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한국인 산모에 있어서 임신중기 혈청 Alpha-fetoprotein치의 정상역
김승욱,신희철,윤보현,전종관,박교훈,구자남,지병철 대한산부인과학회 1995 Obstetrics & Gynecology Science Vol.38 No.12
1. 저자들은 1991년 7월부터 1994년 12월까지 임신15-20주 사이에 MSAFP 검사를 시행 받았던 산모중 단태 만삭아를 분만한 경우 429례를 대상으로 하여 MSAFP의 각 임신주수 별 중앙값 및 상하한선을 설정하였다. 2. MSAFP치의 중앙값은 임신 15, 16, 17, 18, 19 및 20주에 각각 33, 43, 48, 58, 63, 66ng/ml이었다. 3. MSAFP의 중앙값 및 상하한선은 선별검사를 시행하는 병원마다 나름대로의 기중치가 있어야 하며 더 많은 자료수집으로 주기적으로 계속 보강되어야 할 것으로 사료된다. Maternal serum alpha-fetoprotein(MSAFP) in mid-trimester pregnant women has been a world-wide screening program for congenital neural-tube defect and some typed of chromosomal anomaly, but a few data has been reported about the normal range of MSAFP in Korea. Analyzing pregnancy outcomes resulted in single, term living babies without any recognizable congenital anomalies during about 3 years of our clinical experience, we established the normal range of MSAFP according to the gestational age in mid-trimester ; 33, 43, 48, 58, 63, 66 ng/ml in gestational age at 15, 16, 17, 18, 19 and 20th week, respectively. Se recommend every hospital must have their own data about the normal range of MSAFP for the effective screening program and this may be revised periodically.
이우영,조숙,고승권,황성욱,김준미,박지현,원영석,이정례,지용일 대한산부인과학회 1998 Obstetrics & Gynecology Science Vol.41 No.6
저자들은 세계적으로 드물며, 우리 나라에서도 보고된 예가 없는 후복막에 발생한 원발성 장액성 암종 1예를 경험하였기에 이를 문헌 고찰과 함께 보고하는 바이다. Primary retroperitoneal serous carcinoma is extremely rare tumor. It is histologically identical to ovarian serous papillary carcinoma but without any evidence of ovaries, pancreas, gallbladder, appendix or abdominal origin. Histogenesis of this neoplasm remains uncertain. Ectopic ovarian tissue or coelomic metaplasia of retroperitoneal mesothelium has been proposed as its origin. We experienced a case of primary retroperitoneal serous carcinoma in an 53-year-old woman. Therefore, we present this case with a brief review of the literature.
Chang, Seok Woo,Kim, Yu-Chan,Chang, Hyejung,Jee, Kwang-Koo,Zhu, Qiang,Safavi, Kamran,Shon, Won-Jun,Bae, Kwang-Shik,Spangberg, Larz Sw,Kum, Kee-Yeon Informa Healthcare 2013 Acta odontologica scandinavica Vol.71 No.6
<P><B><I>Objective.</I></B> The aim of this study was to investigate the effect of heat treatment on the cyclic fatigue resistance, thermal behavior and microstructural changes of K3 NiTi rotary instruments. <B><I>Materials and methods.</I></B> Twelve control (as-received) and 12 experimental (heat-treated) K3 NiTi rotary instruments were compared in this study. Those experimental K3 instruments were heated in a furnace for 30 min at 450°C and then quenched in water. The cyclic fatigue resistance was measured with a fatigue tester. The thermal characteristic and the microstructures of both instruments were investigated by differential scanning calorimetry (DSC) and transmission electron microscopy (TEM), respectively. <B><I>Results.</I></B> There was a significant increase in the cyclic fatigue resistance between the heat-treated instruments and the as-received instruments (<I>T</I>-test, <I>p</I> < 0.05). DSC showed that the as-received and heat-treated samples were different, with an increased A<SUB>f</SUB> (austenite-finish temperature) for the latter. TEM analysis revealed that both as-received and heat-treated instruments were composed mainly of an austenite phase. However, the heat-treated samples had an increased appearance of larger grains, twinning martensite, TiO<SUB>2</SUB> surface layer and a Ni-rich inner layer. <B><I>Conclusions.</I></B> Heat treatment increased the cyclic fatigue resistance of NiTi files and changed the thermal behavior of the instruments without marked changes in the constituting phases of NiTi alloy.</P>
DY Hwang,JH oh,YK Kim,SB Shim,SW Jee,SH Lee,SJ Seo,YS Song,KT Nam,JY Cho,Jane Hwang,IS Jang,JS Cho 한국실험동물학회 2005 Laboratory Animal Research Vol.21 No.2
Type 2 or noninsulin-dependent diabetes mellitus accounts for over 90% of cases and is characterized by a triad of resistance to insulin action on glucose uptake in peripheral tissues, impaired insulin action to inhibit hepatic glucose production, and dysregulated insulin secretion. To determine whether a transgene-based small interfering RNA (siRNA) for insulin and an overexpression of human insulin degrading enzyme (hIDE) decrease the insulin level in vitro and in vivo or not, several siRNA sequences and hIDE gene for insulin were overexpressed in the insulinoma cells and mouse via tail vein. The siRNAs for insulin suppression were very effective at the regions of 88-99 bp and 109-131 bp in rat insulin sequence. Insulin suppression by the specific siRNA sequences and hIDE significantly induced endoplasmic reticulumn (ER) stress and decreased insulin receptor a expression in insulinoma cells. The insulin level in plasma significantly decreased by overexpressions of the siRNAs and hIDE gene in mouse, which may be caused by the degradation of insulin due to overexpression of hIDE protein in mouse liver. These results suggest that this plasmid for the suppression of insulin secretion by the specific siRNAs and the insulin degradation by an overexpression of hIDE protein may be useful on the development of animal model for Type 2 diabetes.