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축구 동호회 참여가 사회적지지, 스트레스, 생활만족에 미치는 영향
김장환,박순택,박진기 한국스포츠리서치 2004 한국 스포츠 리서치 Vol.15 No.4
This study aims to check the effects of the participation in soccer 치ubs on social support, stress, and life satisfaction and examine, first, the relation between the participation in soccer clubs and social support, second, the relation between the participation in soccer clubs and stress, third, the relation between the social support of soccer club members and stress, and, finally, the causal relation between the participation in soccer clubs and social support & stress. The findings according to these study methods and data analysis methcds are as follows: First, the period and frequency of participation influence the social support. That the soccer club members, as they meet their fellow members over a long time, feel emotional comfort and the social support becomes high. Second, the intensity and period of participation influence the material support. That is, the longer the meetings for club members' activities, the higher the material support is. Third, the period and frequency of participation influence the emotional support. That is, as the fellow members meet together frequently over a long time, the emotional support becomes high. Fourth, the frequency and intensity of participation influence the stress. That is, owing to the regular sports activities, the higher the frequency and intensity of the soccer club activities are, the lower the stress becomes. Fifth, the material support, the social support, and spiritual support influence the stress significantly in that order. Sixth, the social support and stress influence the life satisfaction That is, the soccer club activities remove the spiritual and physical stress, and, through the exchanges between club members, the social support is formed and the life satisfaction becomes high. Putting all these together, it is concluded that the participation in the soccer club activities removes the usual stress of the people of today, and produces the life satisfaction as a result of the social support through the interaction between the soccer club members.
Alpha-Fetoprotein Producing Sertoli-Leydig Cell Tumor of the Ovary - A Case Report -
Kee-Taek Jang,Hye-Rim Park,Duck-Hwan Kim,Chang-Mo Kim,Woo-Seok Sohn,Hyung-Sik Shin 대한병리학회 2002 Journal of Pathology and Translational Medicine Vol.36 No.2
A 26-year-old woman had an ovarian Sertoli-Leydig cell tumor (SLCT) associated with an elevated level of serum alpha-fetoprotein (AFP). The tumor had a heterologous element of intestinal- type mucinous epithelium, retiform and intermediately differentiated tubules of the Sertoli cells, and AFP-producing Leydig cells. AFP was demonstrated within the Leydig cells by an immunohistochemical technique. After surgery, the serum AFP level of the patient fell to the normal range. The present case is the first documented case of AFP producing a SLCT of the ovary reported in Korea.
장기택 ( Kee Taek Jang ) 대한췌장담도학회 2014 대한췌담도학회지 Vol.19 No.3
Ampulla of Vater (AoV) is a small dilated duct less than 1.5 cm long, formed by the union of pancreatic duct and common bile duct. AoV has also anatomic layer of mucosa, sphincter of Oddi, perisphincteric or duodenal submucosa, and duodenal proper muscle, which corresponds to mucosa, muscularis mucosa, submucosa, and proper muscle layer of other gastrointestinal tract organs, respectively. Because of its small compact size and variation of anatomic structure, it is sometimes difficult to identify layering architecture of AoV. This anatomic difficulty may cause some problem in T classification of ampullary carcinoma (AC). The most confusing point in T classification is the vague definition of T2, “Tumor invades duodenal wall”. It seems that duodenal wall includes duodenal mucosa, submucosa, and proper muscle layer. However there is no precise description or definition about duodenal wall that might lead personal variation in T classification of AC staging. We found that clinical course of AC with perisphincteric and/or duodenal submucosal invasion is more close to AC with T2 than T1. Although it is described as T1b according to T classification scheme of ordinary gastrointestinal tract cancer, we thought AC with T1b may have more high-grade malignant potential than those of other gastrointestinal (GI) tract malignancy. AC showed various clinicopatholgic findings that represent heterogeneous tumor groups within category of AC. Recently sitespecific classification of AC was introduced, and it showed relatively well-categorized clinical prognosis. It may be reasonable to understand site-specific tumorigenesis in AC. The standard gross protocol is needed to evaluate pathologic T classification of AC. In conclusion, ampullary neoplasm is composed of various subtypes, which require a separate approach according to anatomic epicenter of ampullary neoplasm. Although submucosal invasion in AC was classified into pT1b, its` biologic behavior is more close to pT2.
Thioacetamide 에 의한 간경변에서 Pentoxifylline 투여가 흰쥐의 간 섬유화와 세포주기조절 단백질에 미치는 영향
장자준,장기택,김미란,정인평,이미숙 대한간학회 2001 Clinical and Molecular Hepatology(대한간학회지) Vol.7 No.3
Background: Thioacetamide is a classic hepatotoxic reagent which leads to the reproducible hepatic fibrosis in rats. Thioacetamide-induced fibrosis is an appropriate model for cirrhosis in humans due to the long duration of course and similiar histology. Thioacetamide produces hepatotoxicity through lipid peroxidation but it is unclear whether lipid peroxidation directly correlated with hepatic fibrosis. Pentoxifylline, a derivative of the methylxanthine, showed an antifibrogenic effect in cell cultures of human fibroblasts and some animal models. But this antifibrogenic effect is controversial. Pentoxifylline revealed a hepatoprotective effect in some toxic hepatitis. This hepatoprotective effect seems to influence cell cycle regulatory protein during regeneration. This study aimed to evaluate an effect of pentoxifylline on fibrosis and cell cycle regulatory protein during liver regeneration in thioacetamide-induced rat cirrhosis. Lipid peroxidation assay was compared with collagen content so as to evaluate the correlation with fibrosis. Mothed: Liver cirrhosis was induced by 0.03% oral administration of thioacetamide. Pentoxifylline was administered simultaneously with thioacetamide, The semiquantitative fibrosis index was measured based on histologic finding. Collagen content was estimated by spectrophotometric assay. Activated hepatic stellate cells were counted using α-SMA immunohistochemistry. Malondialdehyde, lipid peroxidation metabolite, was estimated by thiobarbituric acid reactive substance assay. Cell cycle regulatory protein was evaluated by western blot. Results- There was no difference in semiquantitative fibrosis index, collagen content and hepatic stellate cell count between thioacetamide treated rats and simultaneous pentoxifylline treated rats. Lipid peroxidation product was not correlated with collagen content. Western blot showed no difference in cell cycle regulatory protein. Conclusion- Pentoxifylline does not show an antifibrogenic effect in thioacetamide-induced rat cirrhosis, in which thioacetamide induced hepatocellular damage and fibrosis. Lipid peroxidation may be a secondary effect rather than primary mediating mechanism in hepatic fibrosis. (Korean J Hepatol 20017:281-291)