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만성신부전의 신경전도 검사에서 속도 , 진폭 및 잠복기간
김형규(Hyoung Kyu Kim),변관수(Kwan Soo Byun),권희규(Hee Kyu Kwon),노정우(Jung Woo Noh) 대한내과학회 1987 대한내과학회지 Vol.33 No.1
N/A With modern methods of treatment, overt clinical neuropathy of CRF is rare and a subclinical form is commoner in which only nerve conduction studies are abnormal. It should however be realized that for many such patients the conduction velocity would still be within the normal range, Furthermore, motor conduction velocity by itself gives an incomplete picture of the neurophysiologic abnormality. The amplitude and latency of peripheral nerve may reveal abnormalities when motor nerve conduction velocity values are within normal limits. The development of uremic neuropathy is related to decreasing renal function and accumulating uremic toxins. By the way, the authors examined the nerve conduction study such as velocity, amplitude and latency of the peripheral nerves of which 18 chronic renal failure have been treated only the supportive method. Serum BUN, Creatinine, Creatinine clearance, iPTH and vitamine B(12) which were examied with correlation of the resul1 taking from ulnar, radial, median, tibial and peronea nerves were investigated. The results obtained are as follows: 1) Frequency of abnormal nerve conduction involved to be in order the tibial 61.1%, peroneal 50% in motor nerves and tibial 44.4%, peroneal 42.9% and median 37.5% in sensery nerves. 2) The most frequent abnormalities are a decreased amplitude 55.8% in motor nerves and a delayed latency 40.5% in sensory nerves, 3) The mator nerve conduction velocities and latencies of CRF are delayed longer than those of normal controls significantly. 4) The motor nerve conducting tibial nerve has a negative correlation with serum vitamin-B(12) level significantly (r=-0,57, p<0.05). By the metioned results, we may suggest that not only velocity, amplitude and latency of motor nev , 4W also sensory nerves in upper and lower extrimities should be measured simultaneously in chronic renal failure probably complicated uremic neuropathy.
김형규(Hyoung Kyu Kim),채인정(In Jung Chae) 대한내과학회 1988 대한내과학회지 Vol.35 No.2
N/A Disturbance of calcium metabolism in patients with nephrotic syndrome is not uncommon even at normal level of renal function, but the factors reponsible for the hypocalcemia are not well understood. We studied 32 patients (aged 16-62 years) with the nephrotic syndrome and normal renal function, and evaluated their serum calcium, serum phosphorus, serum creatinine, serum albumin, urinary protein, and ealcium, phosphours excretion, PTH, levels. The leagth of disease duration and underlying glomerular disease. The serum creatinine concentiation was 1.8±0.33 mg/ dl, serum albumin 2.21±0.76g/dl<proteinuria 11.5±6.7g/day, serum Ca 8.07±0.51mg/dl (below 8.5mg/dl) urinary Ca excretion 0.045±0.051 (mg/100ml GFR), serum phosphorus 3.76tl.68mg/dl (94% above 4.8 mg/dl) urinary phosphorus excretion 0.31±0.128mg/100ml GFR, PTH and 0.38±0.7ng/ml, A low calcium concentration was found in severe hypoalbuminemia (>2g/dl) and in heavy proteinuria (10 g/day) patients. However, no differences in serum calcium concentration were abserved in varlations of disease duration, age, PTH and glomerular disease and elsewhere serum phosphorus, urinary calcium and phosphorus excretion. Thus, these data suggest that calcium metaboliam in nephrotic syndrome are influnced by hypoalbuminemia and heavy proteinuria, In addition, it will be necessary to do a follow-up follow it ionized calcium, Vit-D metabolites and bone pathology.
Prostaglandin E2 가 신기능장애에 따라 고혈압에 미치는 영향
김형규(Hyoung Kyu Kim),최원충(Won Choong Choi),노정우(Jung Woo Noh) 대한내과학회 1988 대한내과학회지 Vol.34 No.4
N/A The renal prostaglandin E2 (PGE2) is primarily synthesized in renal medulla, and is entirely removed from blood on passage across the lung before it enters systemic circulation, and so is called the local hormone. The main physiologic effects of the PGE, on kidney are known as the natriuresis, water diuresis, vasodilatation and interaction with renin-angiotensin system. The authors attempted to evaluate the role of PGE2 on hypertension according to the state of impairment of renal function. The study of subjects were classified as following groups; namely Group A is a 24 normal control person without renal disease as well as impariement of renal function, Group B that of a 23 cases is essential hypertension without impairement of renal function. Group C is a 22 cases chronic renal insufficiency with hypertension of which creatinine clearance 21~59 ml/min. and Group D is a 24 cases with chronic renal failure with hypertension of which creatinine clearance is below 20 ml/min, Urine prostaglandin E2 diastolic blood pressure, twenty four hour urine Na excretion, creatinine clearance, plasma renin activity (PRA) and plasma aldosterone were investigated in all subjects and the interrelations among these parameters were analysed by multiple analysis method. Urine prostaglandin E2 was measured by radioim-munoassay with gamma counter. The NEM prostaglandin E2 I125 -radio-immunoassay kit is based on the use of an iodinated analog of prostaglandin E2 as the tracer. The results obtained are as follows: 1) Urine prostaglandin E2 (Mean±S.E): The value of group A, normal control groups is 365.1±20.65pg/ml., group B, essential hypertension; 353.9±20.71pg/ml., group C, chronic renal insufficiency; 149.1±10.70pg/ml. and group D chronic renal failure 83.3±8.30pg/ml. The value of group A is similar with group B (P>0.05), that of group C and D are significantly decreased in the comparsion with group A and B (P<0.01), while in the group C show the significantly high value in the comparsion with group D (P<0.01), group D is the lowest value among the comparison of group A, B and C (P<0.01). 2) Urine prostaglandin E2 has not significant relation to diastolic blood pressure, 24 hour urine Na excretion, creatinine clearance and plasma renin activity in all groups. 3) Urine prostaglandin E2 has significant correlation to plasma aldosterone in essential hypertension (r=0.489) (P<0,05), but has not significant relation to other groups. This findins suggest that the failure of compensatory protective role by renal FGE2 which is due to decreased PGE2 synthesis according to renal function impairment with diminished renal parenchyme may depend on the machanism of hypertension also is possible to mediate Na metabolism.
First ClustalX-MPI for Ultra-fast Protein and Gene Discovery
Teaho Kim(김태호),Jin Han(한진),Jae bum Youm(염재범),Nari Kim(김나리),Won sun Park(박원선),Sunghyun Kang(강성현),Dang Van Cuong,Hyoung kyu Kim(김형규),Tran Min Khoa,Vu Thi Thu,Hyunju Kim(김현주),Hyejin Moon(문혜진),Hyunsuk Lee 한국생물공학회 2005 한국생물공학회 학술대회 Vol.2005 No.10
Role of Protein Kinase A in Long-Term Agonist-Promoted Desensitization of the β-Adrenergic Receptor
Hyunju Kim(김현주),Nari Kim(김나리),Hyun Joo(주현),Jae boum Youm(염재범),Won sun Park(박원선),Sunghyun Kang(강성현),Dang Van Cuong,Teaho Kim(김태호),Hyoung kyu Kim(김형규),Hyejin Moon(문혜진),Hyunsuk Lee(이현숙),Tran MinhKhoa,Vu 한국생물공학회 2005 한국생물공학회 학술대회 Vol.2005 No.10