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Case Reports : Acute Myocardial Infarction with Elevated ST-segment Only in Lead aVR
( Taek Geun Ohk ),( Gu Hyun Kang ),( Jung Rae Cho ),( Yong Soo Jang ),( Hee Cheol Ahn ),( Gyu Jong Cho ),( Jung Hwan Ahn ),( Jun Hwi Cho ) 대한응급의학회 2014 대한응급의학회지 Vol.25 No.2
Lead aVR ST segment elevation in patients with clinically suspected acute coronary syndrome strongly suggests the possibility of occlusion of the left main coronary artery (LMCA), and stenosis or occlusion in this area can cause severe life-threatening left ventricular dysfunction or malignant arrhythmias. Thus, it could be a sign suggestive of a poor prognosis for patients. In this study, we report on the case of a 67-year-old male who presented to the emergency department with total occlusion of LMCA with STsegment elevation in only lead aVR, and without ST-segment elevation in other leads.
Primary Aortoesophageal Fistula Presented as Massive Upper Gastrointestinal Bleeding
( Chan Woo Park ),( Taek Geun Ohk ),( Jun Hwi Cho ),( Joong Beom Moon ),( Myung Chul Shin ),( Ka Eul Kim ),( Go Eun Yang ),( Hui Young Lee ) 대한응급의학회 2017 대한응급의학회지 Vol.28 No.5
An aortoesophageal fistula is one of the very few causes of upper gastrointestinal bleeding but can be fatal if the diagnosis is delayed. This usually occurs secondary to esophageal or aortic surgery. A primary aortoesophageal fistula is rare and less likely to be suspected. Here, we present a case of a primary aortoesophageal fistula that presented as massive upper gastrointestinal bleeding. An 81-year-old man with a history of aortic aneurysm had syncope and bright color hematemesis. The aortoesophageal fistula was not diagnosed early enough and the patient died. Therefore, emergency physicians should consider aortoesophageal fistula as a potential cause when encountering upper gastrointestinal bleeding.
( Jeongkook Seo ),( Jun Hwi Cho ),( Taek Geun Ohk ),( Hui Young Lee ),( Chan Woo Park ) 대한응급의학회 2021 대한응급의학회지 Vol.32 No.6
Objective: Discriminating between pulmonary embolism (PE) and pneumonia in the emergency department (ED) is one of the fastidious tasks. Elderly patients are at more risk of PE and pneumonia than younger patients. This study aimed to determine whether the ratio of D-dimer to C-reactive protein (CRP) could be used as an adjunctive method to differentiate between PE and pneumonia in elderly patients. Methods: Medical records of patients visiting the ED diagnosed with PE and pneumonia were examined. Cutoff values of D-dimer (μg/mL) and the ratio of D-dimer to CRP ([μg/mL]/[mg/dL]) of subjects with PE or pneumonia were analyzed. Results: There were a total of 60 patients with PE and 152 patients with pneumonia. In addition, 15 patients had both PE and pneumonia. The cutoff value of D-dimer to differentiate between PE and pneumonia was 4.26 μg/mL (4,260 ng/mL) (area under curve [AUC], 0.861; sensitivity, 80.0%; specificity, 80.3%; 95% confidence interval [CI], 0.805-0.917). The cutoff value of the ratio of D-dimer to CRP to differentiate between PE and pneumonia was 1.24 ([μg/mL]/[mg/dL]) (AUC, 0.919; sensitivity, 84.4%; specificity, 84.2%; 95% CI, 0.878-0.960). Conclusion: The ratio of D-dimer to CRP can be used as an adjunctive method to determine whether a computed tomography pulmonary angiography or a ventilation-perfusion scan can be performed to differentiate between PE and pneumonia in elderly patients.
( Jun Hwi Cho ),( Chan Woo Park ),( Myoung Chul Shin ),( Taek Geun Ohk ),( Joon Ho Bae ),( Dae Jin Jeong ),( Jun Yeol Lee ),( Ka Eul Kim ),( Ki Joong Kim ),( Doo Hyun Lee ),( Jung Hoon Lee ),( Moo Ho 대한응급의학회 2014 대한응급의학회 학술대회초록집 Vol.2014 No.2
Tanshinone I (TsI) is an important lipophilic diterpene extracted from Danshen (Radix Salvia miltiorrhizae) and has been used in Asia for the treatment of cerebrovascular diseases such as ischemic stroke. In this study, we examined the neuroprotective effect of tanshinone I (TsI) against ischemic damage and its neuroprotective mechanism in the gerbil hippocampal CA1 region (CA1) induced by 5 min of transient global cerebral ischemia. Pre-treatment with TsI protected pyramidale neurons from ischemic damage in the stratum pyramidale of the CA1 after ischemiareperfusion. The pre-treatment with TsI increased the immunoreactivities and protein levels of anti-inflammatory cytokines (interleukin (IL)-4 and IL- 13) in the TsI-treated-sham-operated-groups compared with those in the vehicle-treated-sham-operated-groups; however, the treatment did not increase the immunoreactivities and protein levels of pro-inflammatory cytokines (IL-2 and tumor necrosis factor (TNF)-α). On the other hand, in the TsI-treated-ischemia-operated-groups, the immunoreactivities and protein levels of all the cytokines were maintained in the stratum pyramidal of the CA1 after transient ischemia. In conclusion, these findings indicate that the pre-treatment with TsI can protect against ischemia-induced neuronal death in the CA1 via the increase or maintenance of inflammatory cytokines.
( Jun Hwi Cho ),( Chan Woo Park ),( Myoung Chul Shin ),( Taek Geun Ohk ),( Joon Ho Bae ),( Dae Jin Jeong ),( Jun Yeol Lee ),( Ka Eul Kim ),( Ki Joong Kim ),( Doo Hyun Lee ),( Jung Hoon Lee ),( Moo Ho 대한응급의학회 2014 대한응급의학회 학술대회초록집 Vol.2014 No.2
The receptor for advanced glycation end products (RAGE) is a multi-ligand receptor of the immunoglobulin superfamily that has been implicated in multiple neuronal and inflammatory stress processes. In the present study, we investigated changes in RAGE immunoreactivity and its protein levels in the gerbil hippocampus (CA1-3 regions) after 5 min of transient global cerebral ischemia. The ischemic hippocampus was stained with cresyl violet (CV), NeuN (a neuron-specific soluble nuclear antigen) antibody and Fluro-Jade B (a marker for neuronal degeneration). Five days after ischemia-reperfusion, delayed neuronal death occurred in the stratum pyramidale (SP) of the CA1 region. RAGE immunoreactivity was not detected in any regions of the CA1-3 regions of the sham-group. RAGE immunoreactivity was detected only in the CA1 region from 3 days post-ischemia, and the RAGE immunoreactivity was newly expressed in astrocytes, not in neurons. In addition, the level of RAGE protein was highest at 5 days post-ischemia. In brief, both the RAGE immunoreactivity and protein level were distinctively increased in astrocytes in the ischemic CA1 region from 3 days after transient cerebral ischemia. These results indicate that the increase of RAGE expression in astrocytes at post-ischemia may be related to the ischemia-induced activation of astrocytes in the ischemic CA1 region.
( Jun Hwi Cho ),( Chan Woo Park ),( Myoung Chul Shin ),( Taek Geun Ohk ),( Joon Ho Bae ),( Dae Jin Jeong ),( Jun Yeol Lee ),( Ka Eul Kim ),( Ki Joong Kim ),( Doo Hyun Lee ),( Jung Hoon Lee ),( Moo Ho 대한응급의학회 2014 대한응급의학회 학술대회초록집 Vol.2014 No.2
Obesity induced by high-fat diet (HFD) is one of the most widespread metabolic disorders in current society. However, there has been little study regarding the effects of HFD induced obesity in the septum of animal models of cerebral ischemia. Therefore, in the present study, we investigated effects of HFD on neuronal damage and gliosis in the septum induced by transient cerebral ischemia. Body weight, blood glucose levels and serum lipid profiles levels were measured both in the normal diet (ND) and HFD-group. And, we also investigated the effects of ND and HFD on neuronal damage and gliosis in the septum after transient cerebral ischemia using immunohistochemistry. The levels of blood glucose, serum triglyceride and total cholesterol were significantly increased in the HFD-fed gerbils compared with the ND-fed gerbils, although body weight was not significantly changed after HFD feeding. In the ND-fed gerbils, ischemia-induced neuronal damage was found in the septo-hippocampal nucleus (SHN) of the septum 7 days after ischemia. In the HFD-fed gerbils, ischemia-induced neuronal damage in the SHN was much severer compared with the ND-fed gerbils 4 and 7 days after ischemia. In addition, we found that ischemia-induced glial activation including astrocytes and microglia was accelerated and exacerbated in the HFD-fed gerbils compared with that in the ND-fed gerbils. These results indicate that HFD can lead to much exacerbated effects in ischemia-induced neuronal damage/death in the septum after ischemia-reperfusion and that it may be associated with accelerated change in glial activation.
봄나물 섭취 후 이상 행동을 주요 증상으로 내원한 환자 1례
옥택근,김윤성,박찬우,문중범,최기훈,서정열,안희철,안무업,조준휘,Ohk Taek-Geun,Kim Yoon-Seong,Park Chan-Woo,Moon Joong-Bum,Choi Ki-Hoon,Seo Jeong-Yeul,Ahn Hee-Cheol,Ahn Moo-Wob,Cho Jun-Hwi 대한임상독성학회 2006 대한임상독성학회지 Vol.4 No.1
With well-being boom recently, the common people, but not experts, are increasingly taking in them directly by picking wild herbs. However, only experts can distinguish between edible vegetables and poisonous herbs from the wild herbs. Especially, it is not even easy for experts to distinguish them only by looking into buds in spring without flowers or fruit. So, sometimes many patients are being carried into the emergency room by the toxic symptom after taking by misunderstanding the poisonous herbs as edible vegetables. For herb intoxication as well as drug intoxication, what kinds of vegetables patients took in, and when and how much they took in them are important information for curing. Of course, in the case of intoxicating in the wild herbs, the patients can have a lot of symptoms and sings-hemodynamic alteration and abnormal neurologic sign as well as gastrointestinal symptoms-according to the kinds of herbs which they took in. Therefore, it is difficult to find the kinds of herbs which they took in through the specific symptoms. Recently we experienced a case with showing typical anticholinergic symptoms after ingestion of Scopolia japonica. As a result, the publicity activities about the wild plants needs because if the person without their previous knowledge take in the poisonous herbs, serious side effects can be produced..
( Jun Hwi Cho ),( Chan Woo Park ),( Myoung Chul Shin ),( Taek Geun Ohk ),( Joon Ho Bae ),( Dae Jin Jeong ),( Jun Yeol Lee ),( Ka Eul Kim ),( Ki Joong Kim ),( Doo Hyun Lee ),( Jung Hoon Lee ),( Moo Ho 대한응급의학회 2014 대한응급의학회 학술대회초록집 Vol.2014 No.2
Redd1 (known as RTP801/Dig2/DDIT4) is a stress-induced protein, and it is known to be regulated in response to some stresses including hypoxia and oxidative stress. In the present study, we investigated the time-course changes in Redd1 immunoreactivity and its protein levels in the gerbil hippocampus proper (CA1-3 regions) after 5 min of transient global cerebral ischemia using immunohistochemistry and western blot analysis. Redd1 immunoreactivity was apparently changed in the pyramidal neurons of the ischemic CA1 region, not the ischemic CA2/3 region. Redd1 immunoreactivity in the CA1 pyramidal neurons was significantly increased at 6 h post-ischemia, decreased until 1 day postischemia, increased again at 2 days post-ischemia and weakly observed at 5 days post-ischemia. Especially, at 5 days after ischemic damage, Redd1 immunoreactivity was newly expressed in astrocytes and GABAergic interneurons in the CA1 region. Redd1 protein levels in the ischemic CA1 region were changed like the pattern of the Redd1 immunoreactivity. These results indicate that Redd1 immunoreactivity and protein levels are increased in the ischemic CA1 region at early time after ischemic damage and that the increased Redd1 expression may be closely related to the delayed neuronal death of the CA1 pyramidal neurons following 5 min of transient global cerebral ischemia.
Yoon soo Park,Hyun Jin Tae,Jeong Hwi Cho,In Shik Kim,Taek Geun Ohk,Chan Woo Park,Joong Bum Moon,Myoung Cheol Shin,Tae Kyeong Lee,Jae Chul Lee,Joon Ha Park,Ji Hyeon Ahn,Seok Hoon Kang,Moo Ho Won,Jun Hw 대한해부학회 2018 Anatomy & Cell Biology Vol.51 No.2
Cardiac arrest (CA) is sudden loss of heart function and abrupt stop in effective blood flow to the body. The patients who initially achieve return of spontaneous circulation (RoSC) after CA have low survival rate. It has been known that multiorgan dysfunctions after RoSC are associated with high morbidity and mortality. Most previous studies have focused on the heart and brain in RoSC after CA. Therefore, the aim of this research was to perform serological, physiological, and histopathology study in the lung and to determine whether or how pulmonary dysfunction is associated with low survival rate after CA. Experimental animals were divided into sham-operated group (n=14 at each point in time), which was not subjected to CA operation, and CA-operated group (n=14 at each point in time), which was subjected to CA. The rats in each group were sacrificed at 6 hours, 12 hours, 24 hours, and 2 days, respectively, after RoSC. Then, pathological changes of the lungs were analyzed by hematoxylin and eosin staining, Western blot and immunohistochemistry for tumor necrosis factor (TNF-). The survival rate after CA was decreased with time past. We found that histopathological score and TNF- immunoreactivity were significantly increased in the lung after CA. These results indicate that inflammation triggered by ischemia-reperfusion damage after CA leads to pulmonary injury/dysfunctions and contributes to low survival rate. In addition, the finding of increase in TNF- via inflammation in the lung after CA would be able to utilize therapeutic or diagnostic measures in the future.
( Jun Hwi Cho ),( Chan Woo Park ),( Myoung Chul Shin ),( Taek Geun Ohk ),( Joon Ho Bae ),( Dae Jin Jeong ),( Jun Yeol Lee ),( Ka Eul Kim ),( Ki Joong Kim ),( Doo Hyun Lee ),( Jung Hoon Lee ),( Moo Ho 대한응급의학회 2014 대한응급의학회 학술대회초록집 Vol.2014 No.2
Hydroquinone (HQ) occurs naturally in various plants and food, and is also manufactured for commercial use. Recently, it has been reported that HQ has potent antioxidant properties with radical scavenging activities. In this study, the neuroprotective effect of HQ against ischemic damage following transient focal and global ischemia was examined using 2,3,5-triphenyltetrazoliumchloride (TTC) staining, NeuN immunohistochemistry and F-J B histofluorescence. Fig. It was proven that pre- and post-treatment with 100 mg/kg of HQ protects neurons from ischemic damage. In addition, we also investigated effects of HQ on gliosis and endogenous antioxidants in the hippocampal CA1 region after transient cerebral ischemia using immunohistochemistry. Pre- and post-treatment with 100 mg/kg of HQ notably inhibited the glial activation and maintained the expression of endogenous antioxidants in the gerbil ischemic CA1 region. Briefly, these results indicate that pre- and post-treatment with HQ can protect against ischemic damage induced by transient focal and global ischemia, and the neuroprotective effects of HQ may be closely associated with the attenuation of glial activation and the maintenance of the expression of endogenous antioxidants.