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A new hybrid method for bearing fault diagnosis based on CEEMDAN and ACPSO-BP neural network
Shanshan Song,Shuqing Zhang,Wei Dong,Xiaowen Zhang,Wei Ma 대한기계학회 2023 JOURNAL OF MECHANICAL SCIENCE AND TECHNOLOGY Vol.37 No.11
As an important part of rotating machinery, the failure of bearings will cause serious vibration and noise of mechanical equipment, which will affect the normal operation of the equipment and even lead to economic losses and casualties. To accurately and efficiently diagnose the working state and fault category of bearings, a new fault diagnosis method for rolling bearings based on the complete ensemble empirical mode decomposition with adaptive noise (CEEMDAN), weighted permutation entropy (WPE) and adaptive chaotic particle swarm optimization back propagation (BP) neural network (ACPSO-BP) was proposed. CEEMDAN and WPE were used to extract fault features and optimize the feature vector by mean domain specification principles. ACPSO optimizes the convergence speed and recognition accuracy of the BP neural network by introducing an adaptive tent mapping interval. The experimental results on bearing data from Western Reserve University and actual wind turbine data show that the proposed diagnosis method can achieve high fault recognition accuracy with a small number of training samples.
The complement system: a potential target for the comorbidity of chronic pain and depression
Shanshan Tang,Wen Hu,Helin Zou,Qingyang Luo,Wenwen Deng,Song Cao 대한통증학회 2024 The Korean Journal of Pain Vol.37 No.2
The mechanisms of the chronic pain and depression comorbidity have gained significant attention in recent years. The complement system, widely involved in central nervous system diseases and mediating non-specific immune mechanisms in the body, remains incompletely understood in its involvement in the comorbidity mechanisms of chronic pain and depression. This review aims to consolidate the findings from recent studies on the complement system in chronic pain and depression, proposing that it may serve as a promising shared therapeutic target for both conditions. Complement proteins C1q, C3, C5, as well as their cleavage products C3a and C5a, along with the associated receptors C3aR, CR3, and C5aR, are believed to have significant implications in the comorbid mechanism. The primary potential mechanisms encompass the involvement of the complement cascade C1q/C3- CR3 in the activation of microglia and synaptic pruning in the amygdala and hippocampus, the role of complement cascade C3/C3a-C3aR in the interaction between astrocytes and microglia, leading to synaptic pruning, and the C3a-C3aR axis and C5a-C5aR axis to trigger inflammation within the central nervous system. We focus on studies on the role of the complement system in the comorbid mechanisms of chronic pain and depression.
Shanshan Fang,Yawen Zhou,Man Zhou,ZHONGYU LI,SONG XU,Chao Yao 한국공업화학회 2018 Journal of Industrial and Engineering Chemistry Vol.58 No.-
ZnFe2O4/CdS composites were successfully prepared by a facile process. The obtained samples have beencharacterized by X-ray diffraction (XRD), scanning electron microscopy (FE-SEM), transmission electronmicroscopy (TEM), diffuse reflection spectroscopy (DRS), Raman spectroscopy and photoluminescence(PL). Under visible light irradiation, ZnFe2O4/CdS composite shows higher photocatalytic activity in the Cr(VI) reduction, relative to the pure CdS and ZnFe2O4. The enhanced photocatalytic performance wasascribed to the intimate contact interfaces and matching band potentials between CdS and ZnFe2O4, andcan be related to the efficient transfer and separation of the electron-hole pairs. It was found that themodification of ZnFe2O4 did not affect the morphology of CdS. The result of PL test indicated a lowerintensity, and effective separation of photogenerated electron-hole pairs. In addition, the ZnFe2O4/CdScomposites showed more intense optical absorption than that of pure CdS and ZnFe2O4. Through theanalysis of experiment results, a reasonable photocatalytic mechanism over ZnFe2O4/CdS photocatalystswas also proposed.
Gene expression profiles of skin from cyclin dependent kinases 5-knockdown mice
Yang Shanshan,Jiao Dingxing,Song Tao,Rui Ping,Fan Ruiwen,Ma Zengjun 아세아·태평양축산학회 2024 Animal Bioscience Vol.37 No.4
Objective: This study aimed to identify genes regulated by cyclin dependent kinases 5 (CDK5) that participate in hair pigmentation in mice. Methods: The mRNA expression profiles of skin samples from CDK5-knockdown mice were constructed using high-throughput RNA sequencing and compared with those of wild-type mice. Results: In total, 8,002 known genes were differentially expressed between CDK5-knockdown and wild-type mice. Of these, 3,658 were upregulated and 4,344 were downregulated in the skin of CDK5-knockdown mice. An additional 318 previously unknown genes were also differentially expressed, with 171 downregulated and 147 upregulated genes in the skin of CDK5-knockdown mice. Of the known genes expressed in mouse skin, 80 were associated with hair color, with 61 showing lower expression and 19 exhibiting higher expression in skin of CDK5-knockdown mice. Importantly, the expression of the tyrosinase-related protein 1 (TYRP1) and the calcium signaling pathway were also found to be regulated by CDK5, suggesting that pigmentation is regulated by CDK5 via the calcium signaling pathway and TYRP1. Conclusion: The transcriptome profiles obtained from the skin of CDK5-knockdown mice compared to wild-type mice provide a valuable resource to help understand the mechanism by which CDK5 regulates melanogenesis in mice and other animals.
Fangxia Guan,Xinxin Wang,Shanshan Ma,Nan Meng,Ning Yao,Kun Zhang,Qinghua Li,Yanting Zhang,Qu Xing,Kang Han,Jishi Song,Bo Yang 한국분자세포생물학회 2016 Molecules and cells Vol.39 No.5
Resveratrol (RES) plays a critical role in the fate of cells and longevity of animals via activation of the sirtuins1 (SIRT1) gene. In the present study, we intend to investigate whether RES could promote the self-renewal and neural-lineage differentiation in human umbilical cord derived MSCs (hUC-MSCs) in vitro at concentrations ranging from 0.1 to 10 M, and whether it exerts the effects by modulating the SIRT1 signaling. Herein, we demonstrated that RES at the concentrations of 0.1, 1 and 2.5 M could promote cell viability and proliferation, mitigate senescence and induce expression of SIRT1 and Proliferating Cell Nuclear Antigen (PCNA) while inhibit the expression of p53 and p16. However, the effects were reversed by 5 and 10 M of RES. Furthermore, RES could promote neural differentiation in a dose-dependent manner as evidenced by morphological changes and expression of neural markers (Nestin, Ⅲ-tubulin and NSE), as well as pro-neural transcription factors Neurogenin (Ngn)1, Ngn2 and Mash1. Taken together, RES exerts a dosage-dependent effect on the self-renewal and neural differentiation of hUC-MSCs via SIRT1 signaling. The current study provides a new strategy to regulate the fate of hUC-MSCs and suggests a more favorable in vitro cell culture conditions for hUC-MSCs-based therapies for some intractable neurological disorders.
Wang, Xinxin,Ma, Shanshan,Meng, Nan,Yao, Ning,Zhang, Kun,Li, Qinghua,Zhang, Yanting,Xing, Qu,Han, Kang,Song, Jishi,Yang, Bo,Guan, Fangxia Korean Society for Molecular and Cellular Biology 2016 Molecules and cells Vol.39 No.5
Resveratrol (RES) plays a critical role in the fate of cells and longevity of animals via activation of the sirtuins1 (SIRT1) gene. In the present study, we intend to investigate whether RES could promote the self-renewal and neural-lineage differentiation in human umbilical cord derived MSCs (hUC-MSCs) in vitro at concentrations ranging from 0.1 to $10{\mu}M$, and whether it exerts the effects by modulating the SIRT1 signaling. Herein, we demonstrated that RES at the concentrations of 0.1, 1 and $2.5{\mu}M$ could promote cell viability and proliferation, mitigate senescence and induce expression of SIRT1 and Proliferating Cell Nuclear Antigen (PCNA) while inhibit the expression of p53 and p16. However, the effects were reversed by 5 and $10{\mu}M$ of RES. Furthermore, RES could promote neural differentiation in a dose-dependent manner as evidenced by morphological changes and expression of neural markers (Nestin, ${\beta}III-tubulin$ and NSE), as well as pro-neural transcription factors Neurogenin (Ngn)1, Ngn2 and Mash1. Taken together, RES exerts a dosage-dependent effect on the self-renewal and neural differentiation of hUC-MSCs via SIRT1 signaling. The current study provides a new strategy to regulate the fate of hUC-MSCs and suggests a more favorable in vitro cell culture conditions for hUCMSCs-based therapies for some intractable neurological disorders.
RESEARCH ON MILLIMETER WAVE RADAR SIMULATION MODEL FOR INTELLIGENT VEHICLE
Xin Li,Weiwen Deng,Sumin Zhang,Yaxin Li,Shiping Song,Shanshan Wang,Guanyu Wang 한국자동차공학회 2020 International journal of automotive technology Vol.21 No.2
Radar simulation models can effectively overcome the drawbacks of real vehicle experiment and speed up the development process of intelligent vehicle technologies based on millimeter wave radar via virtual testing. However, there are still many gaps between the radar model using in the virtual driving environment and the real radar. In this paper, a novel simulation model of intelligent vehicle millimeter wave radar is proposed. Based on the analysis of the real radar performance in typical application scenes, the radar model considers the mechanism and characteristics of the vehicle radar synthetically and a systematic radar modeling architecture with innovation is introduced. The highlights of this radar model include the design of the RCS simulation model for radar targets with both high accuracy and real-time performance, the establishment of the quantitative false alarm model, missed detection model and measurement error simulation model. Vast amounts of data collected by real vehicle radar are applied to fetch model parameters and verify the accuracy of the radar model. Simulation results show that the proposed model can reach both high reliability and computational efficiency.
Chen, Jianbo,Li, Meijia,Chen, Lixue,Wang, Yufang,Li, Shanshan,Zhang, Yuwei,Zhang, Lei,Song, Mingjie,Liu, Chang,Hua, Mei,Sun, Yinshi The Korean Society of Ginseng 2018 Journal of Ginseng Research Vol.42 No.1
Background: The use of different methods for the processing of ginseng can result in alterations in its medicinal properties and efficacy. White ginseng (WG), frozen ginseng (FG), and red ginseng (RG) are produced using different methods. WG, FG, and RG possess different pharmacological properties. Methods: WG, FG, and RG extracts and pure ginsenosides were administered to rats to study the pharmacokinetics and tissue distribution characteristics of the following ginsenosides-DRg1, Re, Rb1, and Rd. The concentrations of the ginsenosides in the plasma and tissues were determined using UPLC-MS/MS. Results: The rate and extent of absorption of Rg1, Re, Rb1, and Rd appeared to be affected by the different methods used in processing the ginseng samples. The areas under the plasma drug concentration-time curves (AUCs) of Rg1, Re, Rb1, and Rd were significantly higher than those of the pure ginsenosides. In addition, the AUCs of Rg1, Re, Rb1, and Rd were different for WG, FG, and RG. The amounts of Rg1, Re, Rd, and Rb1 were significantly (p < 0.05) higher in the tissues than those of the pure ginsenosides. The amounts of Re, Rb1, and Rd from the RG extract were significantly higher than those from the WG and FG extracts in the heart, lungs, and kidneys of the rats. Conclusion: Our results show that the use of different methods to process ginseng might affect the pharmacokinetics and oral bioavailability of ginseng as well as the tissue concentrations of Rg1, Re, Rd, and Rb1.