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태양광(Fiber Optical System) 조명 측정 평가
차광석(車光錫) 한국조명·전기설비학회 2002 한국조명·전기설비학회 학술대회논문집 Vol.2002 No.-
본 연구는 태양광시스템의 센서와 프로그램 (타이머스위치)을 혼용한 제어시스템을 측정한 결과이다. 집광부 렌즈 초점의 정확도에 따른 효율 저감을 평가한 결과 실제 그 차이는 조사부 50Cm 떨어진 곳에서 0.1lx에서 5000lx 값 차이를 나타났다. 이는 태양광조명의 가장 중요한 핵심이 바로 태양추적의 정확성에 있는 것이다.
흰쥐 해마절편에서 포도당/산소 고갈에 의한 5-hydroxytryptamine 유리변동에 미치는 Adenosine의 영향
차광은,배영숙,이경은,Cha, Kwang-Eun,Pae, Young-Sook,Lee, Kyung-Eun 대한약리학회 1997 The Korean Journal of Physiology & Pharmacology Vol.1 No.6
The effects of adenosine, adenosine A1 receptor antagonist (DPCPX), or NMDA receptor antagonist (APV) on the spontaneous release of $[^3H]-5-hydroxytryptamine$ ($[^3H]-5-HT$) during normoxic/normoglycemic or hypoxic/hypoglycemic period were studied in the rat hippocampal slices. The hippocampus was obtained from the rat brain and sliced $400\;{\mu}m$ thickness with the tissue slicer. After 30 min's preincubation in the normal buffer, the slices were incubated for 30 min in a buffer containing $[^3H]-5-HT$ ($0.1\;{\mu}M,\;74{\mu}Ci/8\;ml$) for uptake, and washed. To measure the release of $[^3H]-5-HT$ into the buffer, the incubation medium was drained off and refilled every ten minutes through sequence of 14 tubes. Induction of glucose/oxygen deprivation (GOD; medium depleting glucose and gassed with 95% $N_2/5%\;CO_2$) was done in 6th and 7th tube. The radioactivities in each buffer and the tissue were counted using liquid scintillation counter and the results were expressed as a percentage of the total radioactivities. When slices were exposed to GOD for 20 mins, the spontaneous release of $[^3H]-5-HT$ was markedly increased and this increase of $[^3H]-5-HT$ release was blocked by adenosine ($10\;{\mu}M$) or DL-2-amino-5-phosphonovaleric acid (APV; $30\;{\mu}M$). Adenosine $A_1$ receptor specific antagonist, 8-cyclopentyl-1,3-dipropylxanthine (DPCPX) exacerbate GOD-induced increase of spontaneous release of $[^3H]-5-HT$. These results suggest that Adenosine may play a role in the GOD-induced spontaneous release of $[^3H]-5-HT$ through adenosine $A_1$ receptor activity.