http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
박용범 ( Yong Beom Park ),이수곤 ( Soo Kon Lee ) 대한류마티스학회 2003 대한류마티스학회지 Vol.10 No.4
It is well known that rheumatoid arthritis (RA) causes significant morbidity as a result of synovial inflammation, joint destruction, and associated disability. In addition to these articular manifestations of RA, there is growing recognition of an excess mortality, which is due to increased atherosclerosis. Approximately 50% of atherosclerotic coronary artery disease in the community occurs in the absence of traditional risk factors, such as smoking, hypertension, diabetes mellitus, and hypercholesterolemia. Recently, inflammation has emerged as an important pathogenic mechanism of atherosclerosis. Inflammation has a role in both the initiation and the progression of atherosclerosis. C-reactive protein (CRP) is a sensitive marker for underlying systemic inflammation. Prospective studies indicate that baseline levels of CRP are associated with increased risk of myocardial infarction and stroke among apparently healthy individuals. Furthermore, the value of high-sensitivity testing for CRP appears to be additive to that of total and HDL-cholesterol for cardiovascular risk prediction. RA is a typical chronic inflammatory disease and CRP well reflects on the disease activity of RA. Several studies have reported increased cardiovascular disease and mortality among patients with RA. As a potential underlying mechanism for this observation, the inflammation of RA may play an important role. Studies have shown evidences that patients with RA have accelerated atherosclerosis, which is associated with the inflammation of RA. In this article, we reviewed the relationship between RA and accelerated atherosclerosis.
박용범(Park Yong Beom),김학신(Kim Hag Sin) 한국사회체육학회 2003 한국사회체육학회지 Vol.20 No.1
The Purpose of this study is to propose the direction for the policy which should be promoted by the government about the facility and program for the leisure for the old people, and the following conclusion could be introduced after the research with the above purpose. Firstly, in the leisure facility for the old, most of all when newly building the apartment the detailed regulation about the facility of the special section for the old should be strongly presented, and the improvement and repair for the facility of the timeworn special section for the old are needed. Additionally, in the charged facility, the decrease of the facility fee for the old and the plan which enables the old and the young to join together should be considered. Secondly, as for the recreation program for the old, the specific programs which are suitable for each level`s characteristics of the old should be developed, and the physical convention for the old such as the national physical convention for the old should be promoted to be the regular event. As for the leisure education program for the old, it is thought to be desirable that all the national and public universities in the country should manage the attached educational organizations annually. Thirdly, the colleague clubs should be organized according to the hobby and desire of the old, and the government should actively support those groups, and also the education and arrangement of the specialized leader for the leisure of the old should be considered. Additionally, the plan to effectively utilize the supporting system between the leisure facility for the old and local society should be arranged.
통상성 간질성 폐렴 환자 예후인자로서의 섬유모세포병소(fibroblastic foci)의 유용성
박용범 ( Park Yong Beom ),강길현 ( Kang Gil Hyeon ),심태선 ( Sim Tae Seon ),임채만 ( Im Chae Man ),이상도 ( Lee Sang Do ),고윤석 ( Go Yun Seog ),김우성 ( Kim U Seong ),김원동 ( Kim Won Dong ),( M. Kitaichi ),김동순 ( Kim Dong S 대한결핵 및 호흡기학회 2002 Tuberculosis and Respiratory Diseases Vol.53 No.3
박용범 ( Yong Beom Park ),서창희 ( Chang Hee Suh ),고원기 ( Won Ki Ko ),이원기 ( Won Ki Lee ),이충원 ( Choong Won Lee ),이찬희 ( Chan Hee Lee ),송창호 ( Chang Ho Song ),이지수 ( Ji Soo Lee ),이수곤 ( Soo Kon Lee ) 대한류마티스학회 1998 대한류마티스학회지 Vol.5 No.1
Systemic lupus erythematosus (SLE) is a multi systemic disease that can involve the gastrointestinal tract, liver, and biliary system. Symptomatic pancreatic involvement, however, has rarely been reported. It may be part of the primary disease process, such as vasculitic or autoimmune etiology, or associated with drug therapy, in particular corticosteroid. We report here a lupus patient who developed severe pancreatitis within 30 hours of initiation of corticosteroid therapy; we also discuss the relation between pancreatitis and systemic lupus erythematosus.
박용범(Yong Beom Park),이수곤(Soo Kon Lee) 대한내과학회 2002 대한내과학회지 Vol.63 No.1
Recently, inflammation has emerged as an important pathogenic mechanism of atherosclerosis. Inflammation has a role in both the initiation and the progression of atherosclerosis and anti-inflammatory agents may have a role in the prevention of cardiovascular disease. C-reactive protein (CRP) is an acute-phase reactant that is a sensitive marker for underlying systemic inflammation. Elevated plasma concentrations of CRP have been reported in patients with acute ischemia or myocardial infarction. Prospective studies indicate that baseline levels of CRP are associated with increased risk of myocardial infarction and stroke among apparently healthy individuals and those with symptomatic angina pectoris or prior myocardial infarction. Furthermore, the value of high-sensitivity testing for CRP appears to be additive to that of total and HDL-cholesterol for cardiovascular risk prediction. The data demonstrating associations between inflammation markers and cardiovascular disease (CVD) have been based on patients without apparent chronic inflammatory conditions. Therefore, it is logical to investigate if the same implications hold among patients with chronic inflammation as an underlying disease. Rheumatoid arthritis is a typical chronic inflammatory disease and CRP well reflects on the disease activity of RA. A number of studies have suggested increased cardiovascular disease and mortality among patients with rheumatoid arthritis. As a potential underlying mechanism for this observation, we have recently shown active untreated RA is associated with an adverse lipid profile that is conventionally accepted as a risk factor for cardiovascular disease. In a subsequent prospective inception cohort study, we have shown the adverse lipid profile can be improved to an extent that is clinically meaningful by effectively treating RA without using a lipid-lowering agent. Our data indicate that better control of rheumatoid arthritis is associated with a better lipid profile that may reduce the risk of cardiovascular disease. We also found morphologic and functional evidence of subclinical accelerated atherosclerosis in patients with RA. We have shown that RA patients had increased intima-media thickness (IMT) of carotid artery and there is decreased vascular endothelial function in patients with RA. The degree of inflammation is well correlated with IMT and endothelial dysfunction in RA patients. In conclusion, RA patients have risk for accelerated atherosclerosis, which may be associated with the inflammation of RA.