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Good Practices of Ergonomic Science Communication in Japan
Yasuyuki Yamada,Takeshi Ebara,Takashi Toriizuka 대한산업공학회 2022 Industrial Engineeering & Management Systems Vol.21 No.4
Science communication (SciComm) is a practical activity that maximizes the utility of academic findings, knowledge, and information. This study examined the good practices and future issues of SciComm in the field of ergonomics, in Japan. The first good practice was the establishment of the SciComm Committee in the Japan Human Factors and Ergonomics Society (JES) in 2020. The second was the ErgonomicThon workshop—an ergonomic Ideathon to discuss new ideas regarding the uses of ergonomic findings. The third was the original SciComm program using infographics to promote the use of ergonomic words in social networking service (SNS) communication. The fourth practice was the challenging online fieldwork conducted using the Google Street View. The last practice was the virtual convivial party held at the 62nd conference of JES in 2021. Based on these practices, we suggest the following three future issues of SciComm: (1) promotion of SciComm, beyond times of crisis; (2) development of ergonomic guidelines for SciComm; and, (3) promotion of online SciComm within the information society. This study outlines the importance of sharing good practices and future issues of SciComm that will help spread the movement of the SciComm in the field of ergonomics.
Downregulation of bcl-xL Is Relevant to UV-induced Apoptosis in Fibroblasts
Nakagawa, Yuki,Okada, Seiji,Hatano, Masahiko,Ebara, Masaaki,Saisho, Hiromitsu,Tokuhisa, Takeshi Korean Society for Biochemistry and Molecular Biol 2002 Journal of biochemistry and molecular biology Vol.35 No.5
Exposure to ultraviolet light (UV) induces apoptosis in mammalian cells, The caspase group of proteases is required for the appotosis. This pathway is initiated by a release of cytochrome c from the mitochondria into the cytosol. Several Bcl-2 family proteins can regulate the release of cytochrome c by stabilizing the mitochondrial membrane. Here we show that expression of the endogenous bcl-xL was strongly downregulated in NIH3T3 cells within 2 h after UV-C irradiation, and that of bax was upregulated from 8 h after irradiation. Apoptosis was induced in more than 50% of the NIH3T3 cells 48 h after irradiation. Constitutive overexpression of bcl-xL in NIH3T3 cells protected the UV-induced apoptosis by preventing the loss of mitochondrial membrane potential and the activation of caspase 9. There results suggest that downregulation of Bcl-xL is relevant to UV-induced apoptosis of tibroblasts.
Downregulation of bcl - xL Is Relevant to UV - induced Apoptosis in Fibroblasts
(Yuki Nakagawa),(Seiji Okada),(Masahiko Hatano),(Masaaki Ebara),(Hiromitsu Saisho),(Takeshi Tokuhisa) 생화학분자생물학회 2002 BMB Reports Vol.35 No.5
Exposure to ultraviolet light (UV) induces apoptosis in mammalian cells. The caspase group of proteases is required for the apoptosis. This pathway is initiated by a release of cytochrome c from the mitochondria into the cytosol. Several Bcl-2 family proteins can regulate the release of cytochrome c by stabilizing the mitochondrial membrane. Here we show that expression of the endogenous bcl-xL was strongly downregulated in NIH3T3 cells within 2 h after UV-C irradiation, and that of bax was upregulated from 8 h after irradiation. Apoptosis was induced in more than 50% of the NIH3T3 cells 48 h after irradiation. Constitutive overexpression of bcl-xL in NIH3T3 cells protected the UV-induced apoptosis by preventing the loss of mitochondrial membrane potential and the activation of caspase 9. These results suggest that downregulation of Bcl-xL is relevant to UV-induced apoptosis of fibroblasts.