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남혜영(Hyeyoung Nam),김욱현(Wookhyun Kim) 大韓電子工學會 2009 電子工學會論文誌-SP (Signal processing) Vol.46 No.6
In this paper we propose the block based image segmentation method using the cyclic properties of hue components in HSI color model. In proposed method we use center point instead of hue mean values as the hue representatives for regions in image segmentation considering hue cyclic properties and we also use directed distance for the hue difference among regions. Furthermore we devise the simple and effective method to get critical values through control parameter to reduce the complexity in the calculation of those in the conventional method. From the experimental results we found that the segmented regions in the proposed method is more natural than those in the conventional method especially in texture and red tone regions. In the simulation results the proposed method is better than the conventional methods in the in the evaluation of the human segmentation dataset presented Berkely Segmentation Database. 본 논문은 HSI 컬러 모델에서 색상(Hue)의 주기성을 고려한 블록기반 영상분할 방법을 제안한다. 제안한 방법에서는 영역병합 시 사용되는 영역의 색상 대푯값으로 색상의 평균 대신 중앙점을 사용하며, 영역 간 색상차를 계산하기 위해 단방향 거리를 사용한다. 그리고 기존방법에서 사용한 복잡하게 계산된 영역별 임계값을 파라메타를 통해 조절할 수 있는 간단하지만 효율적인 임계값으로 수정한다. 실험결과 제안한 방법의 분할결과가 질감 성분이나 붉은 색상을 가진 영역에서 기존 방법을 사용했을 때 보다 더 자연스러우며, 제안한 방법과 기존 방법으로 버클리 영상분할 데이터베이스에서 제공하는 자연영상들을 분할하여 평가값을 비교해 본 결과 제안한 방법이 기존방법에 비해서 더 우수함을 알 수 있었다.
Hyeyoung Kim,Kwangsoo Nam,Kyung Hwan Kim 대한생리학회-대한약리학회 1998 The Korean Journal of Physiology & Pharmacology Vol.2 No.4
<P> This study investigated the role of nitric oxide on the oxidative damage in gastric mucosa of rats which received ischemia/reperfusion and its relation to mucus. Nitric oxide synthesis modulators such as L-arginine and N<SUP>G</SUP>-nitro-L-arginine methyl ester, and sodium nitroprusside, a nitric oxide donor, were injected intraperitoneally to the rats 30 min prior to ischemia/reperfusion which was induced by clamping the celiac artery and the superior mesenteric artery for 30 min and reperfusion for 1 h. Lipid peroxide production, the contents of glutathione and mucus, and glutathione peroxidase activities of gastric mucosa were determined. Histological observation of gastric mucosa was performed by using hematoxylin-eosin staining and scanning electron microscopy. The result showed that ischemia/reperfusion increased lipid peroxide production and decreased the contents of glutathione and mucus as well as glutathione peroxidase activities of gastric mucosa. Ischemia/reperfusion induced gastric erosion and gross epithelial disruption of gastric mucosa. Pretreatment of L-arginine, a substrate for nitric oxide synthase, and sodium nitroprusside prevented ischemia/reperfusion-induced alterations of gastric mucosa. However, N<SUP>G</SUP>-nitro- L- arginine methyl ester, a nitric oxide synthase inhibitor, deteriorated oxidative damage induced by ischemia/reperfusion. In conclusion, nitric oxide has an antioxidant defensive role on gastric mucosa by maintaining mucus, glutathione, and glutathione peroxidase of gastric mucosa.
Graft-Versus-Host Disease after Liver Transplantation
Hyeyoung Kim,Nam-Joon Yi,Kyung-Suk Suh,Geon Hong,Young Min Jeon,Kwang-Woong Lee,Myung Hee Park,Eung-Ho Cho,Kuhn Uk Lee 한국간담췌외과학회 2010 한국간담췌외과학회지 Vol.14 No.4
Purpose: Graft-Versus-Host Disease (GVHD) is a rare (0.1∼2%) but severe complication after liver transplantation (LT). It is the most lethal complication after LT and there are currently no effective preventive or therapeutic measures available. Approximately 90 such cases have been reported in the literature, but only one case has been reported in Korea. Methods: We performed a retrospective analysis of 767 patients who underwent LT (living donor : deceased donor=554 : 213) at Seoul National University Hospital, Korea from 1998 to 2009. Four patients (4/767, 0.52%) with histologically proven GVHD were found. The diagnosis of GVHD was made according to observing macrochimerism in the peripheral blood and the affected tissue biopsy. Results: Four patients underwent LT due to Hepatitis B virus-related liver cirrhosis and two of these patients had coexisting hepatocellular carcinoma. Three patients received livers from deceased donors and one received a liver from a live donor. All their blood matching were identical. The first diagnosed case underwent human leukocyte antigen (HLA) typing only after LT and it showed complete one-way donor-recipient HLA matching. The onset of GVHD occurred between 10 days and 55 days after LT. All the patients developed high-grade fever, skin rash, neutropenia, diarrhea and the main signs and symptoms related to GVHD. All the patients died because of sepsis despite intensive treatment. Conclusion: GVHD after LT is an extremely rare and fatal complication and it is difficult to diagnose. Therefore, we should perform pre-transplant HLA matching and try to establish an early diagnosis for patients who are clinical suspicious of having GVHD. Further study in this area is needed and physicians need to be alert to detect this malady.
( Hyeyoung Kim ),( Kwang-woong Lee ),( Nam-joon Yi ),( Hae Won Lee ),( Youngrok Choi ),( Hyo-sin Kim ),( Kyung Chul Yoon ),( Suk Kyun Hong ),( Rovgaliyev B. ),( Kyung-suk Suh ) 대한간학회 2016 춘·추계 학술대회 (KASL) Vol.2016 No.1
Aims: The patient with large splenorenal shunt (LSRS) is challenging at liver transplantation (LT), irrespective of organizing portal vein (PV) thrombus. Here, we report the clinical outcomes of 17 patients who received direct LSRS ligation during LT. Methods: We reviewed patients who underwent LT and intraoperative direct ligation of LSRS between Jan. 2010 and Jun. 2013. Results: Among 580 recipients, 17 patients underwent intraoperative direct ligation of LSRS. Pre-LT MELD score was 15.4 ± 6.6 (7 - 33). Main PV diameter on preoperative imaging was mean 7.6 ± 3.1 (3.0 - 13.9) mm. PV thrombectomy was done in 41.2% of patients (n = 7). Except one hospital mortality, 16 patients showed favorable outcome (94.1%). The mortality was related with sepsis, but not with liver dysfunction. There were 2 patients (11.8%) of major complication (Clavien-Dindo grade ≥ IIIa): splenic artery embolization for massive ascites control (#1), and reoperation due to ligation of left renal vein instead of LSRS at the time of LT. Including the patient of splenic artery embolization (#1), massive and prolonged ascites after LT was in 23.5% of patients (n = 4) with small diameter of PV (< 7.5mm). They were living donor recipients, and not related with pre-LT ascites. Except the patient (#1), in the other three, ascites was tolerable and well controlled by conservative manage. After the patient (#1), we performed test clamp of LSRS before direct ligation in small diameter of PV, and applied PV pressure monitoring in patients who showed a sign of portal hypertension such as bowel edema. Three patients underwent total or partial ligation under PV pressure monitoring (within 8mmHg of pressure difference before and after ligation of LSRS). Total 16 patients have maintained normal liver function until last follow-up (94.1%). Conclusions: Direct ligation of LSRS during LT is a safe and effective method to overcome the effects of LSRS. However, meticulous care is needed in isolation and ligation of LSRS. Selective simultaneous intraoperative portal pressure monitoring can be helpful for prevention of severe portal hypertension.