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Effects of Chamaecyparis Obtusa Essential Oil on the Autonomic Nervous System
박사훈,정한성,장수정,김성진,박종성 조선대학교 기초과학연구원 2019 조선자연과학논문집 Vol.12 No.3
The purpose of this study is to explore the effects of Chamaecyparis obtusa essential oil on the activity of autonomic nervous system. Heart rate variability (HRV), as an indicator of autonomic nervous system activity and blood pressure were measured before and after inhalation of Chamaecyparis obtusa essential oil. The systolic blood pressure was decreased by inhalation of Chamaecyparis obtusa essential oil (p<0.05). The diastolic blood pressure was not changed significantly by Chamaecyparis obtusa. High frequency (HF) power level was not changed but High frequency/Low frequency (HF/LF) ratio was decreased by Chamaecyparis obtusa essential oil (p<0.05), meaning that parasympathetic nervous system activity was not affected but sympathetic nervous system activity was decreased. These results indicate that Chamaecyparis obtusa essential oil has a modulatory effect on the autonomic nervous system activity.
속도 축척 모형 실험 방법을 통한 집전계 성능 분석 연구의 실험 방법 및 효용성에 대한 연구
박사훈(Park Sa-Hoon),권삼영(Kwon Sam-Young) 한국철도학회 2008 한국철도학회 학술발표대회논문집 Vol.- No.-
To test the current collection characteristics between overhead contact lines and pantograph in a real speed scale costs too much, therefore, it is very difficult to be realized especially in a high speed region. As a alternative, the speed scale-downed tests is proposed. In this paper, the experimental methodology and effectiveness of the speed scale-downed tests are discussed through the dynamic simulation evaluations. To get a more precise test results to the real speed scale test in the 1/2 and 1/4 speed scale-downed tests, various experimental conditions are discussed. Throughout the simulation in a various conditions the effectiveness are evaluated.
장수정,박사훈 전남대학교 의과학연구소 2018 전남의대학술지 Vol.54 No.1
The antidiabetic drug metformin has been found to have beneficial effects in variousneurological disorders; however, the molecular mechanisms underlying these effectsremain unclear. Here we report that metformin protects neuronal cells from quinolinicacid (QUIN)-induced excitotoxicity. For this, we pretreated N18D3 neuronal cells withmetformin prior to QUIN for 24 h. We found that pretreating the cells with metforminsignificantly improved cell survival rate in a concentration-dependent manner and reducedapoptotic cell death, as revealed by a MTT assay and DAPI staining, respectively. Calcium imaging using fluo-4 showed that metformin (100 μM) inhibited the intracellularcalcium increase that was induced by QUIN. In addition, mRNA expressionof pro-apoptotic genes, p21 and Bax, was decreased and of anti-apoptotic genes, Bcl-2and Bcl-xl, was increased with metformin treatment compared to QUIN-induced cells. The immunoreactivity of phosphorylated ERK1/2 was elevated in cells treated withmetformin, indicating the ERK1/2 signaling pathway in the neuroprotective effects ofmetformin in QUIN-induced cell death. Collectively, our data demonstrates that metforminexerts its neuroprotective effects by inhibiting intracellular calcium increases,allowing it to regulate ERK1/2 signaling and modulate cell survival and death genes.