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Oh, Yu-Ri,Lee, Joong-Bok,Park, Seung-Yong,Song, Chang-Seon,Choi, In-Soo,Kim, Yong-Hyun,Han, Eun-Jung,Lee, Jung-Hee,Lim, Kwang-Sei,Huh, Chul-Sung,Kim, Seong-Hee,Park, Sang-Shin,Lee, Sang-Won The Korean Society of Veterinary Science 2008 大韓獸醫學會誌 Vol.48 No.2
Lactic acid bacteria have been reported their beneficial roles on host including reduction of infectious diarrhea problems. In this study, preventive effect of Lactobacillus (L.) reuteri HY25101 and L. johnsonii HY25103 on porcine epidemic diarrhea virus (PEDV) was investigated in suckling piglets. Two groups of one day old PEDV naïve piglets were orally administered L. reuteri HY25101 and L. johnsonii HY25103 for three days respectively before challenge with lethal dose of PEDV. In second experiment, passive immunized one day old piglets using colostrums containing PEDV specific IgA were used. The survival rates of the L. reuteri HY25101 administered group were significantly higher than that of L. johnsonii HY25103 administered group and viral shedding was rapidly diminished in L. reuteri HY25101 administered group. Interestingly piglets born from the sow immunized with attenuated PEDV vaccine were not completely protected from PEDV challenge, however coadministeration of L. reuteri HY25101 and colostrums containing PEDV specific IgA were more effectively prevent PEDV infection. These results suggested that dietary treatment using L. reuteri HY25101 could reduce diarrheal problem and mortality rate caused by PEDV in suckling pigs. In addition, L. reuteri HY25101 could be used as one of effective compensation treatment with attenuated live vaccine for PED.
Analysis of toxicity of tetrabutyltin: comparing with EDC chemicals
Yu Ri An,Seung Jun Kim,Hye-Won Park,So Yeon Yu,Jeong Han,Jung-Hwa Oh,Seok Joo Yoon,황승용 대한독성 유전단백체 학회 2011 Molecular & cellular toxicology Vol.7 No.1
Exposure to environmental chemicals has been implicated in a number of adverse health effects,including immunotoxic, neurotoxic, reproductive toxic effects, and, above all, cancers. In the present study,we investigated toxicity of three environmental chemicals-bisphenol A (BPA), nonylphenol (NP), and tetrabutyltin (TTBT) in a human breast cancer cell line by HazChem Human array. We then found toxicity markers of TTBT through comparison with BPA and NP. Fifty three genes were differentially expressed and seventeen genes showed similar expression patterns. Ontology analysis showed differential expression of genes related to aging and apoptosis and genes having similarities associated with pregnancy and insemination. In this paper, we revealed potential TTBT toxic markers and endotoxicity of TTBT. This study could be a foundation for additional studies of TTBT toxicity.
Identification of time-dependent biomarkers by EndoTox Array in cells exposed to nonylphenol
Yu Ri An,So-Yeon Yu,Seung Jun Kim,Jung-Mi Ha,Jong-Phil Youn,Jun-Sub Kim,Moon-Ju Oh,Jung-Hwa Oh,류재천,Seok Joo Yoon,Jaehoon Jo,황승용 대한독성 유전단백체 학회 2011 Molecular & cellular toxicology Vol.7 No.4
Nonylphenol (NP) is considered an endocrine disruptor due to its weak ability to mimic estrogen and in turn disrupt the natural balance of hormones in affected organisms. NP is reported to cause negative health effects in humans, such as hormone abnormalities and inhibition of growth and reproduction. In the present study, we developed a molecular tool for the evaluation of endocrine toxicity in mouse. To identify gene regulation effects of NP, we estimated gene expression in mouse Sertoli (TM4) and germ cell (GC)lines after exposure to NP. We measured the IC30 value of NP, then exposed the cells to that concentration for 3 hr and 24 hr and used EndoTox Array. The EndoTox Array was manufactured to monitor the endotoxicity of environmental chemicals. This array contains 1306genes that are influenced in reproductive toxicity or by EDCs. In the expression pattern analysis, 28 genes related to the reproductive process, cell proliferation,and nervous system development were progressively changed over time in NP-exposed cells. The study of gene interaction will increase our understanding of the time-dependent molecular mechanisms of NP.