( Hyung Joon Kim ),( Sun Young Cho ),( Woo Hee Cho ),( Do Hyun Lee ),( Do Hyoung Lim ),( Pil Won Seo ),( Mi Hyun Park ),( Wonae Lee ),( Jai Hyuen Lee ),( Doh Hyung Kim ) 대한결핵 및 호흡기학회 2013 Tuberculosis and Respiratory Diseases Vol.75 No.5
Superior vena cava syndrome (SVCS) is usually caused by extrinsic compression or invasion of the superior vena cava (SVC) by malignant tumors involving mediastinal structures. Although thymomas are well-known causes of SVCS, cases of SVCS caused by malignant thymomas protruding into adjacent vessels draining the SVC with thrombosis have been very rarely reported worldwide. We experienced a 39-year-old female patient with SVCS that developed after the direct invasion of the left brachiocephalic vein (LBCV) and SVC by an anterior mediastinal mass with a high maximum standardized uptake value on the chest computed tomography (CT) and positron emission tomography-CT. Based on these results, she underwent en bloc resection of the tumor, including removal of the involved vessels, and was eventually diagnosed as having a type B2 thymoma permeating into the LBCV and SVC. We present this case as a very rare form of SVCS caused by an invasive thymoma.
A Standardized Pathology Report for Gastric Cancer: 2nd Edition
Sung Hak Lee,Mee-Yon Cho,Young Soo Park,Myeong-Cherl Kook,Baek-Hui Kim,Hye Seung Lee,Dong-Wook Kang,Mi-Jin Gu,Ok Ran Shin,Younghee Choi,Wonae Lee,Hyunki Kim,In Hye Song,Kyoung-Mee Kim,Hee Sung Kim,Guh 대한위암학회 2023 Journal of gastric cancer Vol.23 No.1
The first edition of ‘A Standardized Pathology Report for Gastric Cancer’ was initiated by the Gastrointestinal Pathology Study Group of the Korean Society of Pathologists and published 17 years ago. Since then, significant advances have been made in the pathologic diagnosis, molecular genetics, and management of gastric cancer (GC). To reflect those changes, a committee for publishing a second edition of the report was formed within the Gastrointestinal Pathology Study Group of the Korean Society of Pathologists. This second edition consists of two parts: standard data elements and conditional data elements. The standard data elements contain the basic pathologic findings and items necessary to predict the prognosis of GC patients, and they are adequate for routine surgical pathology service. Other diagnostic and prognostic factors relevant to adjuvant therapy, including molecular biomarkers, are classified as conditional data elements to allow each pathologist to selectively choose items appropriate to the environment in their institution. We trust that the standardized pathology report will be helpful for GC diagnosis and facilitate large-scale multidisciplinary collaborative studies.
Root hair‐specific expansins modulate root hair elongation in rice
ZhiMing, Yu,Bo, Kang,XiaoWei, He,ShaoLei, Lv,YouHuang, Bai,WoNa, Ding,Ming, Chen,Hyung‐,Taeg, Cho,Ping, Wu Blackwell Publishing Ltd 2011 The Plant journal Vol.66 No.5
<P><B>Summary</B></P><P>Root hair growth requires intensive cell‐wall modification. This study demonstrates that root hair‐specific expansin As, a sub‐clade of the cell wall‐loosening expansin proteins, are required for root hair elongation in rice (<I>Oryza sativa</I> L.). We identified a gene encoding <I>EXPA17</I> (<I>OsEXPA17</I>) from a rice mutant with short root hairs. Promoter::reporter transgenic lines exhibited exclusive <I>OsEXPA17</I> expression in root hair cells. The OsEXPA17 mutant protein (OsexpA17) contained a point mutation, causing a change in the amino acid sequence (Gly104→Arg). This amino acid alteration is predicted to disrupt a highly conserved disulfide bond in the mutant. Suppression of <I>OsEXPA17</I> by RNA interference further confirmed requirement for the gene in root hair elongation. Complementation of the <I>OsEXPA17</I> mutant with other root hair EXPAs (OsEXPA30 and Arabidopsis EXPA7) can restore root hair elongation, indicating functional conservation of these root hair EXPAs in monocots and dicots. These results demonstrate that members of the root hair EXPA sub‐clade play a crucial role in root hair cell elongation in Graminaceae.</P>