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박상배 위덕대학교 부설 전자기술연구소 1997 전자기술연구소 논문집 : 위덕대 Vol.1 No.1
수동 소나시스템에서 표적의 추적성능을 향상시키기 위해 표적을 탐지하고 방위각을 추정하여야 한다. 빔형성된 출력을 보간하고 그 첨두치를 이용하여 표적을 탐지하고 방위각을 추정한다. 본 논문에서는 실제 해상실험 데이터를 분석하여 적절한 표적 탐지 문턱값을 제시하였으며 3점 보간기법을 이용한 효율적인 방위각 추정기법을 제안하였다. 그리고 실제 해상 시험 데이터를 이용한 컴퓨터 시뮬레이션을 통하여 제안한 알고리즘의 타당성을 확인하였다. In passive sonar systems, proper target detection and bearing estimation is required to improve the tracking performance. The target is detected and the bearing is estimated using the interpolated peak value of the preformed beam outputs. In this paper, we determined the detection threshold analyzing a real underwater experimentation data. We also proposed an efficient narrowband bearing estimation algorithm using 3-point interpolation. We verified the performance of the proposed algorithms using real underwater experimentation data.
박상배 동양대학교 1995 동양대학교 논문집 Vol.1 No.1
Fusion tracking method has attractive features for tracking multiple target bearings, in which sideband estimate and narrowband estimate are combined using proper probabilistic model in sensor level or in central level.The combining level may affect the performance of the fusion method significantly. In this paper we show that sensor level fusion tracking method is appropriate for underwater acoustic environment, and propose the improved fusion tracking method which utilizes the well-known PDA filler.We demonstrate the improved tracking performance of the proposed fusion method using real underwater acoustic data through various computer simulations.
UWB 레이더를 사용한 수면무호흡환자에 대한 비접촉방식 수면효율 및 수면 단계 추정
박상배,김정하 한국산업융합학회 2020 한국산업융합학회 논문집 Vol.23 No.3
This study proposes a method to improve the sleep stage and efficiency estimation of sleep apnea patients using a UWB (Ultra-Wideband) radar. Motion and respiration extracted from the radar signal were used. Respiratory signal disturbances by motion artifacts and irregular respiration patterns of sleep apnea patients are compensated for in the preprocessing stage. Preprocessing calculates the standard deviation of the respiration signal for a shift window of 15 seconds to estimate thresholds for compensation and applies it to the breathing signal. The method for estimating the sleep stage is based on the difference in amplitude of two kinds of smoothed respirations signals. In smoothing, the window size is set to 10 seconds and 34 seconds, respectively. The estimated feature was processed by the k-nearest neighbor classifier and the feature filtering model to discriminate between the sleep periods of the rapid eye movement (REM) and non-rapid eye movement (NREM). The feature filtering model reflects the characteristics of the REM sleep that occur continuously and the characteristics that mainly occur in the latter part of this stage. The sleep efficiency is estimated by using the sleep onset time and motion events. Sleep onset time uses estimated features from the gradient changes of the breathing signal. A motion event was applied based on the estimated energy change in the UWB signal. Sleep efficiency and sleep stage accuracy were assessed with polysomnography. The average sleep efficiency and sleep stage accuracy were estimated respectively to be about 96.3% and 88.8% in 18 sleep apnea subjects.
백서 대뇌피질에서 허혈에 의한 [^3H] Norepinephrine 유리에 대한 GABA의 영향
박상배,최용우,김영재 의과학연구소 1997 全北醫大論文集 Vol.21 No.1
Ischemia induces massive release of norepinephrine(NE) from brain tissues. Release of NE is influenced by γ-aminobutyric acid(GABA). However, the role of GABAergic neurons in regulation of NE release induced by ischemia. Thus, I examined the influence of GABAergic drugs on ischemia-induced release of [^3H]NE from cortex slices of the rat. Ischemia, deprivation of oxygen and glucose, induced release of [^3H]NE from striatal slices in vitro. Ischemia-evoked release of [^3H]NE was significantly attenuated by 1 μM tetrodotoxin(TTX), 1.2mM Mg^2+ and 10mM glucose. Muscimol(30μM), a GABAa agonist, or bacolfen(30μM), a GABAb agonist, did not influence either resting or ischemia-evoked release of [^3H]NE. Whereas, bicuculline(30μM), a GABAa antagonist, or 5-aminovaleric acid(5-AV) (30μM), attenuated ischemia-evoked release of [^3H]NE in rat cortex slices. Nipecotic acid, an inhibitor for neuronal GABA transporter, β-alanine, an inhibitor for glial GABA transporter, and L-2, 4-diamino-N-butyric acid(DABA), an inhibitor for neuronal and glial GABA transporter, attenuated, not potentiated, the NE release. These results suggest that ischemia-evoked release of NE is regulated by GABA receptors, GABAa and GABAb, also, that the release of GABA in ischemic brain tissue can be achieved by reversal of GABA transporter.
백서 대뇌피질에서 허혈에 의한 [^3H]Norepinephrine 유리에 대한 GABA 의 영향
박상배,김성수,조병춘 의과학연구소 1996 全北醫大論文集 Vol.20 No.2
Ischemia induces massive release of norepinephrine (NE) from brain tissues. Release of NE is influenced by γ-aminobutyric acid(GABA). However, the role of GABAergic neurons in regulation of NE ischemia-induced ha not been elucidated. Thus, we examined the influence of GABAergic drugs on ischemia-induced release of [^3H]NE from cortex slices of the rat. Ischemia, deprivation of oxygen adn glucose, induced release of [^3H]NE from cortex slices in vitro. Ischemia-evoked release of [^3H]NE was significantly attenuated by 1 μM tetrodotoxin (TTX), 1,2 mM Mg^2+ and 10mM glucose. Muscimol(30 μM), a GABAb agonist, did not influence either resting or ischmia-evoked release of [^3H]NE. Whereas, bicuculline(30 μM), a GABAa antagonist, or 5-aminovaleric acid (5-AV) (30 μM), attenuated ischemia-evoked release of [^3H]NE in rat cortex slices. Nipecotic acid, an inhibitor for neuronal GABA transporter, β-alanine, an inhibitor for glial GABA transporter, and L-2, 4-diamino-N-butyric acid (DABA), an inhibitor for neuronal and glial GABA transporter, attenuated, not potentiated, the NE GABA transporter, attenuated, not potentiated, the NE release. These results suggest that ischemia-evoked release of NE is regulated by GABA in ischemic brain tissue, and can be achieved by reversal of GABA transporter. (Key words : Rat, cerebral ischemia, slices, GABA norepinephrine)