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Peter G Walley,Graham R Teakle,Jonathan D Moore,Charlotte J Allender,Vicky Buchanan-Wollaston,David AC Pink,Guy C Barker 한국식물생명공학회 2012 식물생명공학회지 Vol.39 No.1
The vegetable brassicas are an important crop worldwide and are of significant commercial value. In order to ensure our targets for food security are met it is important that these crops are continually improved to increase sustainability of production, increase nutritional quality and reduce waste. Development of resistances against both biotic and abiotic stress are recognised as being key. Plant breeding plays a vital role in addressing these issues through the development of new and improved varieties. This continued improvement is becoming evermore dependent on our ability to identify and introgress beneficial alleles from ‘exotic’ germplasm into elite breeding material. Increasingly, more diverse germplasm such as those found in genebanks is being screened for benificial allelic variation, however, plant breeders often find it difficult to make use of such material due to the time required to remove undesirable characteristics from progeny due to linkage drag. This article describes how we have attempted to overcome this and develop resources that make the diversity available within the Brassica oleracea genepool more accessible.
Walley, Peter G.,Teakle, Graham R.,Moore, Jonathan D.,Allender, Charlotte J.,Pink, David A.C.,Buchanan-Wollaston, Vicky,Barker, Guy C. The Korean Society of Plant Biotechnology 2012 식물생명공학회지 Vol.39 No.1
The vegetable brassicas are an important crop worldwide and are of significant commercial value. In order to ensure our targets for food security are met it is important that these crops are continually improved to increase sustainability of production, increase nutritional quality and reduce waste. Development of resistances against both biotic and abiotic stress are recognised as being key. Plant breeding plays a vital role in addressing these issues through the development of new and improved varieties. This continued improvement is becoming evermore dependent on our ability to identify and introgress beneficial alleles from 'exotic' germplasm into elite breeding material. Increasingly, more diverse germplasm such as those found in genebanks is being screened for benificial allelic variation, however, plant breeders often find it difficult to make use of such material due to the time required to remove undesirable characteristics from progeny due to linkage drag. This article describes how we have attempted to overcome this and develop resources that make the diversity available within the $Brassica$ $oleracea$ genepool more accessible.
Kim, Jeongsik,Park, Su Jin,Lee, Il Hwan,Chu, Hyosub,Penfold, Christopher A,Kim, Jin Hee,Buchanan-Wollaston, Vicky,Nam, Hong Gil,Woo, Hye Ryun,Lim, Pyung Ok Oxford University Press 2018 Journal of experimental botany Vol.69 No.12
<▼1><P>Ethylene and cytokinin play antagonistic roles in the regulation of leaf senescence via EIN2/ORE3-dependent transcriptional regulation of stress responses and AHK3/ORE12-dependent transcriptional maintenance of the translational machinery, respectively.</P></▼1><▼2><P><B>Abstract</B></P><P>Leaf senescence involves degenerative but active biological processes that require balanced regulation of pro- and anti-senescing activities. Ethylene and cytokinin are major antagonistic regulatory hormones that control the timing and progression rate of leaf senescence. To identify the roles of these hormones in the regulation of leaf senescence in Arabidopsis, global gene expression profiles in detached leaves of the wild type, an ethylene-insensitive mutant (<I>ein2</I>/<I>ore3</I>), and a constitutive cytokinin response mutant (<I>ahk3/ore12</I>) were investigated during dark-induced leaf senescence. Comparative transcriptome analyses revealed that genes involved in oxidative or salt stress response were preferentially altered in the <I>ein2</I>/<I>ore3</I> mutant, whereas genes involved in ribosome biogenesis were affected in the <I>ahk3/ore12</I> mutant during dark-induced leaf senescence. Similar results were also obtained for developmental senescence. Through extensive molecular and physiological analyses in <I>ein2</I>/<I>ore3</I> and <I>ahk3/ore12</I> during dark-induced leaf senescence, together with responses when treated with cytokinin and ethylene inhibitor, we conclude that ethylene acts as a senescence-promoting factor via the transcriptional regulation of stress-related responses, whereas cytokinin acts as an anti-senescing agent by maintaining cellular activities and preserving the translational machinery. These findings provide new insights into how plants utilize two antagonistic hormones, ethylene and cytokinin, to regulate the molecular programming of leaf senescence.</P></▼2>
Lim, Pyung Ok,Kim, Yumi,Breeze, Emily,Koo, Ja Choon,Woo, Hye Ryun,Ryu, Jong Sang,Park, Don Ha,Beynon, Jim,Tabrett, Alex,Buchanan-Wollaston, Vicky,Nam, Hong Gil Blackwell Publishing Ltd 2007 The Plant journal Vol.52 No.6
<P>Summary</P><P>Leaf senescence is the final stage of leaf development and is finely regulated via a complex genetic regulatory network incorporating both developmental and environmental factors. In an effort to identify negative regulators of leaf senescence, we screened activation-tagged Arabidopsis lines for mutants that exhibit a delayed leaf senescence phenotype. One of the mutants (<I>ore7-1D</I>) showed a highly significant delay of leaf senescence in the heterozygous state, leading to at least a twofold increase in leaf longevity. The activated gene (<I>ORE7</I>/<I>ESC</I>) encoded a protein with an AT-hook DNA-binding motif; such proteins are known to co-regulate transcription of genes through modification of chromatin architecture. We showed that ORE7/ESC, in addition to binding to a plant AT-rich DNA fragment, could also modify the chromatin architecture, as illustrated by an altered distribution of a histone–GFP fusion protein in the nucleus of the mutant. Globally altered gene expression, shown by microarray analysis, also indicated that activation of <I>ORE7</I>/<I>ESC</I> results in a younger condition in the mutant leaves. We propose that ectopically expressed <I>ORE7</I>/<I>ESC</I> is negatively regulating leaf senescence and suggest that the resulting chromatin alteration may have a role in controlling leaf longevity. Interestingly, activation of <I>ORE7</I>/<I>ESC</I> also led to a highly extended post-harvest storage life.</P>