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Ya-Xiong Wang,Fei-Fei Qin,Kai Ou,Young-Bae Kim 전력전자학회 2015 ICPE(ISPE)논문집 Vol.2015 No.6
DC/DC converters with hybrid proton exchange membrane (PEM) fuel cell and battery power sources are designed and implemented using sliding mode control (SMC). The converters are typically comprised by unidirectional boost converter and bidirectional converter which are used for regulating the bus voltage and managing power distribution. SMC is selected to control the converters since it can cope with the high non-linear characteristic of the coupled system as well as ensure the stability. The hybrid power control system is constructed in MATLAB/Simulink platform. A comparative approach based on conventional proportional-integral (PI) control is developed to compare the control performance with SMC. The experimental validation is carried out with National Instruments (NI) LabVIEW-based hybrid PEMFC/battery supplied DC/DC converters system prototype.
Temperature Control for a Polymer Electrolyte Membrane Fuel Cell by Using Fuzzy Rule
Ya-Xiong Wang,Fei-Fei Qin,Kai Ou,Young-Bae Kim IEEE 2016 IEEE transactions on energy conversion Vol.31 No.2
<P>Temperature control for a polymer electrolyte membrane fuel cell (PEMFC) is important in improving power efficiency and increasing fuel cell lifetime. If the temperature of the cell is too low, then the electrochemical reaction response becomes slow, thereby preventing evaporations of liquid water in the membrane. As a result, cell performance is decreased. However, too high temperature leads to waste in catalyst and heat because of excessive chemical reactions and to liquid water evaporation, which decreases proton conductivity. This study develops an electrochemical dynamical model and a thermal model of a PEMFC using MATLAB/Simulink for simulation. Fuzzy control rules are also built to regulate the temperature of a PEMFC. The fuzzy inputs include temperature error, its derivative, and external load current. The cooling fan speed is chosen as an output variable to regulate the temperature of a fuzzy control because the fuel cell utilizes the air cooling method. After confirming that the designed fuzzy rules are effective for controlling cell temperature, a real experimental device is built using an H-100 fuel cell and a cooling fan to verify the effectiveness of the proposed method.</P>
Fei-Fei Qin,Ya-Xiong Wang,Young-Bae Kim 대한기계학회 2015 대한기계학회 춘추학술대회 Vol.춘계 No.-
고분자 전해질 연료전지의 열관리는 연료전지 작업 효율을 향상시키고 수명을 연장시키기는 매우 중요한 역할을 한다. 스택 온도가 너무 낮으면 전기 화학 반응의 속도가 늦어지고 반응에 의해 생성되는 물의 증발을 방해 한다. 그러나, 너무 높으면 온도는 촉매 낭비, 열 손실, 폐기물 방출 등 연료 전지의 수명을 줄인다. 본 논문은 고분자 전해질 연료전지(PEMFC) 열 관리를 위한 연구이다. 우선, 연료전지의 열역학 이론에 기초하여, MATLAB / Simulink를 통해 연료전지의 동적 모델을 만들었다. 실험을 통해 수학적인 시뮬레이션 모델링을 검증했다. PEMFC 동적 모델에 기초하여, 본 연구는 퍼지 논리 전략을 통하여 열 관리 제어 시스템을 설계 하였다. 이상적인 온도(PEMFC 효율 최적의 온도 곡선)와 스택 실제 온도의 오차, 그 오차의 미분 항, 및 외부 간섭 조건 전류(PEMFC 현재 스택)로 퍼지 제어기의 입력으로 구성 되었으며, 제어기의 출력은 냉각 팬의 제어전압이다. 본 연구는 열 관리를 위한 처음으로 외부 간섭 조전, 즉, 스택 전류를 제어기에 도입하여 복잡한 한 퍼지 제어기를 만들었다. 3차원 퍼지 제어기는 간단한 퍼지 제어기를 통해 발생한 정적 오류를 줄일 수 있다. 뿐만 아니라 제어 최적화 성능을 향상시켰다. 퍼지 논리 제어방법을 통해 연료전지 열 관리를 빠른 응답 시간과 작은 오차의 오버 슈트를 얻을 수 있다. 즉, 더 효율적으로 제어 성능을 향상시켰다.
Chen, Hai-Fei,Li, Zheng-Yang,Tang, Jie-Qing,Shen, Hong-Shi,Cui, Qing-Ya,Ren, Yong-Ya,Qin, Long-Mei,Jin, Ling-Juan,Zhu, Jing-Jing,Wang, Jing,Ding, Jie,Wang, Ke-Yuan,Yu, Zi-Qiang,Wang, Zhao-Yue,Wu, Tian Asian Pacific Journal of Cancer Prevention 2012 Asian Pacific journal of cancer prevention Vol.13 No.9
Objective: To investigate the relationship between the efficacy and safety of different doses of thalidomide (Thal) plus dexamethasone (Dex) as the initial therapy in elderly patients with newly diagnosed multiple myeloma (MM). Methods: Clinical data of 28 elderly patients with newly diagnosed MM who underwent the TD regimen as the initial therapy were analyzed retrospectively. The patients were divided into two groups according to the maximal sustained dose of Thal: lower dose (group A) and higher dose (group B). The overall response rate (ORR), progression free survival (PFS), overall survival (OS), and adverse events (AES) were compared between the two groups. Results: A total of 28 patients were followed up with a median of 18 months. The ORR was 60.1%. The median response time and PFS were 2.0 and 17.0 months, respectively. The mean sustained dose of Thal in group B was significantly higher than group A (292.9 mg v 180.4 mg, P=0.01). There was no significantly difference in ORR (57.1% v 64.3%, P=1.00) and PFS (9.63months v 17.66 months, P=0.73) between groups A and B. During the follow up, only five patients died (<40%) and, therefore, median OS values were not available. It is estimated, however, that the mean survival time in the two groups was 35.6 and 33.4 months (P>0.05), respectively. All of the patients tolerated the treatment well. The incidence of AES in patients with a grading above 3 in group B was significantly higher than in group A (P=0.033). Conclusions: The TD regimen results in a high response rate and manageable AES as the initial therapy in elderly patients with MM. TD should be considered as the front line regimen for the treatment of elderly patients with MM in areas with financial constraints. The clinical response can be achieved at a low dose Thal with minimal toxicity.
Fei Xiaowei,Dou Ya-nan,Sun Kai,Wei Jialiang,Guo Qingdong,Wang Li,Wu Xiuquan,Lv Weihao,Jiang Xiaofan,Fei Zhou 생화학분자생물학회 2023 Experimental and molecular medicine Vol.55 No.-
The tripartite motif (TRIM) 22 and mitogen-activated protein kinase (MAPK) signaling pathways play critical roles in the growth of glioblastoma (GBM). However, the molecular mechanism underlying the relationship between TRIM22 and MAPK signaling remains unclear. Here, we found that TRIM22 binds to exon 2 of the sphingosine kinase 2 (SPHK2) gene. An ERK1/2-driven luciferase reporter construct identified TRIM22 as a potential activator of MAPK signaling. Knockout and overexpression of TRIM22 regulate the inhibition and activation of MAPK signaling through the RING-finger domain. TRIM22 binds to Raf-1, a negative regulator of MAPK signaling, and accelerates its degradation by inducing K48-linked ubiquitination, which is related to the CC and SPRY domains of TRIM22 and the C1D domain of Raf-1. In vitro and in vivo, an SPHK2 inhibitor (K145), an ERK1/2 inhibitor (selumetinib), and the nonphosphorylated mutant Raf-1S338A inhibited GBM growth. In addition, deletion of the RING domain and the nuclear localization sequence of TRIM22 significantly inhibited TRIM22-induced proliferation of GBM cells in vivo and in vitro. In conclusion, our study showed that TRIM22 regulates SPHK2 transcription and activates MAPK signaling through posttranslational modification of two critical regulators of MAPK signaling in GBM cells.