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BiFeO3 Thickness Dependence of the Exchange Bias in Polycrystalline BiFeO3/NiFe Bilayers
Xiaobo Xue,Biao You,Wenbing Rui,Wei Zhang,Jun Du,Xueyong Yuan,Qingyu Xu 한국물리학회 2013 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.62 No.12
A clear exchange bias has been observed at room temperature in a series of BiFeO3(t)/NiFe(3.6nm) bilayers, in which the BiFeO3 layers are single-phase polycrystalline with t varied from 8 nmto 240 nm. With increasing t, both the exchange bias field (HE) and the coercivity (HC) increasesharply and approach maxima when t is about 40 nm, which is close to one half the spin cycloidalmodulation period (64 nm) of the bulk BiFeO3 material. The oscillatory variations of HE and HCwith the BiFeO3 layer thickness suggest that the cycloidal spin structure may exist in polycrystallineBiFeO3 thin films. The angular dependence of the exchange bias exhibits collinear unidirectionaland uniaxial anisotropies in the present BiFeO3/NiFe bilayers.
Jingcui Yu,Songbin Fu,Peng Liu,Xiaobo Cui,Yu Sui,Guohua Ji,Rongwei Guan,Donglin Sun,Wei Ji,Fangli Liu,An Liu,Yuzhen Zhao,Yang Yu,Yan Jin,Jing Bai,Jingshu Geng,Yingwei Xue,Jiping Qi,Ki-Young Lee 한국분자세포생물학회 2011 Molecules and cells Vol.32 No.1
Previously, we identified 3 overlapping regions showing loss of heterozygosity (LOH, R_1-R_3 from 11 to 30 cM) on chromosome 17 in 45 primary gastric cancers (GCs). The data indicated the presence of tumor suppressor genes (TSGs) on chromosome 17 involved in GC. Among the putative TSGs in these regions, HIC1 (in SR_1) and TOB1 (in SR_3) remain to be examined in GC. By immunohistochemistry (IHC), methylation-specific PCR (MSP) and western blot, we evaluated the expression and regulation status for HIC1 and TOB1 protein in GC. We narrowed down the deletion intervals on chromosome 17 and defined five smaller LOH subregions, SR_1-SR_5 (0.54 to 3.42 cM), in GC. We found that HIC1 had downregulated expression in 86% (91/106) and was methylated in 87% (26/30) of primary GCs. Of the primary GCs showing downregulation of HIC1 protein, 75% (18/24) had methylated HIC1 gene. TOB1 was either absent or expressed at reduced levels in 75% (73/97) of the GC samples. In addition, a general reduction was found in total and the ratio of unphosphorylated to phosphorylated TOB1 protein levels in the differentiated GC cell lines. Further analysis revealed significant simultaneous downregulation of both HIC1 and TOB1 protein in GC tissue microarray samples (67%, 52/78) and in primary GCs (65%, 11/17). These results indicate that silencing of HIC1 and TOB1 expression is a common occurrence in GC and may contribute to the development and progression of the disease.