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RISS
Oxygen derived free redicals are thought to cause myocardial damage during reperfusion of ischemic myocardium. We have used the isolated working heart model and 60 minutes of hypothermic cardioplegic arrest to evaluate whether Allopurinol (xanthine ox...
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RISS 인기검색어
https://www.riss.kr/link?id=A40018055
- 저자
- 발행기관
- 학술지명
- 권호사항
-
발행연도
1989
-
작성언어
Korean
-
KDC
510.000
-
자료형태
학술저널
-
수록면
597-610(14쪽)
- 제공처
- 소장기관
-
0
상세조회 -
0
다운로드
부가정보
다국어 초록 (Multilingual Abstract)
Oxygen derived free redicals are thought to cause myocardial damage during reperfusion of ischemic myocardium. We have used the isolated working heart model and 60 minutes of hypothermic cardioplegic arrest to evaluate whether Allopurinol (xanthine oxidase inhibitor) or tocopherol (antioxidant) can improve postischemic myocardial recovery. Thirty rabbit hearts were divided into three groups (control, allopurinol and tocopherol group). Allopurinol(100㎎/㎏) or tocopherol (200IU/㎏) was administered orally 24 hour before experiment.
Rabbits heart were subjected to one hour of global ischemia dunng which myocardial protection was provided by hypothermia (10℃) along with multidose St. Thomas cardioplegic solution cardioplegia. Both drugs significantly improved postischemic recovery of cardiac function, oxygen consumption and oxygen extraction. Leakage of creatine kinase was low in tocopherol and allopurinol groups. We conclude that pretreatment of allopurinol and tocopherol prevents myocardial injury in reperfused hypothermic ischemic heart.
Rabbits heart were subjected to one hour of global ischemia dunng which myocardial protection was provided by hypothermia (10℃) along with multidose St. Thomas cardioplegic solution cardioplegia. Both drugs significantly improved postischemic recovery of cardiac function, oxygen consumption and oxygen extraction. Leakage of creatine kinase was low in tocopherol and allopurinol groups. We conclude that pretreatment of allopurinol and tocopherol prevents myocardial injury in reperfused hypothermic ischemic heart.
Oxygen derived free redicals are thought to cause myocardial damage during reperfusion of ischemic myocardium. We have used the isolated working heart model and 60 minutes of hypothermic cardioplegic arrest to evaluate whether Allopurinol (xanthine oxidase inhibitor) or tocopherol (antioxidant) can improve postischemic myocardial recovery. Thirty rabbit hearts were divided into three groups (control, allopurinol and tocopherol group). Allopurinol(100㎎/㎏) or tocopherol (200IU/㎏) was administered orally 24 hour before experiment.
Rabbits heart were subjected to one hour of global ischemia dunng which myocardial protection was provided by hypothermia (10℃) along with multidose St. Thomas cardioplegic solution cardioplegia. Both drugs significantly improved postischemic recovery of cardiac function, oxygen consumption and oxygen extraction. Leakage of creatine kinase was low in tocopherol and allopurinol groups. We conclude that pretreatment of allopurinol and tocopherol prevents myocardial injury in reperfused hypothermic ischemic heart.
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