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강태석(Kang Tae Seok) 한국지적학회 2005 한국지적학회지 Vol.21 No.2
국가 토지행정의 기반을 이루는 지적제도는 정보화 시대에 알맞은 데이터의 정확성과 신속한 이동지 조사를 통한 현지와의 일치를 근간으로 하고 있다. 그러나 그 동안 많은 노력에도 불구하고 지적의 정보화를 추진하는 과정에서 지적공부와 현지 상황이 정확히 일치해야 하는 데이터의 정확성 측면에서 다양한 문제점이 발견됨으로써 이를 신속히 개선할 필요성이 대두되어 왔다. 정확한 데이터베이스의 구축을 위해서는 지적재조사를 통한 새로운 접근방식이 요구되고 있다. 조사기간이 장기적인 점 등을 감안하고 정보화 업무 추진의 시급성으로 인하여 우선 현행 지적 등록사항을 정비하면서 건축물을 등록 하는 수치지적측량을 실시하는 방향으로 지적재조사 사업이 추진되어야 할 것이다. The cadastral system which accomplishes the base of national land administration with accuracy of proper data is agreeing with the actual place where it leads the maintenance which is quick in information age in foundation. But even in spite of many efforts, the problems were discovered variously from the accuracy side of the cadastral data and local situation must agree accurately in the process of cadastral information and It will be improved quickly for this projet. Construction of the accurate data base it leads the cadastral renovation and the new approach method is demanded. It will be processed under the consideration of the government and social charge to be big, also needs long surveying duration. Thus the cadastral renovation must be propelled with the direction which executes the digital cadastral surveying methods accompany with the building registration and the current cadastral registration items be improved with urgent characteristic of information business propulsion.
지적측량 검사의 실효성 향상을 위한 검사제도 개선 연구
강태석(Kang Tae Seok),홍성은(Hong Sung Eon) 한국지적학회 2006 한국지적학회지 Vol.22 No.1
현행 지적측량 검사제도는 법률, 제도 운영 면에서 여러 문제점의 발생으로 실질적인 현지검사가 되지 못하고 있어 국가 공신력 약화와 많은 민원이 우려되고 있다. 즉, 지적측량성과 검사를 실지검사 위주가 아닌 도상검사 위주로 하고 있고, 1년에 한 번씩 발췌검사를 하는 정도라서 측량의 오류에 대한 정확한 발견 및 정정에는 무리가 있어 불부합지 양산의 원인과 경계분쟁의 실마리를 제공하고 있다.<BR> 본 연구에서는 현행 수행되는 지적측량에 대해 실제적이고 효율적으로 검사할 수 있도록 장·단기적인 검사제도의 개선방안을 제시하고자 하였다. 이를 위해 현행 지적측량 검사제도의 문제점 해결을 위해 도입 가능한 검사제도 대안들을 탐색하였고, 새로운 제도 도입을 위한 도입의 기본방향, 운영방법, 장·단점을 분석하였으며, 검사제도 개선에 대한 지적실무자를 대상으로 설문조사 분석을 통하여 제안되는 검사제도에 대하여 안정성과 타당성을 확보하고자 하였다. The current cadastral surveying inspection system has been occurred many problems in law and operation from the substantial inspection does not become in the field and with public trust weakening the many civil appeal is worried. The cadastral surveying inspection result carried out currently with way on the maps and the actual field inspection does 2∼3 sampling to discover and error correction are difficult. So it provides the clue of cause of many cadastral non correspondence and disputes of land boundaries.<BR> In this study, the existent cadastral surveying which is accomplished actual and it will be able to inspect efficiently, It presented the improvement program of short and long-term period. For the solutions of hazard are searched in current cadastral surveying inspection system, the new alternative systems are introduced and also it analyzed the general directions and operation method of the introduction for a new system improvement. Finally I tried to examine the stability and propriety of the proposed system against the cadastral surveying experts by the questionnaire.
에스트로겐 수용체를 통한 카드뮴 독성 및 항산화제에 의한 독성경감에 관한 연구
김태성(Tae Sung Kim),강태석(Tae Seok Kang),강호일(Hoil Kang),문현주(Hyun Ju Moon),강일현(Il Hyun Kang),이영주(Young Joo Lee),최은희(Eun Hee Choi),홍진태(Jin Tae Hong),한순영(Soon Young Han),홍진환(Jin Hwan Hong) 한국환경성돌연변이발암원학회 2006 한국환경성돌연변이·발암원학회지 Vol.26 No.1
Cadmium, a human carcinogen, can induce toxicity in various cell lines and organs. Despite extensive research, the mechanisms of cadmium-induced cell toxicity and estrogenic potential in human are not clear. This study was performed to investigate cadmium-induced toxicity on human breast cancer cells :MCF-7 cells, an estrogen receptor (ER) positive breast cancer cells, and MDA-MB-231 cells, an ER negative breast cancer cells. MCF-7 cells was proved to be more sensitive than the other cell lines (IC50 = 50 μM at MCF-7 cells and 120 μM at MDA-MB-231). The expression of JNK and AP-1 transcription factors such as c-Jun and c-Fos dependent transcription were increased by cadmium treatment. Inhibition of ER activation by ER antagonist (tamoxifen or ICI 182,780) significantly recovered the viablity and inhibited apoptotic cell death. This suggested that cadmium-induced cell death in ER (+) cells was mediated by JNK/AP-1 pathway and this pathway was more stimulated by ER activated by cadmium. Co-treatment of antioxidants such as selenium (Se), butylated hydroxyanisole (BHA), glutathione (GSH), or N-acetyl-L-cysteine (NAC) recovered the cadmium-induced cell death in MCF-7 cells. Cadmium-induced lipid peroxidation was decreased by GSH, NAC, or BHA in MCF-7 cells. The expression of SOD protein was decreased by cadmium (100 μM) but recovered by GSH, NAC, BHA, or Se. Our data showed that the cadmium-induced cell toxicity in human breast cancer cells could be protected by the antioxidants (Se, BHA, NAC, GSH, or NAC) and ER antagonist (tamoxifen or ICI 182,780). Therefore, toxicity of cadmium in breast cancer were mediated by oxidative stress and ERα.
강완 ( Wan Kang ),강태석 ( Tae Seok Kang ) 서울교육대학교 초등교육연구원 2003 한국초등교육 Vol.14 No.1
분수의 크기 비교 지도 방법의 교수학적 변환 분석 An Analysis of the Order Relation of Fractions in Elementary School Mathematics Textbooks
스테로이드 합성을 교란하는 내분비계장애물질 검색을 위한 라이디히 세포 분리 및 배양조건 확립
강일현(Il Hyun Kang),강태석(Tae Seok Kang),강호일(Ho Il Kang),문현주(Hyun Ju Moon),김태성(Tae Sung Kim),기호연(Ho Hyun Ki),류혜원(Hye Won Ryu),신재호,동미숙(Mi Sook Dong),한순영(Soon Young Han),김승희(Seung Hee Kim),홍진환(Jin Hwan Hong 한국환경성돌연변이발암원학회 2006 한국환경성돌연변이·발암원학회지 Vol.26 No.2
Normally, environmental toxicants are classified as endocrine disruptors if they interfere with regulation of cellular function by endogeneous steroids through inhibition of receptor binding and/or transcriptional activation. So, many studies have been performed about agonist/antagonist of hormone receptor to study mechanisms of endocrine disruptors. If toxicants affect steroid biosynthesis and/or degradation and alter hormone homeostasis, these also are classified as endocrine disruptors. But there are not many studies of the mechanisms of endocrine disruptors on the basis of alteration of steroid biosynthesis and/or degradation. Isolation and culture of Leydig cells from testis is one of methods for the steroidogenesis screening assays to evaluate a substance for altering steroidogenesis. Leydig cells were harvested using the method described by Klinefelter with modifications. Leydig cells were purified by perfusion of testis and incubation (34℃, 80cycles/minute, 20 minutes) with collagenase (0.25 ㎎/㎏), centrifugal elutriation, percoll gradient centrifugation and BSA multidensity gradient centrifugation. To confirm if this method is one of appropriate tools to evaluate a substance for altering steroidogenesis, ketoconazole, positive control was administered to purified Leydig cells. Ketoconazole (10??M and above) significantly reduced testosterone production in purified Leydig cells. From above results, we suggest that this method for steroidogenesis screening assay appears to be a appropriate tool to detect suspected compounds for altering steroidogenesis.
항안드로겐성 물질이 성 성숙 이전 단계의 정소에서 미치는 영향 연구
홍진,한순영,문현주,강태석,강일현,김태성,김승희,권기성,Hong Jin,Han Soon-Young,Moon Hyun-Ju,Kang Tae-Seok,Kang Il-Hyun,Kim Tae-Sung,Kim Seung-Hee,Kwon Ki-Sung 환경독성보건학회 2006 환경독성보건학회지 Vol.21 No.3
The experiments investigated whether early exposure to testosterone propionate (TP) during prepuberty alters testis development in Sprague-Dawley male rats. We performed Hershberger assay using the stimulated weanling male rats by OECD protocols, cDNA microarray, and Western blot. TP was subcutaneously injected to uncastrated Sprague-Dawley male rat of 22 days old for 10 consecutive days at doses of 0.4, 0.8, 1.0, 1.2, 1.6 mg/kg per day. At necropsy, the following tissues were removed and weighed: combined testes, epididymides (Epi), Cowper's glands (COW), levator am, and bulbocavernosus muscles (LABC), seminal vesicles, together with coagulating gland (SV) and ventral prostate (VP). We found that TP increased the weights of Epi, VP, SV, COW, and LABC, while testis was decreased in a dose-dependent manner. In cDNA microarray analysis of testis, there were significant reductions in the expression of cytochrome P450 11A (CYP11A), the rate-limiting enzyme of steroidogenesis. Taken together these results, TP exposure before puberty in male rats may produce the delay in testis development by inhibiting the CYP11A gene expression.