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Cho, Y.,Cho, H.,Park, M.,Ma, K. Korean Association of Organic Agriculture 2011 韓國有機農業學會誌 Vol.19 No.S
Organic production practices varied among producers. Generally, organic producers were relying on imported input materials such as organic compost and liquid fertilizer even more than conventional producers. Very few organic farmers had composting facilities or sites for the own supply of compost in need. The productivity of organic kiwifruit orchard (92%) was not as low as that of conventional while the net income (243%) was more than double that of conventional. This was mainly attributed to high farm gate price of organic fruits, low paid labour use and electricity. As a consequence, organic kiwifruit production seems to become a feasible option in Korea. However, high dependence on imported farming material, fuel and labour for too frequent liquid fertilizer spray should be addressed to achieve long term sustainability of organic kiwifruit production.
Miniaturised dual-band implantable antenna for wireless biotelemetry
Cho, Y.,Yoo, H. IET 2016 Electronics letters Vol.52 No.12
<P>A miniaturised implantable antenna with dual-band operation the Medical Implant Communications Service (MICS) (402-405 MHz) and Industrial, Scientific, and Medical (ISM) (2400.0-2483.5 MHz) bands is presented. The size of the proposed antenna is 31.5 mm(3) (8.75 mm x 7.2 mm x 0.5 mm) which is the smallest size compared to previous implantable antennas. A serpentine-shaped radiating patch and open-end slot placed on the ground plane are used for miniaturisation. The performance of the antenna was evaluated from measurements and is based on good agreement with simulations.</P>
Cho, Y.,Lee, J.,Nguyen, T.T.H.,Bae, J.W.,Yu, T.,Lim, B. Korean Society of Industrial and Engineering Chemi 2016 Journal of industrial and engineering chemistry Vol.37 No.-
<P>We describe a facile synthetic route to alpha-Co(OH)(2) nanostructures by heating a solution containing CoCl2, ethylene glycol, and oleylamine. Electron microscopy and X-ray diffraction analyses revealed that flower-like nanostructures of a-Co(OH)2 were formed by assembly of thin alpha-Co(OH)(2) nanosheets. These nanostructures exhibited high activity for electrochemical water splitting with a current density comparable to that obtained on the commercial Pt wire electrode. In addition, these flower-like alpha-Co(OH)(2) nanostructures showed specific capacitance as high as 440 F/g at a current density of 1 A/g and excellent stability without any noticeable loss in capacitance after 5000 cycles of the galvanostatic charge-discharge test. (C) 2016 Published by Elsevier B.V. on behalf of The Korean Society of Industrial and Engineering Chemistry.</P>
Cho, Y.,Lee, S.E.,Lee, H.C.,Hur, J.,Lee, S.,Youn, S.W.,Lee, J.,Lee, H.J.,Lee, T.K.,Park, J.,Hwang, S.J.,Kwon, Y.W.,Cho, H.J.,Oh, B.H.,Park, Y.B.,Kim, H.S. Elsevier Biomedical 2011 JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY - Vol.57 No.1
Objectives: We investigated the effects of human resistin on atherosclerotic progression and clarified its underlying mechanisms. Background: Resistin is an adipokine first identified as a mediator of insulin resistance in murine obesity models. But, its role in human pathology is under debate. Although a few recent studies suggested the relationship between resistin and atherosclerosis in humans, the causal relationship and underlying mechanism have not been clarified. Methods: We cloned rabbit resistin, which showed 78% identity to human resistin at the complementary deoxyribonucleic acid level, and its expression was examined in 3 different atherosclerotic rabbit models. To evaluate direct role of resistin on atherosclerosis, collared rabbit carotid arteries were used. Histological and cell biologic analyses were performed. Results: Rabbit resistin was expressed by macrophages of the plaque in the 3 different atherosclerotic models. Peri-adventitial resistin gene transfer induced macrophage infiltration and expression of various inflammatory cytokines, resulting in the acceleration of plaque growth and destabilization. In vitro experiments elucidated that resistin increased monocyte-endothelial cell adhesion by upregulating very late antigen-4 on monocytes and their counterpart vascular cell adhesion molecule-1 on endothelial cells. Resistin augmented monocyte infiltration in collagen by direct chemoattractive effect as well as by enhancing migration toward monocyte chemotactic protein-1. Administration of connecting segment-1 peptide, which blocks very late antigen-4 x vascular cell adhesion molecule-1 interaction, ameliorated neointimal growth induced by resistin in vivo. Conclusions: Our results indicate that resistin aggravates atherosclerosis by stimulating monocytes, endothelial cells, and vascular smooth muscle cells to induce vascular inflammation. These findings provide the first insight on the causal relationship between resistin and atherosclerosis.
Injury-Induced HDAC5 Nuclear Export Is Essential for Axon Regeneration
Cho, Y.,Sloutsky, R.,Naegle, Kristen M.,Cavalli, V. Cell Press ; MIT Press 2013 Cell Vol.155 No.4
Reactivation of a silent transcriptional program is a critical step in successful axon regeneration following injury. Yet how such a program is unlocked after injury remains largely unexplored. We found that axon injury in peripheral sensory neurons elicits a back-propagating calcium wave that invades the soma and causes nuclear export of HDAC5 in a PKCμ-dependent manner. Injury-induced HDAC5 nuclear export enhances histone acetylation to activate a proregenerative gene-expression program. HDAC5 nuclear export is required for axon regeneration, as expression of a nuclear-trapped HDAC5 mutant prevents axon regeneration, whereas enhancing HDAC5 nuclear export promotes axon regeneration in vitro and in vivo. Components of this HDAC5 pathway failed to be activated in a model of central nervous system injury. These studies reveal a signaling mechanism from the axon injury site to the soma that controls neuronal growth competence and suggest a role for HDAC5 as a transcriptional switch controlling axon regeneration.
Cho, Y.,Challa, S.,Moquin, D.,Genga, R.,Ray, T.D.,Guildford, M.,Chan, F.K.M. Cell Press ; MIT Press 2009 Cell Vol.137 No.6
Programmed necrosis is a form of caspase-independent cell death whose molecular regulation is poorly understood. The kinase RIP1 is crucial for programmed necrosis, but also mediates activation of the prosurvival transcription factor NF-κB. We postulated that additional molecules are required to specifically activate programmed necrosis. Using a RNA interference screen, we identified the kinase RIP3 as a crucial activator for programmed necrosis induced by TNF and during virus infection. RIP3 regulates necrosis-specific RIP1 phosphorylation. The phosphorylation of RIP1 and RIP3 stabilizes their association within the pronecrotic complex, activates the pronecrotic kinase activity, and triggers downstream reactive oxygen species production. The pronecrotic RIP1-RIP3 complex is induced during vaccinia virus infection. Consequently, RIP3<SUP>-/-</SUP> mice exhibited severely impaired virus-induced tissue necrosis, inflammation, and control of viral replication. Our findings suggest that RIP3 controls programmed necrosis by initiating the pronecrotic kinase cascade, and that this is necessary for the inflammatory response against virus infections.
Adaptive live streaming system performance of MMT and DASH over a deployed LTE network
Cho, Y.,Park, S.,Kim, K.,Suh, D.Y. IET 2016 Electronics letters Vol.52 No.13
<P>Moving Picture Expert Group (MPEG) media transport and dynamic adaptive streaming over Hypertext Transfer Protocol are compared, which have been standardised by the MPEG for internet-based video services. Their performances as adaptive live streaming systems are evaluated over a deployed long term evolution network. The results provide their operational limitations and optimal operation parameters to improve resource usage and the quality of the users' experience.</P>