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Kwon, Hyeok Gyu,Jang, Sung Ho Taylor Francis 2014 Brain injury Vol.28 No.4
<P>Many studies have demonstrated neural injury in patients with mild traumatic brain injury, using diffusion tensor imaging (DTI). However, knowledge regarding injury of the corticoreticular pathway (CRP) is limited. This study reports on a patient with mild TBI who showed delayed gait disturbance due to injury of the CRP following head trauma, which was demonstrated by DTI.</P>
Suppression of pine wilt disease by an antibacterial agent, oxolinic acid
Kwon, Hyeok Ran,Choi, Gyung Ja,Choi, Yong Ho,Jang, Kyoung Soo,Sung, Nack-Do,Kang, Mun Seong,Moon, Yilseong,Lee, Seung Kyu,Kim, Jin-Cheol John Wiley Sons, Ltd. 2010 PEST MANAGEMENT SCIENCE Vol.66 No.6
<P>BACKGROUND: Pine wilt disease (PWD) is very complex and has been reported to be caused by pine wood nematode, Bursaphelenchus xylophilus (Steiner & Buhrer) Nickle, and its accompanying bacteria. However, there is no report on the control of PWD by antibacterial agent. The present study was performed to investigate disease control efficacy of antibacterial agents against PWD.</P><P>RESULTS: Among six antibacterial antibiotics tested, oxolinic acid (OA) showed the strongest antibacterial activity against five bacteria isolated from three strains of pine wood nematode. In in vivo assay, it effectively suppressed the development of PWD in three-year-old seedlings of Pinus densiflora Sieb. & Zucc.; it showed 71% control when injected at 3 mg per seedling. A mixture of OA and the nematicidal agent abamectin (Ab) showed higher disease control efficacy against PWD than either OA or Ab alone. In addition, OA alone and a mixture of OA and Ab also controlled PWD in approximately 20-year-old pine trees under field conditions.</P><P>CONCLUSION: This is the first report on the suppression of PWD by OA. The result strongly indicates that PWD could be controlled by antibacterial antibiotic alone and a combination of antibacterial and nematicidal agents. Copyright © 2010 Society of Chemical Industry</P>
Basic, HCCbasic : PO-21 ; Increased apolipoprotein E in hepatocellular carcinoma
( Hyeok Choon Kwon ),( Seung Woo Nam ),( Jae Youn Cheong ),( Soon Sun Kim ),( Marie Yeo ),( Dong Kyu Kim ),( Sung Won Cho ) 대한간학회 2012 춘·추계 학술대회 (KASL) Vol.2012 No.1
Background: Hepatocellular carcinoma (HCC) is one of the most malignant tumors worldwide. The goal of our study was to identify new biomarkers for HCC for early detection and for gaining an improved understanding of hepatocarcinogenesis by performing a comparative proteomic analysis of HCC and adjacent non-tumor tissue. Methods: Pairs of tumor and adjacent non-tumor tissue were obtained from 167 HCC patients after surgical resection at Ajou University Hospital, Korea. We assessed the histopathological features and additional clinical data. Proteomic analysis of tumor and non-tumor tissue revealing differential expression using 2D electrophoresis and MALDI-TOF MS analysis reveals some xenobiotic enzymes and apo E was selected. The associations between the expression of apoE and the clinical and pathological parameters were analyzed using SPSS. Results: One of the xenobiotic enzymes, apoE was significantly increased in tumor tissue compared with surrounding nontumor tissue. Paired T-test showed significant high level of apoE in tumor compared with non-tumor (209.91 vs 87.59, p < .01). Oneway ANOVA revealed that levels of apoE were elevated independently among different Child classes, etiologies, sex and consumption of alcohol. Conclusions: We compared the protein profiles between tumor and non-tumor tissue in HCC patients. This study revealed that apoE was significantly higher in HCC than in non-tumor tissue. The multivariate analysis confirmed apoE was higher in regardless of Child class, etiology, sex and alcohol consumption. ApoE has long been known as plasma lipoproteins responsible for cholesterol transport and metabolism. Plasma apoE is produced mainly in the liver but also in the brain, adrenal glands, kidney, and macrophage and not increased in gene expression and serum level in patients with HCC. Some proteomic studies reports a strong relationship between other cancers and apoE. We suppose that this report add a clue to understand hepatocarcinogenesis from hepatic steatosis to consequently hepatic carcinoma.
Kwon, Hyeok Choon,Cheong, Jae Youn,Cho, Sung Won,Choi, Jae Myoung,Hong, Sun Pyo,Kim, Soo-Ok,Yoo, Wang Don Blackwell Publishing Asia 2009 Journal of gastroenterology and hepatology Vol.24 No.1
<P>Abstract</P><P>Background: </P><P>To evaluate the effect of reversion to YMDD wild-type on emergence of adefovir (ADV)-resistant mutation and antiviral activity of ADV in lamivudine (LAM)- resistant patients.</P><P>Methods: </P><P>We determined YMDD mutations and ADV-resistant mutations before and every 3 months during ADV monotherapy in 33 LAM-resistant patients using the restriction fragment mass polymorphism (RFMP) method.</P><P>Results: </P><P>Reversion to pure YMDD wild-type hepatitis B virus (HBV) occurred in 6% (2/33), 9% (3/33), 20% (4/20) and 35% (6/17) of patients after 12, 24, 36 and 48 weeks, respectively. Five (29%) patients were found to have pure YMDD mutants at 48 weeks of therapy. Among 33 patients, 4 (12%) patients developed ADV-resistant mutations at 48 weeks of therapy. Adefovir-resistant mutants emerged in all patients after reversion to YMDD wild-type HBV. The mean serum HBV reductions, evaluated at 24 weeks of therapy, were not different between patients with and without reversion to YMDD wild-type HBV (−3.1 log<SUB>10</SUB> copies/mL <I>vs</I>−3.4 log<SUB>10</SUB> copies/mL, <I>P</I> > 0.05).</P><P>Conclusions: </P><P>ADV-resistant mutations emerged after reversion to YMDD wild-type in LAM-resistant patients who received ADV monotherapy. Thus, ADV add-on therapy may be necessary to reduce the incidence of developing ADV resistance in patients with LAM resistance.</P>
Dependence of the RFQ Output Beam Current on the Proton Injector Operation Parameters
Hyeok-Jung Kwon,Han-Sung Kim,Dae-Il Kim,Ji-Ho Jang,Yong-Sub Cho 한국물리학회 2009 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.54 No.5
A 20-MeV proton accelerator has been developed by the Proton Engineering Frontier Project (PEFP). It consists of a 50-keV proton injector, a 3-MeV radio-frequency quadrupole (RFQ) and a 20-MeV drift tube linac (DTL). An ion source and a low-energy beam transport (LEBT) system constitute the proton injector. The main roles of the proton injector are to generate the proton beams and to transport and match the beam from the ion source into the RFQ. The output beam current and the transmission of the RFQ are directly influenced by the operation parameters of the proton injector, especially the two solenoid magnets. The output beam current of the RFQ was measured for various values of the solenoid magnet current and the results were compared with that of the simulation.