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황태식,나병관,Hung Thuan Tran,안대희,박두현 한국생물공학회 2008 Biotechnology and Bioprocess Engineering Vol.13 No.6
In this study, a novel three-compartmented electrochemical bioreactor (3-CEB) was designed in an effort to overcome the disadvantages of the two-compartmented electrochemical bioreactor (2-CEB) separated with a cation-selective membrane for enrichment of strict anaerobes. The 3-CEB was comprised of an anode, outlet, and a cathode compartment. The outlet compartment was positioned between the anode and cathode compartment, and it was separated with the anode side by a rubber plate and with the cathode side by a porous glass membrane. A platinum wire bridging the anode and outlet compartment operated as a redox passage, however, through which no material could permeate. Butyrate fermentation bacteria were enriched on the basis of the metabolite production. Butyrate generated by strict anaerobes was significantly more abundant in the 3-CEB than in the 2-CEB. Acetic acid and lactic acid generated by facultative anaerobes was relatively higher in the 2-CEB than in the 3-CEB. Meanwhile, butyrate was not generated in the bioreactor utilized for the control test, to which the electrochemical potential was not charged. In a continuous culture using the 3-CEB, the majority of the glucose was fermented to butyrate, and the acetate additionally supplied to the bacterial culture was metabolically reduced to butyrate. More lactate than butyrate was generated from glucose in the 2-CEB.
태반조직 배양에서 β₂-Adrenergic Agonist가 Progesterone 분비에 미치는 영향
황태식,안재영,김성도,이숙환,김종민,양성우,엄수하 대한산부인과학회 1990 Obstetrics & Gynecology Science Vol.33 No.7
조기진통 억제제로 쓰이는 β₂-adrenergic agonist인 terbutaline이 progesterone 분비에 미치는 영향을 보기 위하여 임신 38주 이후에 분만된 태반을 조직배양하여 다음과 같은 결과를 얻었다. 1. 태반조직 배양에서 β₂-adrenergic agonist 는 progesterone 분비를 증가 시켰다. 2. 2-adrenergic agonist 의 농도가 높을수록 doserelate 하게 progesterone의 분비도 증가했다. 이상과 같으 β₂-adrenergic agonist는 태반에서 분비되는 progesterone 양을 증가시켜 자궁평활근에서 흥분한계치(excitation threshold)를 감소시키고 gap junction의 형성도 억제시켜 자극의 감소와 함께 자극이 전파되는 것을 막을 수 있는 효과를 기대할 수 있다. Using a placental monolayer preparation, we have examined the effects of β₂-adrenergic agonist on progesterone production. The results were as follows; 1. In human placenta, β₂-adrenergic agonists may enhance progesterone production. 2. The response to β₂-adrenergic agonist was dose relate; Increased placental progesterone production may decreased the excitation threshold in uterine smooth muscle and may inhibit the propagation of stimuli with the restriction of stimuli by blocking the gap juction formation.