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Estimation for a Hand Position and Orientation Robust to Motion of Fingers Using a Web Camera
Y. Mizuchi,H. Kawai,Y. Hagiwara,H. Imamura,Y. Choi 제어로봇시스템학회 2010 제어로봇시스템학회 국제학술대회 논문집 Vol.2010 No.10
We present a method to estimate the position and the orientation of a hand from a captured image for applications which need to manipulate instinctively and freely using a hand. We use a hand as a tool instead of an input device such as a mouse or a marker, and we use only a web camera instead of multiple-camera or an infrared camera, in order to improve mobility and usability. We focused on four valley-points between neighboring fingers as invariant feature points. By detecting four valley-points, the method makes possible the robust estimation for the position and the orientation of a hand regardless of the bending and the closing of fingers. In the method, we are able to move fingers freely when all valley-points are viewable. In an experiment, we qualitatively evaluated the robustness and usefulness of the proposed method by drawing 3DCG objects.
Iwamura Y,Imamura T,Tokanaga E,Matsuo K 대한수의사회 1995 대한수의사회지 Vol.31 No.8
일본에서 시판되고 있는 4종류의 생독백신은 본병방어에 있어 어느 것이나 유효하다. 그러나 각기 백신의 성상은 각자 특성이 있음으로 유효성의 정도에 차이가 있다고 본다. 이런 관점에서 백신 유효성을 공격주를 바꾸어서 또한 이행항체 보유돈에서 비교 검토한 결과, 백신 D가 종합적으로 볼 때 가장 높고 다음 C, B, 최후의 A의 순이었다.
Applicability of Nuclear Reaction Models Implemented in PHITS to Simulations on Single-event Effects
S. Abe,S. Hirayama,Y. Watanabe,N. Sano,Y. Tosaka,M. Tsutsui,H. Furuta,T. Imamura 한국물리학회 2011 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.59 No.23
The nuclear reaction models implemented in the PHITS (Particle and Heavy Ion Transport code System), i.e., the INC model, the QMD model and "event generator mode (e-mode)" with the JENDL-3.3 are validated to apply it to simulations on single-event effects. The model calculations are compared with available experimental data of light-ion (proton and alpha particle) production from neutron-induced reactions on ^(nat)Si and ^(16)O for neutron energies below 100 MeV. Since there is no measurement of lightion production from Si for neutron energies more than 100 MeV, proton-induced reactions on ^(27)Al are also used in the model validation. Comparisons of calculated and measured data for lightion production show that the e-mode calculation with JENDL-3.3 provides better agreement with the experimental data below 20 MeV than the INC and QMD models, and the QMD model reproduces them well above 20 MeV.
Miyake, N.,Tsukaguchi, H.,Koshimizu, E.,Shono, A.,Matsunaga, S.,Shiina, M.,Mimura, Y.,Imamura, S.,Hirose, T.,Okudela, K.,Nozu, K.,Akioka, Y.,Hattori, M.,Yoshikawa, N.,Kitamura, A.,Cheong, H.,Kagami, S University of Chicago Press [etc.] 2015 American journal of human genetics Vol.97 No.4
The nuclear pore complex (NPC) is a huge protein complex embedded in the nuclear envelope. It has central functions in nucleocytoplasmic transport, nuclear framework, and gene regulation. Nucleoporin 107 kDa (NUP107) is a component of the NPC central scaffold and is an essential protein in all eukaryotic cells. Here, we report on biallelic NUP107 mutations in nine affected individuals who are from five unrelated families and show early-onset steroid-resistant nephrotic syndrome (SRNS). These individuals have pathologically focal segmental glomerulosclerosis, a condition that leads to end-stage renal disease with high frequency. NUP107 is ubiquitously expressed, including in glomerular podocytes. Three of four NUP107 mutations detected in the affected individuals hamper NUP107 binding to NUP133 (nucleoporin 133 kDa) and NUP107 incorporation into NPCs in vitro. Zebrafish with nup107 knockdown generated by morpholino oligonucleotides displayed hypoplastic glomerulus structures and abnormal podocyte foot processes, thereby mimicking the pathological changes seen in the kidneys of the SRNS individuals with NUP107 mutations. Considering the unique properties of the podocyte (highly differentiated foot-process architecture and slit membrane and the inability to regenerate), we propose a ''podocyte-injury model'' as the pathomechanism for SRNS due to biallelic NUP107 mutations.