http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
One step synthesis and densification of ultra-fine WC by high-frequency induction combustion
Kim, Hwan-Cheol,Shon, In-Jin,Yoon, Jin-Kook,Lee, Sang-Kwon,Munir, Z.A. Elsevier 2006 International journal of refractory metals & hard Vol.24 No.3
<P><B>Abstract</B></P><P>Dense WC with grain size of 0.43μm was synthesized by high-frequency induction combustion synthesis from milled elemental powders of W and C. The milled W powders had a grain size in the range 45–73nm. Dense product (98.5%) could be obtained within 2min under a pressure of 60MPa. Due to loss of carbon (by interaction with surface oxides), products made from stoichiometric powders (W:C=1:1) contained the sub-carbide W<SUB>2</SUB>C. With excess carbon, products containing the WC phase only were obtained. The effect of initial grain size (of W) and the W:C stoichiometry on the grain size of the product WC was investigated. The grain size of WC increased with an increase in the amount of excess carbon. The maximum values for fracture toughness and hardness obtained for the dense WC were 4.8MPam<SUP>1/2</SUP> and 2708kg/mm<SUP>2</SUP>, respectively.</P>
위암 및 전구병변에서 Cyclooxygenase-2와 p53의 발현
민상운 ( Sang Woon Min ),손영해 ( Young Hae Shon ),박창환 ( Chang Hwan Park ),이완식 ( Wan Sik Lee ),주영은 ( Young Eun Joo ),최성규 ( Sung Kyu Choi ),유종선 ( Jong Sun Rew ),박창수 ( Chang Soo Park ),김세종 ( Sei Jong Kim ) 대한소화기학회 2003 대한소화기학회지 Vol.41 No.4
Background/Aims: The mechanism that controls cyclooxygenase-2 (COX-2) expression is not clear yet. Recently, it is reported that COX-2 expression is significantly high in the presence of mutated p53 genes. Thus, we investigated the expression of COX-2 and p53 in each stage of chronic gastritis, intestinal metaplasia, dysplasia, and gastric cancer, and studied their correlations. Methods: The expressions of COX-2 and p53 in 128 local lesions were assessed immunohistochemically by using Micro-Probe System in a total of 101 patients. There were 23 chronic gastritis, 50 intestinal metaplasia, 34 dysplasia, and 21 intestinal type of gastric cancer. Results: The expression of COX-2 was increased in 30.4% (7/23) of chronic gastritis, 68% (34/50) of intestinal metaplasia, 82.3% (28/34) of dysplasia, and 85.7% (18/21) of intestinal type gastric cancer. The expression of COX-2 was significantly increased as the disease progressed into gastric cancer (p<0.001). On the other hard, p53 was expressed positively in 38% (8/21) of gastric cancer, and 5.9% (2/34) of dysplasia. Although the difference was not statistically significant, expression of COX-2 seemed to be increased in gastric cancer with positive p53. Conclusions: COX-2, one of the major factors involved in the gastric carcinogenesis, may be involved in the early stage. Additionally, co-expression of COX-2 and p53 may have a role in promoting gastric carcinogenesis. (Korean J Gastroenterol 2003;41:268-276)
슬관절에 히야루론산 주입후 발생한 활막성 연골종증 : 증례 보고
최의성,김용민,김동수,손현철,박경진,조병기,배승환 충북대학교 의과대학 충북대학교 의학연구소 2009 忠北醫大學術誌 Vol.18 No.2
활막성 연골종증은 비교적 드문 양성 종양으로 관절내 활액막 결체조직의 양성 반응성 이형성에 의해 여러 개의 연골성 결절을 형성하고 이것이 관절내로 유리되어 유리체를 형성하는 질환으로 주로 슬관절에 발생한다. 활막성 연골종증의 정확한 발생 기전은 아직 밝혀지지 않았으나 외상, 감염 등이 원인으로 제시되고 있다. 저자들은 슬관절 내 히야루론산 주입후 발생한 슬관절내 활막성 연골종증을 1 례 경험하였으며, 관절경적 제거술을 통해 만족스런 결과를 얻었다. As a relatively rare benign tumor, synovial chondromatosis forms several cartilaginous nodules by the benign reactive metaplasia of synovial connective tissues within joints, which are loosed into the joints and cause a disease forming loose body mainly in knee joint. Accurate mechanism of synovial chondromatosis has not been clarified yet; however, trauma, infections and others have been suggested as its possible causes. The authors of this study experienced an example of a patient with synovial chondromatosis in knee joint occurred after the injection of hyaluronic acid in the joints and had a satisfactory outcome through the arthroscopic resection.
( Kil Hwan Shon ),( Kyung Rim Sung ) 대한안과학회 2014 Korean Journal of Ophthalmology Vol.28 No.4
Purpose: To evaluate patterns of macular retinal ganglion cell (RGC) loss measured by spectral domain optical coherence tomography in patients with neurologic lesions mimicking glaucoma. Methods: We evaluated four patients with neurological lesions who showed characteristic patterns of RGC loss, as determined by ganglion cell thickness (GCT) mapping. Results: Case 1 was a 30-year-old man who had been treated with glaucoma medication. A left homonymous vertical pattern of RGC loss was observed in his GCT map and a past brain magnetic resonance imaging (MRI) revealed a hemorrhagic lesion around the right optic radiation. Case 2 was a 72-year-old man with a pituitary adenoma who had a binasal vertical pattern of RGC loss that corresponded with bitemporal hemianopsia. Case 3 was a 77-year-old man treated for suspected glaucoma. His GCT map showed a right inferior quadratic pattern of loss, indicating a right superior homonymous quadranopsia in his visual field (VF). His brain MRI revealed a left posterior cerebral artery territory infarct. Case 4 was a 38-year-old woman with an unreliable VF who was referred for suspected glaucoma. Her GCT map revealed a left homonymous vertical pattern of RGC loss, which may have been related to a previous head trauma. Conclusions: Evaluation of the patterns of macular RGC loss may be helpful in the differential diagnosis of RGC-related diseases, including glaucoma and neurologic lesions. When a patient’s VF is unavailable, this method may be an effective tool for diagnosing and monitoring transneuronal retrograde degeneration-related structural changes.