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      KCI등재 SCIE SCOPUS

      How to die chemically? whole body apoptosis

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      https://www.riss.kr/link?id=A104668758

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      다국어 초록 (Multilingual Abstract)

      What would you do if your body decided to diewhen you were not ready? It appears that some biologicalprogram can shut down all bodily functions, in much thesame way as apoptosis does so at the cellular level. Pacificsalmon and annual cicada die after reproduction. How dothey die chemically? Their programmed death after reproductionshould have chemical signals. Similarly, proinflammatorycytokines, particularly tumor necrosis factoraand interleukin-1b, could induce death in mammals.
      Acute massive production of them in sepsis and chronic tinyproduction in aging could lead to death. Thus, the mechanismof the whole body suicide program could be determinedand some people could be rescued from this type ofdeath by elucidating the death program. I propose that theconcept of whole body apoptosis, defined as programmedwhole body death, be adopted by scientific community.
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      What would you do if your body decided to diewhen you were not ready? It appears that some biologicalprogram can shut down all bodily functions, in much thesame way as apoptosis does so at the cellular level. Pacificsalmon and annual cicada die after ...

      What would you do if your body decided to diewhen you were not ready? It appears that some biologicalprogram can shut down all bodily functions, in much thesame way as apoptosis does so at the cellular level. Pacificsalmon and annual cicada die after reproduction. How dothey die chemically? Their programmed death after reproductionshould have chemical signals. Similarly, proinflammatorycytokines, particularly tumor necrosis factoraand interleukin-1b, could induce death in mammals.
      Acute massive production of them in sepsis and chronic tinyproduction in aging could lead to death. Thus, the mechanismof the whole body suicide program could be determinedand some people could be rescued from this type ofdeath by elucidating the death program. I propose that theconcept of whole body apoptosis, defined as programmedwhole body death, be adopted by scientific community.

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      참고문헌 (Reference)

      1 Chan, J. L., "The role of falling leptin levels in the neuroendocrine and metabolic adaptation to short-term starvation in healthy men" 111 (111): 1409-1421, 2003

      2 Jones, M. W., "The influence of male parr body size and mate competition on fertilization success and effective population size in Atlantic salmon" 86 (86): 675-684, 2001

      3 Bernard, A. M., "The immune response : targets for the treatment of severe sepsis" 2012 : 697592-, 2012

      4 Hardie, D. G., "The AMP-activated protein kinase pathway–new players upstream and downstream" 117 (117): 5479-5487, 2004

      5 Namas, R., "Sepsis: something old, something new, and a systems view" 27 (27): 314.e1-314.e11, 2012

      6 Semeraro, N., "Sepsis, thrombosis and organ dysfunction" 129 (129): 290-295, 2012

      7 Fry, D. E, "Sepsis, systemic inflammatory response, and multiple organ dysfunction : the mystery continues" 78 (78): 1-8, 2012

      8 Mommsen, T. P, "Salmon spawning migration and muscle protein metabolism : the August Krogh principle at work" 139 (139): 383-400, 2004

      9 Fraser, D. A., "Reduction in serum leptin and IGF-1 but preserved T-lymphocyte numbers and activation after a ketogenic diet in rheumatoid arthritis patients" 18 (18): 209-214, 2000

      10 Chung, H. Y., "Molecular inflammation as an underlying mechanism of the aging process and age-related diseases" 90 (90): 830-840, 2011

      1 Chan, J. L., "The role of falling leptin levels in the neuroendocrine and metabolic adaptation to short-term starvation in healthy men" 111 (111): 1409-1421, 2003

      2 Jones, M. W., "The influence of male parr body size and mate competition on fertilization success and effective population size in Atlantic salmon" 86 (86): 675-684, 2001

      3 Bernard, A. M., "The immune response : targets for the treatment of severe sepsis" 2012 : 697592-, 2012

      4 Hardie, D. G., "The AMP-activated protein kinase pathway–new players upstream and downstream" 117 (117): 5479-5487, 2004

      5 Namas, R., "Sepsis: something old, something new, and a systems view" 27 (27): 314.e1-314.e11, 2012

      6 Semeraro, N., "Sepsis, thrombosis and organ dysfunction" 129 (129): 290-295, 2012

      7 Fry, D. E, "Sepsis, systemic inflammatory response, and multiple organ dysfunction : the mystery continues" 78 (78): 1-8, 2012

      8 Mommsen, T. P, "Salmon spawning migration and muscle protein metabolism : the August Krogh principle at work" 139 (139): 383-400, 2004

      9 Fraser, D. A., "Reduction in serum leptin and IGF-1 but preserved T-lymphocyte numbers and activation after a ketogenic diet in rheumatoid arthritis patients" 18 (18): 209-214, 2000

      10 Chung, H. Y., "Molecular inflammation as an underlying mechanism of the aging process and age-related diseases" 90 (90): 830-840, 2011

      11 Taleux, N., "Lack of starvationinduced activation of AMP-activated protein kinase in the hypothalamus of the Lou/C rats resistant to obesity" 32 (32): 639-647, 2008

      12 Baker, D. M., "Insulin-like growth factor I increases follicle-stimulating hormone(FSH)content and gonadotropin-releasing hormone-stimulated FSH release from coho salmon pituitary cells in vitro" 63 (63): 865-871, 2000

      13 Yamashita, M., "Increase in catheptic activity and appearance of phagocytes in the white muscle of chum salmon during spawning migration" 50 (50): 565-567, 1991

      14 Lindberg, N., "Growth hormone-insulin-like growth factor-1 axis, leptin and sleep in anorexia nervosa patients" 47 (47): 78-85, 2003

      15 Lu, J. Y., "Endotoxic shock in AUF1 knockout mice mediated by failure to degrade proinflammatory cytokine mRNAs" 20 (20): 3174-3184, 2006

      16 Kale, S. S., "Effects of aging on inflammation and hemostasis through the continuum of critical illness" 2 (2): 501-511, 2011

      17 Cui, B., "Determinants of annualperennial plant zonation across a salt-fresh marsh interface : a multistage assessment" 166 (166): 1067-1075, 2011

      18 Ulukaya, E., "Apoptosis: why and how does it occur in biology?" 29 (29): 468-480, 2011

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      학술지 이력

      학술지 이력
      연월일 이력구분 이력상세 등재구분
      2023 평가예정 해외DB학술지평가 신청대상 (해외등재 학술지 평가)
      2020-01-01 평가 등재학술지 유지 (해외등재 학술지 평가) KCI등재
      2010-01-01 평가 등재학술지 유지 (등재유지) KCI등재
      2008-01-01 평가 등재학술지 유지 (등재유지) KCI등재
      2006-01-01 평가 등재학술지 유지 (등재유지) KCI등재
      2004-01-01 평가 등재학술지 유지 (등재유지) KCI등재
      2001-01-01 평가 등재학술지 선정 (등재후보2차) KCI등재
      1998-07-01 평가 등재후보학술지 선정 (신규평가) KCI등재후보
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      학술지 인용정보

      학술지 인용정보
      기준연도 WOS-KCI 통합IF(2년) KCIF(2년) KCIF(3년)
      2016 1.96 0.2 1.44
      KCIF(4년) KCIF(5년) 중심성지수(3년) 즉시성지수
      1.07 0.87 0.439 0.05
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