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Enhance Health Risks Prediction Mechanism in the Cloud Using RT-TKRIBC Technique
Konduru, Venkateswara Raju,Bharamgoudra, Manjula R The Korea Institute of Information and Commucation 2021 Journal of information and communication convergen Vol.19 No.3
A large volume of patient data is generated from various devices used in healthcare applications. With increase in the volume of data generated in the healthcare industry, more wellness monitoring is required. A cloud-enabled analysis of healthcare data that predicts patient risk factors is required. Machine learning techniques have been developed to address these medical care problems. A novel technique called the radix-trie-based Tanimoto kernel regressive infomax boost classification (RT-TKRIBC) technique is introduced to analyze the heterogeneous health data in the cloud to predict the health risks and send alerts. The infomax boost ensemble technique improves the prediction accuracy by finding the maximum mutual information, thereby minimizing the mean square error. The performance evaluation of the proposed RT-TKRIBC technique is realized through extensive simulations in the cloud environment, which provides better prediction accuracy and less prediction time than those provided by the state-of-the-art methods.
Quantitative measurements of nanoscale thin frost layers using surface plasmon resonance imaging
Jeong, Chan Ho,Shin, Dong Hwan,Konduru, Vinaykumar,Allen, Jeffrey S.,Choi, Chang Kyoung,Lee, Seong Hyuk Elsevier 2018 INTERNATIONAL JOURNAL OF HEAT AND MASS TRANSFER - Vol.124 No.-
<P><B>Abstract</B></P> <P>This study reports the presence of a nanoscale thin frost layer. During the frosting process, the surface plasmon resonance (SPR) imaging method can be used to overcome conventional optical limits and quantify this layer. The research outlined here also provides quantitative thickness measurement of the thin frost layer via a proposed calibration method based on the measured SPR intensity. The SPR system established in this study consists of a 50 nm gold-coated BK7 cover glass, a prism, a light source, a polarizer, a lens and a filter for the collimated light of a 600 ± 5 nm wavelength, and a CCD camera. The SPR angle of the ice phase is 72°, which corresponds to the ice refractive index of 1.307. The gold-glass specimen is cooled from room temperature (23 ± 1 °C) to −4.0 ± 0.8 °C by using a thermoelectric cooler to maintain the relative humidity of 20 ± 3% (at the room temperature). As a result, it is found that the nanoscale thin frost layer between the frozen condensates exists on the surface. Also, the present study yields the spatial distribution of reflectance that is associated with the frost layer thickness, indicating that the local information about thin frost layer thickness can be obtained through this SPR imaging method. It is found that the SPR imaging method enables successful capture of the depthwise spatial variations of the thin frost layer, showing that the frost layer was grown over time as a result of the de-sublimation of water vapor.</P> <P><B>Highlights</B></P> <P> <UL> <LI> Quantitative measurements of nanoscale thin frost layers are conducted. </LI> <LI> The surface plasmon resonance imaging enables successfully to capture the existence of frost layers. </LI> <LI> The frost layer is grown over time due to the de-sublimation of water vapor. </LI> </UL> </P>
EVI1 acts as an inducible negative-feedback regulator of NF-κB by inhibiting p65 acetylation.
Xu, Xiangbin,Woo, Chang-Hoon,Steere, Rachel R,Lee, Byung Cheol,Huang, Yuxian,Wu, Jing,Pang, Jinjiang,Lim, Jae Hyang,Xu, Haidong,Zhang, Wenhong,Konduru, Anuhya S,Yan, Chen,Cheeseman, Michael T,Brown, S Williams Wilkins 2012 JOURNAL OF IMMUNOLOGY Vol.188 No.12
<P>Inflammation is a hallmark of many important human diseases. Appropriate inflammation is critical for host defense; however, an overactive response is detrimental to the host. Thus, inflammation must be tightly regulated. The molecular mechanisms underlying the tight regulation of inflammation remain largely unknown. Ecotropic viral integration site 1 (EVI1), a proto-oncogene and zinc finger transcription factor, plays important roles in normal development and leukemogenesis. However, its role in regulating NF-κB-dependent inflammation remains unknown. In this article, we show that EVI1 negatively regulates nontypeable Haemophilus influenzae- and TNF-α-induced NF-κB-dependent inflammation in vitro and in vivo. EVI1 directly binds to the NF-κB p65 subunit and inhibits its acetylation at lysine 310, thereby inhibiting its DNA-binding activity. Moreover, expression of EVI1 itself is induced by nontypeable Haemophilus influenzae and TNF-α in an NF-κB-dependent manner, thereby unveiling a novel inducible negative feedback loop to tightly control NF-κB-dependent inflammation. Thus, our study provides important insights into the novel role for EVI1 in negatively regulating NF-κB-dependent inflammation, and it may also shed light on the future development of novel anti-inflammatory strategies.</P>