http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Ecabet sodium이 Helicobacter pylori에 감염된 인체 위상피세포에서의 핵전사인자 활성과 chemokine 유전자 발현에 미치는 영향
김정목 ( Kim Jeong Mog ),김주성 ( Kim Ju Seong ),정현채 ( Jeong Hyeon Chae ),강신재 ( Kang Sin Jae ),김나영 ( Kim Na Yeong ),이동호 ( Lee Dong Ho ),송인성 ( Song In Seong ) 대한내과학회 2003 대한내과학회지 Vol.65 No.2
Background : Helicobacter pylori stimulates nuclear factor-kappa B (NF-κB) activation and chemokine expression of gastric epithelial cells. Although ecabet sodium (ecabet), a locally acting anti-ulcer drug, is known to have an anti-H. pylori activity, the
H. pylori에 감염된 마우스에서 p65 antisense oligonucleotide에 의한 친염증성 사이토카인 발현의 억제
김상균 ( Kim Sang Gyun ),김정목 ( Kim Jeong Mog ),정현채 ( Jeong Hyeon Chae ),김주성 ( Kim Ju Seong ),송인성 ( Song In Seong ) 대한소화기학회 2003 대한소화기학회 추계학술대회 Vol.2003 No.-
<목적> Helicobacter pylori는 만성 위염, 소화성 궤양, 위선암 및 위림프종과 관련이 있음이 알려져 있으나, 감염 초기에 발생하는 위상피세포와의 상호 작용에 대해서는 NF-κB와 친염증성 사이토카인의 발현이 중요한 역할을 담당할 것이라는 사실이 세포주를 통한 연구에서만이 일부 밝혀져 있을 뿐이다. 이에 저자들은 생체내에서 H. pylori 감염이 NF-κB의 활성화와 친염증성 사이토카인의 발현을 유발하며, NF-κB 경로를 억제하였을 때
일차 제균요법 실패 후 이차 삼제 병합요법에 의한 헬리코박터 파이로리 감염 재치료
김나영 ( Kim Na Yeong ),임선희 ( Im Seon Hui ),이계희 ( Lee Gye Hui ),구명숙 ( Gu Myeong Sug ),김정목 ( Kim Jeong Mog ),황진혁 ( Hwang Jin Hyeog ),김진욱 ( Kim Jin Ug ),이동호 ( Lee Dong Ho ),정현채 ( Jeong Hyeon Chae ),송인성 ( S 대한소화기학회 2003 대한소화기학회지 Vol.42 No.3
Background/Aims: Helicobacter pylori (H. pylori) treatments fail at least in 10-20% of patients. However, retreatment strategies after failure of initial treatment have not been established. This study was conducted to evaluate the eradication rate of retreatment choices. Methods: Twenty-seven peptic ulcer patients who were retreated with OAC (omeprazole+amoxicillin+clarithromycin) or BMT (bismuth+metronidazole+tetracycline) after failure of BMT or OAC were included. Quadruple therapy (omeprazole+BMT) was also tried after failure of two successive triple therapies. Furthermore, the effect of resistance of metronidazole or clarithromycin on the eradication of H. pylori was evaluated. Results: Among 13 patients who were retreated with OAC after failure of BMT regimen, H. pylori was eradicated in 10 patients (76.9%). Among 14 patients retreated with BMT after failure of OAC regimen, H. pylori was eradicated in 11 patients (78.6%). Resistance of H. pylori to metronidazole or clarithromycin decreased the efficacy of BMT or OAC, respectively. Conclusions: Eradication regimen should be decided considering the resistance to H. pylori. However, in case of unknown state of resistance, OAC can be chosen if BMT fails. Similarly, BMT can be tried in cases that OAC therapy failed. After failures of both triple therapies, quadruple therapy can be tried as the next step. (Korean J Gastroenterol 2003;42:195-203)