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Cysteamine 이 백서의 십이지장궤양 형성시 미치는 영향
함기백,이상인,임대순,김원호,전재윤,문영명,강진경,박인서,최홍재,박형우 ( Ki Baik Hahm,Sang In Lee,Dae Soon Yim,Won Ho Kim,Chae Yoon Chon,Young Myung Moon,Jin Kyung Kang,In Suh Park,Heung Jai Choi,Hyung Woo Park ) 대한소화기학회 1992 대한소화기학회지 Vol.24 No.4
The administration of cysteamine is koown to produce duodenal ulcer in rat. Mucosal damage starts to appear in the duodenal bulb after a delay of 4-8 hous after administration of cysteamine, but despite of several studies, the pathogenesis is not fully understood yet. The aim of the present study was to investigate the role of the various factors such as acid, motility, GABA, and primary afferent neuron on cysteamine-induced duodenal ulcer through the experiments using the Sprague-Dawley rats. (continue...)
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알코올에 의한 간손상시 적혈구막 지질 과산화현상의 역할
함기백,김원호,전재윤,문영명,강진경,박인서,최홍재,장학철,김한수,백승운,신용운 ( Ki Baik Hahm,Won Ho Kim,Chae Yoon Chon,Young Myung Moon,Jin Kyung Kang,In Suh Park,Heung Jai Choi,Hak Chul Jang,Han Soo Kim,Seung Woon Paik,Yong Woon Shin 대한소화기학회 1992 대한소화기학회지 Vol.24 No.2
The question of whether or not ethanol-induced liver injury is related to or accompanied by lipid peroxidation has been a subject of controversy. Short-term ethanol administration was shown by some to produce hepatic lipid peroxidation in rats, but very large doses of ethaol had to be used. Furthermore, some investigators failed to confirm these effects of ethanol. Fewer studies have explored the relationship between alcohol consumption and lipid peroxidation. In order to study this question and clarify the role of oxygen derived free radicals and lipid peroxidation of the RBC in inducing the alcoholic liver diseases, we measured the thiobarbituric acid reactivities in normal controls and patients with alcoholic liver disease. The subjects were divided into three groups according to the liver function tests and the amounts of alcohol drinking: group I-normal controls, group II-cases showing normal liver function test in spite of frequent alcohol drinking over 3 days per week and 45 g alcohol per one time, group III-patients with alcoholic liver disaesae, drinking alcohol over 4 times per week. The thiobarbituric acid reactive substances were measured as a marker of lipid peroxidation by the method of Uchiyama and Mihara. The colorimetric method was used by a measurment of malonyldialdehyde [MDA, nmole/ml (packed cells)] concentration. The results were as follows; (continue...)
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