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Poppe, A.R.,Fatemi, S.,Garrick-Bethell, I.,Hemingway, D.,Holmstrom, M. Academic Press 2016 Icarus Vol.266 No.-
<P>Remanent magnetization has long been known to exist in the lunar crust, yet both the detailed topology and ultimate origin(s) of these fields remains uncertain. Some crustal magnetic fields coincide with surface albedo anomalies, known as lunar swirls, which are thought to be formed by differential surface weathering of the regolith underlying crustal fields due to deflection of incident solar wind protons. Here, we present results from a three-dimensional, self-consistent, plasma hybrid model of the solar wind interaction with two different possible source magnetizations for the Reiner Gamma anomaly. We characterize the plasma interaction with these fields and the resulting spatial distribution of charged-particle weathering of the surface and compare these results to optical albedo measurements of Reiner Gamma. The model results constrain the proposed source magnetizations for Reiner Gamma and suggest that vertical crustal magnetic fields are required to produce the observed 'dark lanes.' (c) 2015 Elsevier Inc. All rights reserved.</P>
Sun, N.,Yun, J.,Liu, J.,Malide, D.,Liu, C.,Rovira, Ilsa I.,Holmstrom, Kira M.,Fergusson, Maria M.,Yoo, Y.,Combs, Christian A.,Finkel, T. Cell Press 2015 Molecular Cell Vol.60 No.4
Alterations in mitophagy have been increasingly linked to aging and age-related diseases. There are, however, no convenient methods to analyze mitophagy in vivo. Here, we describe a transgenic mouse model in which we expressed a mitochondrial-targeted form of the fluorescent reporter Keima (mt-Keima). Keima is a coral-derived protein that exhibits both pH-dependent excitation and resistance to lysosomal proteases. Comparison of a wide range of primary cells and tissues generated from the mt-Keima mouse revealed significant variations in basal mitophagy. In addition, we have employed the mt-Keima mice to analyze how mitophagy is altered by conditions including diet, oxygen availability, Huntingtin transgene expression, the absence of macroautophagy (ATG5 or ATG7 expression), an increase in mitochondrial mutational load, the presence of metastatic tumors, and normal aging. The ability to assess mitophagy under a host of varying environmental and genetic perturbations suggests that the mt-Keima mouse should be a valuable resource.