<P><B>Summary</B></P><P>Autoimmune diseases are characterized by the body's ability to mount immune attacks on self. This results from recognition of self‐proteins and leads to organ damage due to increased producti...
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https://www.riss.kr/link?id=A107571748
2011
-
SCI,SCIE,SCOPUS
학술저널
145-157(13쪽)
0
상세조회0
다운로드다국어 초록 (Multilingual Abstract)
<P><B>Summary</B></P><P>Autoimmune diseases are characterized by the body's ability to mount immune attacks on self. This results from recognition of self‐proteins and leads to organ damage due to increased producti...
<P><B>Summary</B></P><P>Autoimmune diseases are characterized by the body's ability to mount immune attacks on self. This results from recognition of self‐proteins and leads to organ damage due to increased production of pathogenic inflammatory molecules and autoantibodies. Over the years, several new potential therapeutic targets have been identified in autoimmune diseases, notable among which are members of the tumour necrosis factor (TNF) superfamily. Here, we review the evidence that certain key members of this superfamily can augment/suppress autoimmune diseases.</P>
Toll‐like receptor 4 signalling attenuates experimental allergic conjunctivitis