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- 저자 : 최홍재 ( Ki Baik Hahm (검색결과 4 건)
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위암환자에서 수술전 Polyadenylic-Polyuridylic Acid 투여가 말초혈액과 영역림프절 단핵구의 NK 및 LAK 활성도에 미치는 영향
김원호,함기백,강진경,박인서,최홍재,전미연,노성훈,민진식,윤정구 ( Won Ho Kim,Ki Baik Hahm,Jin Kyung Kang,In Suh Park,Heung Jai Choi,Mi Youn Jun,Sung Hoon Noh,Jin Sik Min,Jung Koo Youn ) 대한소화기학회 1992 대한소화기학회지 Vol.24 No.2
Although the biological significance of the regional lymphnode as a barrier to tumor remains unclear, the clinical prognostic relevance of the neoplastic infiltration of these nodes is accepted. The most potent antitumor effector cells may be expected to be present within or around the site of tumor growth. However, lymphocytes isolated from solid tumor, cancer ascites fluid, and carcinomatous pleural effusion have been shown to expresse low natural killer (NK) activity or none at all. Similarly, it has been reported that lymphnode can be regarded as an NK-deficient tissue, since more limited cytotoxic activity is recorded in the lymphnode cells. (continue...)
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각종 간질환 환자에서 혈중 lipoprotein ( a ) 농도의 변화
함기백,김원호,한광협,전재윤,강진경,박인서,최홍재 ( Ki Baik Hahm,Won Ho Kim,Kwang Hyub Han,Chae Yoon Chon,Jin Kyung Kang,In Suh Park,Heung Jai Choi ) 대한소화기학회 1992 대한소화기학회지 Vol.24 No.2
Lipoprotein (a) [Lp (a)] is a LDL-like lipoprotein whose concentrations in plasma are significantly are correlated with atherosclerosis. The characteristic protein compoment of Lp (a) is apoliporotein (a) which is linked to apolipoprotein B-100 by disulfide bonds. Although Lp (a) might play an important role in the development of premature atherosclerotic vascular lesions, only scarse information about the metabolism of this lipoprotein is available. The major site of synthesis of Lp (a) appers to be the liver because it was found that a complete conversion of the Lp (a) phenotype of thc receipient to that of the donor occurs after liver transplantation and presence of apo (a) mRNA in human liver and hepatoma cell line. Until the present time, little information is available about determining the influence of liver disease on Lp (a) concentration. This study was designed to find out the changes of Lp (a) concentrations in 88 patients with various liver diseases, diagnosed histologically through the peritoneoscopic liver biopsy. The Lp (a) concentrations were checked by the method of radioimmunoassay. The mean Lp (a) concentrations of CAH with LC (6.1+4.8 mg/ml) and LC group (4.1+2,2 mg/ml) were significantly lower than those of normal control (24.4+19.1 mg/ml), CPH (25.4+18.5mg/ml), and CAH group (16.6+13.7 mg/ml). The Lp (a) concentrations were significantly correlated with serum albumin concentrations (r=0.54 p<0.05) and prothrombin time (r=0.62, p<0.01). There was a significant difference in mean Lp (a) coincentration between the cases with serum albumin above and below 3.0 g/ml in patients with liver cirrhosis. These data suggest that lipoprotein (a) may be secreted frorn the liver in accordance with the capability of liver to synthesize the protein and we find out that as the liver diseases progressed and deteriorated, the levels of Lp (a) concentrations were significantly decreased.
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알코올에 의한 간손상시 적혈구막 지질 과산화현상의 역할
함기백,김원호,전재윤,문영명,강진경,박인서,최홍재,장학철,김한수,백승운,신용운 ( Ki Baik Hahm,Won Ho Kim,Chae Yoon Chon,Young Myung Moon,Jin Kyung Kang,In Suh Park,Heung Jai Choi,Hak Chul Jang,Han Soo Kim,Seung Woon Paik,Yong Woon Shin 대한소화기학회 1992 대한소화기학회지 Vol.24 No.2
The question of whether or not ethanol-induced liver injury is related to or accompanied by lipid peroxidation has been a subject of controversy. Short-term ethanol administration was shown by some to produce hepatic lipid peroxidation in rats, but very large doses of ethaol had to be used. Furthermore, some investigators failed to confirm these effects of ethanol. Fewer studies have explored the relationship between alcohol consumption and lipid peroxidation. In order to study this question and clarify the role of oxygen derived free radicals and lipid peroxidation of the RBC in inducing the alcoholic liver diseases, we measured the thiobarbituric acid reactivities in normal controls and patients with alcoholic liver disease. The subjects were divided into three groups according to the liver function tests and the amounts of alcohol drinking: group I-normal controls, group II-cases showing normal liver function test in spite of frequent alcohol drinking over 3 days per week and 45 g alcohol per one time, group III-patients with alcoholic liver disaesae, drinking alcohol over 4 times per week. The thiobarbituric acid reactive substances were measured as a marker of lipid peroxidation by the method of Uchiyama and Mihara. The colorimetric method was used by a measurment of malonyldialdehyde [MDA, nmole/ml (packed cells)] concentration. The results were as follows; (continue...)
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Cysteamine 이 백서의 십이지장궤양 형성시 미치는 영향
함기백,이상인,임대순,김원호,전재윤,문영명,강진경,박인서,최홍재,박형우 ( Ki Baik Hahm,Sang In Lee,Dae Soon Yim,Won Ho Kim,Chae Yoon Chon,Young Myung Moon,Jin Kyung Kang,In Suh Park,Heung Jai Choi,Hyung Woo Park ) 대한소화기학회 1992 대한소화기학회지 Vol.24 No.4
The administration of cysteamine is koown to produce duodenal ulcer in rat. Mucosal damage starts to appear in the duodenal bulb after a delay of 4-8 hous after administration of cysteamine, but despite of several studies, the pathogenesis is not fully understood yet. The aim of the present study was to investigate the role of the various factors such as acid, motility, GABA, and primary afferent neuron on cysteamine-induced duodenal ulcer through the experiments using the Sprague-Dawley rats. (continue...)
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