http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
오늘 본 자료
Properties of Fe-based Cr3C2 Coatings Produced with a High-Velocity Arc-Spraying Process
Can-Ming Wang,Hong-Fei Sun,Zhiyan Qu,Qiang Song 한국물리학회 2009 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.54 No.3
Fe-based Cr3C2 coatings were sprayed with a high-velocity spraying process. The main phases of the coatings were α-Fe, (Fe, Cr)23C6, (Fe, Cr)7C3, FeCr2O4, Cr3C2 and a small amount of Fe3Si. The abrasive wear performances of the coatings were tested by using a rubber wheel abrasive tester. The wear mechanisms of the coatings were mainly plastic cutting and brittle fracture of some ceramic phases. At 650℃ the erosion rate of the coatings had a peak value at 40℃ impingement angles. The coatings exhibited excellent erosion resistance at high temperatures and were suitable for use in circulating fluid-bed boilers. Fe-based Cr3C2 coatings were sprayed with a high-velocity spraying process. The main phases of the coatings were α-Fe, (Fe, Cr)23C6, (Fe, Cr)7C3, FeCr2O4, Cr3C2 and a small amount of Fe3Si. The abrasive wear performances of the coatings were tested by using a rubber wheel abrasive tester. The wear mechanisms of the coatings were mainly plastic cutting and brittle fracture of some ceramic phases. At 650℃ the erosion rate of the coatings had a peak value at 40℃ impingement angles. The coatings exhibited excellent erosion resistance at high temperatures and were suitable for use in circulating fluid-bed boilers.
H-형태 양친매성 펜타블록 공중합체의 화학효소적 합성과 자기회합거동 평가
Peng Chen,Ya Peng Li,Can Jin Li,Xin Lei Meng,Bao Zhang,Ming Zhu,Yan-jing Liu,Jing Yuan Wang 한국고분자학회 2013 폴리머 Vol.37 No.3
H-shaped amphiphilic pentablock copolymers (PSt)2-b-PCL-b-PEO-b-PCL-b-(PSt)2 was synthesized via chemoenzymatic method by combining enzyme-catalyzed ring-opening polymerization (eROP) of ε-caprolactone (ε-CL) and atom transfer radical polymerization (ATRP) of styrene. By this process, we obtained copolymers with controlled molecular weight and low polydispersity. The structure and composition of the obtained copolymers were characterized by nuclear magnetic resonance (NMR), gel permeation chromatography (GPC) and infrared spectroscopy analysis (IR). The crystallization behavior of the copolymers was analyzed by differential scanning calorimetry (DSC) and X-ray diffraction (XRD). The crystallization behavior of the H-shaped block copolymers demonstrated a PCL dominate crystallization. The self-assembly behavior of the copolymers was investigated in aqueous media. The hydrodynamic diameters of the copolymer micelles in aqueous solution were measured by dynamic light scattering (DLS). The morphology of the copolymer micelles was observed by atomic force microscopy (AFM) and transmission electron microscopy (TEM). The hydrodynamic diameters of spherical micelles declined gradually with the increase of the hydrophobic chain lengths of the copolymers. The critical micelle concentration (CMC) values were determined from fluorescence emission, and it was found that the CMCs decreased with an increase of PSt hydrophobic block lengths.
Lei Chen,Kui-Po Yan,Xin-Can Liu,Wei Wang,Chao Li,Ming Li,Chun-Guang Qiu 대한약학회 2018 Archives of Pharmacal Research Vol.41 No.1
This study investigated the interaction amongvalsartan (VAL), TGF-b pathways, and long non-codingRNA (lncRNA) cardiac hypertrophy-related factor (CHRF)in doxorubicin (DOX)-induced heart failure (HF), andexplored their roles in DOX-induced HF progression. HFmice models in vivo were constructed by DOX induction. The expression of CHRF and TGF-b1 in hearts wasdetected, along with cardiac function, caspase-3 activity,and cell apoptosis. Primary myocardial cells were pretreatedwith VAL, followed by DOX induction in vitro forfunctional studies, including the detection of cell apoptosiswith terminal deoxynucleotidyl transferase dUTP nick-endlabeling and the expression of proteins associated withTGF-b1 pathways. HF models were established in vivo andin vitro. Expression of CHRF and TGF-b1 was up-regulated,and cell apoptosis and caspase-3 activity wereincreased in the hearts and cells of the HF models. VALsupplementation alleviated the cardiac dysfunction andinjury in the HF process. Moreover, overexpressed CHRFup-regulated TGF-b1, promoted myocardial cell apoptosis,and reversed VAL’s cardiac protective effect, while interferenceof CHRF (si-CHRF) did the opposite. Down-regulationof CHRF reversed the increased expression of TGFb1and the downstream proteins induced by pcDNA-TGFb1in HL-1 cells, while overexpression of CHRF reversedthe VAL’s cardiac protective effect in vivo. In conclusion,VAL regulates TGF-b pathways through lncRNA CHRF toimprove DOX-induced HF.