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Obstructive ileus caused by phlebosclerotic colitis
Seung Hyun Lee,Jong Wook Kim,Se Jin Park,Ju Yeol Heo,Woo Hyun Paik,Won Ki Bae,Nam-Hoon Kim,Kyung-Ah Kim,June Sung Lee 대한장연구학회 2016 Intestinal Research Vol.14 No.4
A 57-year-old man with chronic kidney disease and a history of using numerous herbal medications visited Inje University IlsanPaik Hospital for abdominal pain and vomiting. An abdominal radiograph showed diffuse small bowel distension containingmultiple air-fluid levels and extensive calcifications along the colon. Computed tomography showed colon wall thickeningwith diffuse calcification along the colonic mesenteric vein and colonic wall. Colonoscopy, performed without bowel preparation,showed bluish edematous mucosa from the transverse to the distal sigmoid colon, with multiple scar changes. At the midtransverse colon, a stricture was noted and the scope could not pass through. A biopsy of the stricture site revealed nonspecificchanges. The patient was diagnosed with phlebosclerotic colitis. After the colonoscopy, the obstructive ileus spontaneouslyresolved, and the patient was discharged without an operation. Currently, after 2 months of follow-up, the patient has remainedasymptomatic. Herein, we report the rare case of an obstructive ileus caused by phlebosclerotic colitis with a colon stricture.
백승(Seung Paik),김준명(June Myeong Kim),정재복(Jae Bock Chung),박준용(Jun Yong Park),김응(Eung Kim),홍천수(Chein Soo Hong),최흥재(Heung Jai Choi) 대한내과학회 1988 대한내과학회지 Vol.35 No.5
N/A In a nine-year retrospective study, there were S5 episodes (2.24%) of bacteremia among 3789 patients with liver cirrhosis. The total number of causative microorganisms was 87 strains. The mean patient age was 49.6±10.5 years. Of 85 patients, 69 were male and 16 were female giving a ratio of approximately 4.3 to 1. The peak of highest age incidence was the fourth and fifth decades. Community acquired bacteremia numbered 59 episodes and hospital acquired, 26 episodes. In hospital acquired bacteremia, twenty patients (76.9%) underwent one or more major procedures such as gastroscopy, endoscopic sclerotherapy, balloon tamponade, paracentesis, or intraabdominal surgery just before the onset of bacteremia. The causative microorganisms were 72 strains of gram-negative bacteria (82.8%) and 15 strains of gram-positive bacteria (17.2%). Of the more common microorganisms, E. coli accounted for 37.9 percent, Klebsiella 16.1 percent and Staphylococcus 14.9 percent. The incidence of Staphylococcus was relatively high in community acquired bacteremia, and Klebsiella was high in hospital acquired bacteremia. In community acquired bacteremia, E, coli were highly susceptible to aminoglycoside, cefamandole and third generation cephalosporin. Klebsiella were susceptible to aminoglycoside and second and third generation cephalosporin. Staphylococcus were highly susceptible to cephalothin, methicillin and clindamycin. In hospital acquired bacteremia, E. coli were highly susceptible to aminoglycoside and third generation cephalosporin, Klebsiella to aminoglycoside, cephalosporin and chloramphenicol, and Staphylococcus were susceptible to cephalothin, but 50% were susceptible to methicillin, clindamycin, erythromycin and chloramphenicol. Of 85 bacteremic patients with liver cirrhosis, 31 patients (36.5%) died; in community acquired bacteremia, 23 patients (37.7%) died and in hospital acquired bacteremia, 8 patients (30.8%) died. The causes of death were sepsis and/or septic shock, bleeding, hepatic coma, etc. The severity of the cirrhosis was assessed according to Child's grading; 2 patients were found to be in claw A (2.4/c ), 12 in class B (14,1%), and 71 in class C (83.5%), Seventy-three patients suffered from one or more of the following complications: ascites, encephalopathy, hematemesis, and spontaneous bacterial peritonitis. Two or more (mean 2.2) complications were associated with mortality eases, whereas approximately one (mean 1.3) complication was seen in improved patients. In conclusion, patients suffering from liver cirrhosis were highly susceptible to infection because of various defects in the defence system, and when bacteremia developed, the prognosis was very poor. Therefore, bacteremia should be considered a serious complication in liver cirrhosis.
중합효소연쇄반응을 이용한 늑막액과 늑막생검조직에서의 결핵균 DNA 검출
한승범,허정숙,전영준,백성덕,백원기,서민호 啓明大學校 醫科大學 1994 계명의대학술지 Vol.13 No.2
Polymerase Chain Reaction(PCR) amplification was used to detect Mycobacterium tuberculosis DNA in pleural biopsy tissues and plerual fluids from patients suspected as tuberculosis. Oligonucleotide pairs for M. tuberculosis IS6110 DNA were used as primers and M. tuberculosis H37Rv strain was used as a positive control. Amplified products of 123 base pair fragments were detected by agarose gel electrophoresis and by Southern blotting with digoxigenin-labeled M. tuberculosis IS6110 DNA probe. DNA were isolated directly form the clinical specimens by bead-beating technic. Positive results were obtained in 13 of 15(86.6%) pleural biopsy tissues and 17 of 21(80.9%) pleural fluids. All the positive results were positive both in agarose gels and in Southern blottings. The relationships between the biopsy findings and PCR results were evaluated. Two 「tuberculosis」, 10 「granulomatous inflammations」, and one 「non-made」 findings of biopsy results were all positive by PCR. But one 「chronic inflammation」 and one 「inadequate specimen」 findings of biopsy results were all negative by PCR. In conclusion, these results showed that the PCR provides a very sensitive and efficient tool for the accurate and repaid diagnosis of tuberculosis infection.
( Kwang Min Kim ),( Sung June Eo ),( Geum Youn Gwak ),( Moon Seok Choi ),( Joon Hyeok Lee ),( Kwang Cheol Koh ),( Byung Chul Yoo ),( Seung Woon Paik ) 대한간학회 2011 Gut and Liver Vol.5 No.4
Background/Aims: The notion that acute hepatitis A superimposed on chronic hepatitis B infection leads to a worse outcome than acute hepatitis A alone remains controversial. The aim of this study was to determine the infl uence of the presence of hepatitis B surface antigen (HBsAg) on the severity of acute hepatitis A. Methods: We retrospectively analyzed 449 patients hospitalized for acute hepatitis A from January 2000 to February 2010 and compared clinical outcomes based on the presence of HBsAg. Results: Of the 449 patients, 30 patients were in the HBsAg-positive group and 419 in the HBsAg-negative group. The HBsAg-positive group was older than the HBsAg-negative group (36.1±8.3 vs 31.8±8.5 years, p=0.004); however, other baseline characteristics were similar between the 2 groups. Mean peak values of prothrombin time, serum total bilirubin, and serum creatinine at admission were significantly higher in the HBsAg-positive group. When comparing clinical outcomes between the 2 groups, gastrointestinal bleeding, acute renal failure, and acute liver failure were more frequently observed in the HBsAg-positive group. In particular, the incidence of acute liver failure was approximately 9-fold higher in the HBsAg-positive group than in the HBsAg-negative group (23.3% vs 3.3%; odds ratio [OR], 8.80; p<0.001). Multivariate analysis showed that HBsAg (OR, 7.43; 95% confi dence interval [CI], 2.56 to 21.57) and age (OR, 1.07; 95% CI, 1.02 to 1.13) were independent risk factors for the occurrence of acute liver failure. Conclusions: In patients with chronic hepatitis B infection, acute hepatitis A is associated with more severe clinical outcomes, including acute liver failure, compared with patients with acute hepatitis A alone. (Gut Liver 2011;5:500-505)